Human Physiology: From Cells to Systems (MindTap Course List)
9th Edition
ISBN: 9781285866932
Author: Lauralee Sherwood
Publisher: Cengage Learning
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Chapter 4, Problem 3TAHL
Summary Introduction
To write:
The pattern of postsynaptic potentials that is expected to be started as a reflex in the cell bodies of neurons regulating the EPSPs and IPSPs. The pattern of postsynaptic potentials that are voluntarily produced in the neurons that control the biceps and triceps despite the painful stimulus.
Introduction:
Synapse can be defined as a junction present between the two neurons. It comprises of a tiny gap through which the impulses pass by the diffusion of a neurotransmitter. The electrical or chemical signal is passed from one neuron to another neuron or to the target.
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Chapter 4 Solutions
Human Physiology: From Cells to Systems (MindTap Course List)
Ch. 4.1 - Name the two types of excitable tissue.Ch. 4.1 - Prob. 2CYUCh. 4.1 - State the factor responsible for triggering gate...Ch. 4.2 - Prob. 1CYUCh. 4.2 - Prob. 2CYUCh. 4.2 - Prob. 3CYUCh. 4.3 - Draw and label an action potential, indicating the...Ch. 4.3 - Prob. 2CYUCh. 4.3 - Prob. 3CYUCh. 4.3 - Prob. 4CYU
Ch. 4.4 - Explain why synapses operate only in the direction...Ch. 4.4 - Prob. 2CYUCh. 4.4 - Prob. 3CYUCh. 4.4 - Prob. 4CYUCh. 4.5 - Define target cell.Ch. 4.5 - Distinguish among the four types of extracellular...Ch. 4.5 - Outline the three general means by which binding...Ch. 4.5 - Prob. 4CYUCh. 4.6 - Distinguish between cytokines and eicosanoids.Ch. 4.6 - Discuss the roles of phospholipase A2,...Ch. 4.6 - Prob. 3CYUCh. 4.7 - Prob. 1CYUCh. 4.7 - Prob. 2CYUCh. 4.7 - Prob. 3CYUCh. 4.8 - Prob. 1CYUCh. 4.8 - Prob. 2CYUCh. 4 - Conformational changes in channel proteins brought...Ch. 4 - Prob. 2RECh. 4 - Prob. 3RECh. 4 - Prob. 4RECh. 4 - Second-messenger systems ultimately bring about...Ch. 4 - Each steroidogenic organ has all the enzymes...Ch. 4 - Prob. 7RECh. 4 - Prob. 8RECh. 4 - Prob. 9RECh. 4 - Prob. 10RECh. 4 - Prob. 11RECh. 4 - Prob. 12RECh. 4 - Prob. 13RECh. 4 - A common membrane-bound intermediary between the...Ch. 4 - Prob. 15RECh. 4 - Prob. 16RECh. 4 - Prob. 17RECh. 4 - Prob. 18RECh. 4 - Define the following terms: polarization,...Ch. 4 - Prob. 2UCCh. 4 - Prob. 3UCCh. 4 - Prob. 4UCCh. 4 - Compare the four kinds of gated channels in terms...Ch. 4 - Prob. 6UCCh. 4 - Prob. 7UCCh. 4 - Prob. 8UCCh. 4 - Prob. 9UCCh. 4 - Define signal transduction.Ch. 4 - Compare the tyrosine kinase and JAK/STAT pathways.Ch. 4 - Prob. 12UCCh. 4 - Prob. 13UCCh. 4 - Describe how arachidonic acid is converted into...Ch. 4 - Prob. 15UCCh. 4 - Prob. 16UCCh. 4 - Explain how the cascading effect of hormonal...Ch. 4 - Prob. 18UCCh. 4 - Answer the following questions regarding...Ch. 4 - Prob. 2SQECh. 4 - Prob. 3SQECh. 4 - Prob. 1ACRCh. 4 - The rate at which the Na+K+ pump operates is not...Ch. 4 - Which of the following would occur if a neuron...Ch. 4 - Prob. 3TAHLCh. 4 - Assume presynaptic excitatory neuron A terminates...Ch. 4 - Prob. 5TAHL
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Similar questions
- Assume presynaptic excitatory neuron A terminates on a postsynaptic cell near the axon hillock and presynaptic excitatory neuron B terminates on the same postsynaptic cell on a dendrite located on the side of the cell body opposite the axon hillock. Explain why rapid firing of presynaptic neuron A could bring the postsynaptic neuron to threshold through temporal summation, thus initiating an action potential, whereas firing of presynaptic neuron B at the same frequency and the same magnitude of EPSPs may not bring the postsynaptic neuron to threshold.arrow_forwardSpastic paralysis (resulting from the failure of muscles to relax), induced by the neurotoxin strychnine, occurs when this poison blocks the binding of: the excitatory neurotransmitter acetylcholine to post-synaptic sodium channels the excitatory neurotransmitter glutamate to post-synaptic calcium channels the excitatory neurotransmitter serotonin to post-synaptic potassium channels the inhibitory neurotransmitter glycine to post-synaptic chloride channels the inhibitory neurotransmitter melatonin to post-synaptic bicarbonate channelsarrow_forwardAn action potential has reached the axon terminal of a presynaptic cell. As a result of the action potential you would expect the movement of __________ into the presynaptic cell and the movement of __________ out of the presynaptic cell. Calcium : neurotransmitter Sodium : calcium Neurotransmitter : calcium Sodium : neurotransmitterarrow_forward
