Using quail and chick embryos, quail-specific antibody and fluorescent tissue-specific antibodies, design an experiment where you investigate the tissues the cranial neural crest can give rise to. What are four derivatives of the cranial neural crest that you expect to see in the resulting chimeric embryos?

Using quail and chick embryos, quail-specific antibody and fluorescent tissue-specific antibodies, design an experiment where you investigate the tissues the cranial neural crest can give rise to. What are four derivatives of the cranial neural crest that you expect to see in the resulting chimeric embryos?

Biology (MindTap Course List)

11th Edition

ISBN:9781337392938

Author:Eldra Solomon, Charles Martin, Diana W. Martin, Linda R. Berg

Publisher:Eldra Solomon, Charles Martin, Diana W. Martin, Linda R. Berg

Chapter51: Animal Development

Section: Chapter Questions

Problem 13TYU

See similar textbooks

Similar questions

The gene controlling ABO blood type and the gene underlying nail-patella syndrome are said to show linkage. What does that mean in terms of their relative locations in the genome? What does it mean in terms of how the two traits are inherited with respect to each other?
A teratogen is an agent that produces nongenetic abnormalities during embryonic or fetal development. Suppose a teratogen is present at conception. As a result, during the first mitotic division the centromeres fail to divide. The teratogen then loses its potency and has no further effect on the embryo. What is the chromosomal complement of this embryo?
How would mutations that completely ablate the function of the androgen receptor impact the phenotypic development of humans with XY chromosomes? Patients would appear phenotypically female Patients would appear phenotypically male with underdeveloped secondary sex characteristics Patients would appear phenotypically male, but cannot produce sperm Patients would express both male and female secondary sex characteristics.
Researchers are learning how to transfer sperm-making cells from fertile male mice into infertile male mice in the hopes of learning more about reproductive abnormalities. These donor spermatogonia cells have developed into mature spermatozoa in 70% of cases, and some recipients have gone on to father pups (as baby mice are called). This new advance opens the way for a host of experimental genetic manipulations. It also offers enormous potential for correcting human genetic disease. One potentially useful human application of this procedure is treating infertile males who wish to be fathers. a. Do you foresee any ethical or legal problems with the implementation of this technique? If so, elaborate on the concerns. b. Could this procedure have the potential for misuse? If so, explain how.
Figure 42.16 The Rh antigen is found on Rh positive red blood cells. An Rh-negative female can usually carry an Rh-positive fetus to term without difficulty. However, if she has a second Rh-positive fetus, her body may launch an immune attack that causes hemolytic disease of the newborn. Why do you think hemolytic disease is only a problem during the second or subsequent pregnancies?
Organ Transplants Must Be Immunologically A couple has a young child who needs a bone marrow transplant. They propose that preimplantation screening be done on several embryos fertilized in vitro to find a match for their child. a. What do they need to match in this transplant procedure? b. The couple proposes that the matching embryo be transplanted to the mothers uterus and serve as a bone marrow donor when old enough. What are the ethical issues involved in this proposal?
VISUALIZE Label the following diagram and explain what this diagram illustrates.
Can conjoined (Siamese) twins be dizygotic twins in light of the theory that conjoined twins result from incomplete division of the embryo?
Analysis of X-Linked Dominant and Recessive Traits In the eighteenth century, a young boy with a skin condition known as ichthyosis hystrix gravior was identified. The phenotype of this disorder includes thickening of skin and the formation of loose spines that are sloughed off periodically. This man married and had six sons, all of whom had the same condition. He also had several daughters, all of whom were unaffected. In all succeeding generations, the condition was passed on from father to son. What can you theorize about the location of the gene that causes ichthyosis hystrix gravior?
Two genes associated with breast cancer, BRCA1 and BRCA2, were discovered in 1994 and 1995, respectively, and shortly thereafter, were patented by Myriad Genetics, a company based in Utah. Under the patents, testing for mutations in these genes could only be performed by Myriad, at costs from 300 to 3,000. Myriad also patented the process of analyzing the results of such tests, preventing anyone who obtains the sequence of their BRCA genes by other means (which itself would probably be patent infringement) from interpreting the information. The idea that genes can be patented has been a contentious issue from the beginning. Patents are not granted for products of nature, meaning that genes inside the body are not patentable, but biotech companies successfully argued that by removing a gene from the human body, purifying it, and then obtaining its DNA sequence, they created something not found in nature, and which is therefore a patentable invention. The U.S. Patent Office found the argument persuasive, but opponents argue that genes are parts of our bodies and can be identified but not invented. Biotech companies argue that without the protection offered by patents, they would have no incentive for research and development of diagnostic tests. In Europe, patents for BRCA1 and BRCA2 were revoked in 2004 because they did not meet the standards for a patent. After more than a decade of legal disputes, the patents were partially restored in 2008 on a very restricted basis. In the United States, a lawsuit, focused on the patents for the BRCA genes, was filed in May 2009. The suit challenges the basic idea that genes are patentable. In November 2009, the judge ruled that the lawsuit can proceed, and the case is moving forward. In March 2010, a federal court invalidated Myriad Genetics patent on these genes. In August 2011, the U.S. Court of Appeals reversed the lower courts decision and ruled that gene sequences isolated from cells are not a product of nature and are therefore patentable. The case went to the U.S. Supreme Court, which ordered the appeals court to reconsider the case. The Federal Appeals Court did not change its decision, and the case once again, went to the U.S. Supreme Court. A unanimous decision in June 2013 invalidated Myriads patents on the basis that isolating a gene from nature does not make it patentable. This is a landmark decision on gene patenting with widespread ramifications for the biotechnoloogy industry. Will this decision reduce the incentives for companies to invest in new diagnostic tests that would be used by cancer victims or those with serious genetic disorders?
In the embryonic development of the eye: a. the optic vesicle cells permanently adhere to each other to prevent movement, whereas the optic cup cells are very motile. b. the optic cup induces the surface ectoderm to form the lens placode. c. gradients determine that the ectoderm overlying the lens vesicle develops into the optic vesicle. d. microtubules powered by myosins and microfilaments powered by dyneins move the eye components around in the head region. e. cadherins function in the presence of calcium to allow the lens placode and optic cup to break apart.
Almost all calico cats (one is pictured in FIGURE 10.7B) are female. Why? B When this calico cat was an embryo, one of the two X chromosomes was inactivated in each of her cells. The descendants of the cells formed her adult body, which is a mosaic for expression of X chromosome genes. Black fur arises in patches where genes on the X chromosome inherited from one parent are expressed; orange fur arises in patches where genes on the X chromosome inherited from the other parent are expressed. FIGURE 10.7 Animated X chromosome inactivation.