Concept explainers
Interpretation:
The reason for obesity therapy to not to be a miracle cure for overweight individuals should be explained.
Concept introduction:
Leptin is AN acid amide created by fat cells in response to hormone and governs each intake habits and energy physiological state. a lot of specifically Leptin works by indicating the triglycerol level in adipocytes or fat cells to the central system. This dictates appetence. If a low quantity of Leptin is gift, then appetence will increase and contrariwise. The
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Check out a sample textbook solution- Give typed explanation of all subparts otherwise leave itarrow_forwardArrange the step in the dropdown menu in the image.arrow_forwardThis question is related to psychoneuroimmunology and the benefits of the supplement +omega-3 fatty acid Should people be looking further at nutritional health in response to chronic stress conditions, and incorporating this more into clinical care, or should they believe that therapy and medication alone should suffice in treating these chronic conditions?arrow_forward
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- For the mutation of the enzyme, describe the effect it will have on carbohydrate metabolism. Also, make sure to think about the effect it has on muscles versus the liver: Mutation that converted the active site glutamate in phosphohexose isomerase to an glutamine.arrow_forwardThe antidiabetes drugs known as thiazolidinediones induce the production of glycerol kinase in adipocytes. Explain why this would decrease the concentration of serum fatty acids, which are often elevated in diabetics.arrow_forwardFatty acids can be gluconeogenic precursors in plants, but not animals. O True Falsearrow_forward
- Consider a defect in MIG12 that prevents the binding of this protein to acetyl-CoA carboxylase. How does this defect impact the production of ketone bodies? Increased ketone bodies. Decreased ketone bodies. No effect. A new five-carbon ketone body forms.arrow_forwardWhich of the following are true about how pancreatic β cells sense blood glucose levels and release insulin? (select all that apply) Group of answer choices The first step of glycolysis (producing glucose 6-phosphate) is more efficient in pancreatic β cells than in muscle glucose transporters in pancreatic β cells have a higher affinity for glucose than the transporters in other tissues Increased production of ATP is used as a signal that blood glucose is high Insulin is released when blood glucose levels are high Increases in ATP open a K+ channel Influx of Ca2+ stimulates insulin releasearrow_forward7. During several days, Patient A got a meal with high caloric value, and Patient B- with a low caloric value. What are the differences in the lipid metabolism in these two patients? What is the insulin/glucagon ratio in these patients after a meal? Which metabolic pathways rather activated in Patient A and how is it being controlled? 8. The adipose tissuc is not only store triacylglycerols but also is active endocrine organ. Explain this function of the adipose tissuc. For that: a) explain the sclinical obesity and possible conscouences of the discase:arrow_forward
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