Study Guide for Campbell Biology
11th Edition
ISBN: 9780134443775
Author: Lisa A. Urry, Michael L. Cain, Steven A. Wasserman, Peter V. Minorsky, Jane B. Reece, Martha R. Taylor, Michael A. Pollock
Publisher: PEARSON
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Chapter 18, Problem 23TYK
Summary Introduction
Introduction: Apoptosis is also called programmed cell death. It involves biochemical mechanisms that lead to cell death. The apoptosis helps to prevent the development of cancer. The apoptosis is initiated by various factors in response to internal and external factors.
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Mutations to proto-oncogenes act toa. increase cell division. c. repress oncogenes.b. increase DNA repair. d. decrease cell division
Which of the following statements
concerning p53 is NOT correct?
O a. p53-dependent transcription of target genes leads
to cell cycle arrest or cell death (apoptosis).
O b.
O C.
Loss of p53 function results in loss of checkpoint
controls.
Loss of p53 function creates an environment that
is permissive for genome instability--that is, more
damaged cells with chromosome aberrations and
mutations survive and propagate.
d. p53 is a tumor suppressor protein which is largely
responsible for protecting cells from cancer-
causing DNA-damaging agents.
Hypermethylation is thought to contribute to cancer by
a. inhibiting DNA replication.
b. inhibiting the expression of tumor-suppressor genes.
c. stimulating the translation of oncogenes.
d. stimulating telomerase.
Chapter 18 Solutions
Study Guide for Campbell Biology
Ch. 18 - In the following diagram of the lac operon, an...Ch. 18 - a. Repressible enzymes usually function in _____...Ch. 18 - a. Give an example of highly methylated and...Ch. 18 - Label the components of the following diagram that...Ch. 18 - a. How is the process of alternate RNA splicing...Ch. 18 - a. Describe how miRNAs regulate gene expression....Ch. 18 - a. What is the difference between determination...Ch. 18 - What type of evidence established that Bicoid...Ch. 18 - a. List three genetic changes that can convert a...Ch. 18 - Complete the following concept map to help you...
Ch. 18 - Fill in the following table to help you organize...Ch. 18 - Prob. 3SYKCh. 18 - Prob. 1TYKCh. 18 - Prob. 2TYKCh. 18 - Prob. 3TYKCh. 18 - Prob. 4TYKCh. 18 - Prob. 5TYKCh. 18 - DNA methylation of cytosine bases a. initiates the...Ch. 18 - Which of the following is not true of enhancers?...Ch. 18 - Prob. 8TYKCh. 18 - A eukaryotic gene typically has all of the...Ch. 18 - Prob. 10TYKCh. 18 - Prob. 11TYKCh. 18 - Which of the following statements explains why a...Ch. 18 - Prob. 13TYKCh. 18 - Prob. 14TYKCh. 18 - Prob. 15TYKCh. 18 - Prob. 16TYKCh. 18 - What would be the fate of a Drosophila larva that...Ch. 18 - Prob. 18TYKCh. 18 - Prob. 19TYKCh. 18 - Prob. 20TYKCh. 18 - Prob. 21TYKCh. 18 - Prob. 22TYKCh. 18 - Prob. 23TYKCh. 18 - Which of the following would most likely account...
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- Sometimes, malignant cells do not stay properly anchored in tissues because of the loss of function of: a. cell walls b. oncogenes c. plasma membrane adhesion proteins d. nuclear membranes e. tumor suppressor genesarrow_forwardNormal (nonmutant) tumor-suppressor genes often functiona. as negative regulators of cell division.b. in the maintenance of genome integrity.c. in the stimulation of cell division.d. as both a and b.arrow_forwardAssuming that apoptosis fails and the cell continues through several cell cycles... what would the repercussions of this be? A. a tumor (possibly malignant) may develop- result of cells actively dividing when they are not supposed to B. organ failure C. too many cells would be in G0 and not carrying out daily functions D. too many mutated cells would be in the body and the body would shut downarrow_forward
- Most forms of cancer involvea. the activation of a single oncogene.b. the inactivation of a single tumor-suppressor gene.c. the activation of multiple oncogenes.d. the activation of multiple oncogenes and the inactivation ofmultiple tumor-suppressor genes.arrow_forwardWhich of the following types of mutations would be advantageous to a cancer cell (select all that apply)? A. An inactivating mutation in a tumor suppressor gene B. Methylation of the promoter of a tumor suppressor gene C. An inactivating mutation in an oncogene D. Mutation that inactivated DNA repair gene E. An inactivating mutation in an oncogenearrow_forwardWhich of the following is typical of cancer cells? A. The parent cell of the tumor contains a single mutation in a single checkpoint gene. B. Malignant cells migrate. C. Cancer cells lose the ability to divide. D. Products of oncogenes inhibit mitosis E. Benign tumors invade normal tissue.arrow_forward
- A tumor suppressor genea. inhibits cell division.b. opposes oncogenes.c. prevents cancer.d. is subject to mutations.e. All of these are correct.arrow_forwardThe protein that ensures the fidelity of DNA replication is the a. tumor repressor P53 protein. b. Mitotic arrest deficient protein. c. transcription factor E2F protein. d. Ataxia telangiectasia mutated protein.arrow_forwardWhich of the following is NOT a way in which proto-oncogenes can change to become genes that induce cancer? Group of answer choices a. changes in a control element (enhancer) to increase transcription b. gene amplification c. changes in DNA sequence to produce a product that degrades rapidly d. movement of the gene adjacent to a different control element to increase transcription e. changes in DNA sequence to produce a product that isarrow_forward
- Which of the following accurately compares tumor suppressor genes and proto-oncogenes? A. Both tumor suppressor and proto-oncogenes prevent the spread of cancer by targeting and destroying existing cancer cells. B. A person who has both tumor suppressor and proto-oncogenes will always develop cancer. C. Proto-oncogenes code for growth factors, while tumor suppressor genes inhibit cell division of damaged cells. D. When a proto-oncogene mutates, it becomes a tumor suppressor gene.arrow_forwardP53 is a tumor suppressor protein. The protein is present in the cell at all times but is activated by protein kinases that are, in turned activated by DNA damage. What would be the result of a mutation of the P 53 gene in cell that resulted in a form of P53 that was permanently active? Group of answer choices A. the cell cycle would be inhibited in all cells in a tumor if it forms B. the cell cycle would be inhibited C. the cell would form a tumor D. the cell has a higher probability of forming a tumor E. the cell cycle of all cells in the body would be inhibitedarrow_forwardCells in a tumor contain mutated copies of a particular gene that promotes tumor growth. Gene therapy can be used to introduce a normal copy of this gene into the tumor cells. Would you expect this therapy to be effective if the mutated gene were an oncogene? A tumor-suppressor gene? Explain your reasoning.arrow_forward
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