Cdc5/Polo is a phosphatase. An overactive, gain of function mutation in Mad2 would cause metaphase arrest. A overactive, gain of function mutation in Cdc14 would cause prolonged metaphase or metaphase arrest. O The outcome of Cdh1/Hct1 activity is a rapid rise in MPF/M-CdkC activity.
Cdc5/Polo is a phosphatase. An overactive, gain of function mutation in Mad2 would cause metaphase arrest. A overactive, gain of function mutation in Cdc14 would cause prolonged metaphase or metaphase arrest. O The outcome of Cdh1/Hct1 activity is a rapid rise in MPF/M-CdkC activity.
Human Anatomy & Physiology (11th Edition)
11th Edition
ISBN:9780134580999
Author:Elaine N. Marieb, Katja N. Hoehn
Publisher:Elaine N. Marieb, Katja N. Hoehn
Chapter1: The Human Body: An Orientation
Section: Chapter Questions
Problem 1RQ: The correct sequence of levels forming the structural hierarchy is A. (a) organ, organ system,...
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Question

Transcribed Image Text:Which statement is likely true given the infomation in the diagram?
Net1
Cdc20
APC
АРС
Cdc5/Polo
Cdc14
--
Cdc20
APC
АРС
Hct1
Hct1
Lag
Mad2
Cdc20
Hct1
Cdc14
APC
APC
Incomplete
spindle
Sict
Sic1
В
Cdk1-cyclin
Anaphase inhibitors
Mitotic cyclins
S/G2
Metaphase
Anaphase
Spindle disassembly
Cytokinesis
DNA re-replication
O Cdc5/Polo is a phosphatase.
O An overactive, gain of function mutation in Mad2 would cause metaphase arrest.
O A overactive, gain of function mutation in Cdc14 would cause prolonged metaphase or metaphase arrest.
O The outcome of Cdh1/Hct1 activity is a rapid rise in MPF/M-CdkC activity.
H-1
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