A carnitine deficiency might manifest itself as...accumulation of long chain fatty acids in the mitochondrial matrix.an inability to run ß-oxidation on 16-carbon fatty acids.excess medium chain fatty acyl-CoA molecules in the mitochondrial matrix.O ketoacidosis.the inability to synthesize long chain fatty acids.

Introduction: Carnitine is a quaternary ammonium compound and is an important component of the…

Answered: A carnitine deficiency might manifest…

Biochemistry

9th Edition

ISBN:9781319114671

Author:Lubert Stryer, Jeremy M. Berg, John L. Tymoczko, Gregory J. Gatto Jr.

Publisher:Lubert Stryer, Jeremy M. Berg, John L. Tymoczko, Gregory J. Gatto Jr.

Chapter1: Biochemistry: An Evolving Science

Section: Chapter Questions

Problem 1P

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Similar questions

Which of the following enzymes is unique to the liver? Beta-hydroxybutyrate dehydrogenase HMG-CoA lyase Thiolase 3-Ketoacyl CoA transferase
For the mutation of the enzyme, describe the effect it will have on carbohydrate metabolism. Also, make sure to think about the effect it has on muscles versus the liver: Mutation that converted the active site cysteine in glyceraldehyde 3-phosphate dehydrogenase to a serine
Indicate whether each of the following B vitamins is involved (as a cofactor) in β-oxidation pathway, ketogenesis, lipogenesis, or conversion of ketone bodies to acetyl CoA. There may be more than one correct response or no correct response for a particular B vitamin.
How many molecules of Acetyl-CoA will be produced after complete ẞ-oxidation pathway of a 20:0 fatty acid? 12 14 10 8
What explains the side effect of "acetone-breath" on dieters following a low carbohydrate diet? O Decreased dietary carbohydrates stimulates fatty acid synthesis and ketogenesis; decarboxylation of acetoacetate generates acetone. Decreased dietary carbohydrates leads to decreased fatty acid oxidation and ketogenesis; decarboxylation of alpha-ketoglutarate generates acetone. Decreased dietary carbohydrates leads to increased fatty acid oxidation and gluconeogenesis; decarboxylation of pyruvate generates acetone. Decreased dietary carbohydrates leads to increased fatty acid oxidation and ketogenesis; decarboxylation of acetoacetate generates acetone.
A patient who has been drinking large amounts of alcohol for long periods of time shows thefollowing symptoms: apathy, loss of memory, and a rhythmical to-and-fro motion of the eyeballs.Which of the following reactions are most likely to be affected in the patient? A. Conversation of pyruvate to acetyl-CoA B. Conversation of a-ketoglutarate to succinyl-CoA C. Both A and B D. Neither A nor B
Can u help me to explain to me, please? I stuggled. I dont understand to explain the difference between ketogenesis and ketoacidosis. What's the difference between ketogenesis and ketoacidosis? Can you help me to explain to me about the difference between ketogenesis and ketoacidosis?
For the mutation of the enzyme, describe the effect it will have on carbohydrate metabolism. Also, make sure to think about the effect it has on muscles versus the liver: Mutation that converted the active site glutamate in phosphohexose isomerase to an glutamine.
A fatty acid (a long straight-chain carboxylic acid with an even number of carbons) is metabolized to acetyl- CoA, which can then enter the citric acid cycle to be further metabolized (Section 24.9). A fatty acid with anodd number of carbons is metabolized to acetyl-CoA and one equivalent of propionyl-CoA. Propionyl-CoA cannot enter the citric acid cycle. Two coenzyme-requiring enzymes are needed to convert it to succinyl-CoA, acompound that can enter the citric acid cycle. Write the two enzyme-catalyzed reactions and include the names of the required coenzymes.
A patient exhibiting all the symptoms of beriberi is placed on a thiamine-enriched diet; however, the symptoms do not disappear. Genetic screening suggests a defect in pyruvate dehydrogenase phosphatase. Briefly explain why the genetic defect caused beriberi symptoms andy why thiamine supplementation had no effect.
Match the statement to the corresponding metabolic pathway. Choices. (Lipogenesis or Lipolysis) Fatty Acid Synthase Fatty Acid to Acetyl CoA Primary sites involve liver, adipose tissue and mammary glands Beta-Oxidation Inner matrix of the Mitochondria Pyruvate from Glucose Occurs in Cell Cytosol Enters TCA Cycle Acetyl-CoA Carboxylase Triglyceride to Glycerol
Which enzyme involved in the breakdown of fatty acids requires activated vitamin B12? methylmalonyl-CoA mutase O propionyl-CoA carboxylase O acyl-CoA dehydrogenase enoyl-CoA isomerase

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