- Explain what happens when an action potential reaches the axon terminus (terminal) by activating the receptors on the postsynaptic membrane. Explanation addressing the question above When an action potential, or nerve impulse, arrives at the axon terminal, it activates voltage-gated calcium channels in the cell membrane. Which is present at a much higher concentration outside the neuron than inside, rushes into the cell. The Ca2+ allows synaptic vesicles to fuse with the axon terminal membrane, releasing neurotransmitters into the synaptic cleft. The molecules of neurotransmitters diffuse across the synaptic cleft and bind to receptor proteins on the postsynaptic cell. Activation of postsynaptic receptors leads to the opening or closing of ion channels in the cell membrane. This may be depolarizing, making the inside of the cell more positive, or hyperpolarizing making the inside more negative depending on the ions involved. The excitatory and inhibitory postsynaptic potentials…arrow_forwardNeuron A is an inhibitory neuron that is activated when the person feels heat in the tip of their index finger. Neuron A sends signals to neuron B (the postsynaptic neuron). First consider how a stimulus impacts the firing rate of neuron A. For example, when the person touches ice with their index finger vs. when the person touches a hot cup of coffee with their index finger? How would each of these impact the frequency of action potentials, the size of action potentials and the amount of neurotransmitter released. Second, consider Drug-D that causes all sodium channels in neurons to close immediately. How would taking Drug-D affect the firing rate of neuron A when the person touches a hot pan? In turn, how would this affect the firing rate of post-synaptic neuron B? Rubric: Impact of heat on A Impact of ice on A ( Impact of drug X on A Impact of drug X on B 1arrow_forwardAn undisclosed drug (Drug A) is a drug that can block voltage-gated K+ channels when being introduced into the bloodstream. Explain the effects of Drug A on the action potentials produced by a neuron. If Drug A could be applied selectively to a presynaptic neuron that releases an excitatory neurotransmitter, analyse how would it alter the synaptic effect of the neurotransmitter on the presynaptic cell. Remark: The word count limit (250 words only), no less than 100 wordsarrow_forward
- Describe how an action potential in the presynaptic neuron is generated in the postsynaptic neuron. Label the presynaptic neuron, postsynaptic neuron and synapse. Explain what is happening in figure on the left. Use key terms in order AND label on diagram: presynaptic axon terminal, action potential, calcium, vesicle, neurotransmitters, synapse Explain what is happening in the figure on the right. Use key terms in order AND label on diagram: neurotransmitter, postsynaptic receptor, sodium ions, excitatory postsynaptic potential (EPSP), depolarization, action potential, postsynaptic neuronarrow_forwardWhich of the following are the likely explanations for the failure of a postsynaptic neuron to trigger an action potential? Select all that apply. The EPSPs are neutralized by the neurotransmitter The summation of IPSPs and EPSPs is insufficient to depolarize the cell to threshold The synapses generating the EPSPs are too close to the axon hillock The synapses generating the EPSPs are too far from the axon hillockarrow_forwardSynaptic transmission depends upon :- a-direct transmission of impulses from the presynaptic neuron to the postsynaptic neuronb- diffusion of neurotransmitters from synaptic knobs into the soma and dendrites of postsynaptic neuronsc- presence of voltage-gated Ca ++ channels in membrane of synaptic knobsd- presence of voltage-gated Ca ++ channels in the subsynaptic membranearrow_forward
- I have stimulated a neuron with dopamine for a second followed by treating it with cocaine and calcium chelating agent (which chelates out all calcium from the system making them unavailable for function), what will be the effect of such impulse on generating an action potential in the post-synaptic neuron? Will it be different if I pretreat the set of nerve cells with cocaine and calcium chelating agent followed by stimulating the neuron with dopamine? Justify your answer with proper reasoning in brief.arrow_forwardSummarize the steps in generating an action potential as a flowchart. You can make your flowchart on paper and take a picture of it, or make it electronically. Be sure you’ve included: the location in the neuron and components of the neuron involved, the types of cellular transport and ions involved, how action potentials can be stimulated and inhibited. you can get the information from this: https://youtu.be/HYLyhXRp298arrow_forwardExcitatory neurotransmitters open up some sodium ion channels in the postsynaptic dendrite. This allows some sodium ions (Na+) to enter the postsynaptic dendrite, making the membrane potential more positive and bringing it closer to the threshold potential. This increases the likelihood of an action potential. Inhibitory neurotransmitters open up ion channels in the postsynaptic membrane for positive potassium ions (K+) to flow out OR negative chloride ions (Cl-) to flow in. Whether potassium ions go out or chloride ions go in, the postsynaptic cell becomes more negative than the resting membrane potential (further from the threshold potential). This decreases the likelihood of an action potential. The effect of the inhibitory neurotransmitter on the postsynaptic membrane is to a. maintain the resting potential b. hyperpolarize the postsynaptic membrane c. depolarize the postsynaptic membrane d. decrease the threshold levelarrow_forward
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