A white blood cell has pathogen recognition proteins embedded in its plasma membrane. Initially, the the pathogen recognition proteins are spread over the whole surface of the white blood cell. The white blood cell comes into contact with a bacterial cell. Some of the pathogen recognition proteins bind to the lipoteichoic acid molecules on the surface of the bacterial cell. As time goes on, more and more of the pathogen recognition proteins bind to the lipoteichoic acid molecules on the bacterial cell surface. The white blood cell membrane at the point of contact between these 2 cells now contains a dense cluster pathogen recognition proteins. How is it possible for the pathogen recognition proteins to go from being equally distributed on the white blood cell surface to being clustered at the point of contact?
Bacterial Genomics
The study of the morphological, physiological, and evolutionary aspects of the bacterial genome is referred to as bacterial genomics. This subdisciplinary field aids in understanding how genes are assembled into genomes. Further, bacterial or microbial genomics has helped researchers in understanding the pathogenicity of bacteria and other microbes.
Transformation Experiment in Bacteria
In the discovery of genetic material, the experiment conducted by Frederick Griffith on Streptococcus pneumonia proved to be a stepping stone.
Plasmids and Vectors
The DNA molecule that exists in a circular shape and is smaller in size which is capable of its replication is called Plasmids. In other words, it is called extra-chromosomal plasmid DNA. Vectors are the molecule which is capable of carrying genetic material which can be transferred into another cell and further carry out replication and expression. Plasmids can act as vectors.
- A white blood cell has pathogen recognition proteins embedded in its plasma membrane. Initially, the the pathogen recognition proteins are spread over the whole surface of the white blood cell. The white blood cell comes into contact with a bacterial cell. Some of the pathogen recognition proteins bind to the lipoteichoic acid molecules on the surface of the bacterial cell. As time goes on, more and more of the pathogen recognition proteins bind to the lipoteichoic acid molecules on the bacterial cell surface. The white blood cell membrane at the point of contact between these 2 cells now contains a dense cluster pathogen recognition proteins. How is it possible for the pathogen recognition proteins to go from being equally distributed on the white blood cell surface to being clustered at the point of contact?
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