Question 1: How do actryloside and bongkrekic acid affect mitochondrial respiration?What is the target of these molcules and how does it do what you think it does?Question 2: Why can proton ionophores be marketed as diet drugs and is it a good idea totake them?

Q. 2 AnswerProton ionophores are compounds that disrupt the proton gradient across the mitochondrial…

Answered: Question 1: How do actryloside and…

Biochemistry

9th Edition

ISBN:9781319114671

Author:Lubert Stryer, Jeremy M. Berg, John L. Tymoczko, Gregory J. Gatto Jr.

Publisher:Lubert Stryer, Jeremy M. Berg, John L. Tymoczko, Gregory J. Gatto Jr.

Chapter1: Biochemistry: An Evolving Science

Section: Chapter Questions

Problem 1P

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answer question #3
You experiementally apply the compund antimyctin, which inhinits the functioning of complex III, cytochrome reductase, from participating in redox reactions. You follow this with the addition of 6 molecules of FADH2. How many total ATP molecules can be created using the number of protons that were pumped?
How does the presence of cristae in the mitochondrial membrane increase the productivity of aerobic respiration in eukaryocytes?
this question is about 16 carbon fatty acid A) what are the end procuts of the eight cycles of beta oxidation of a 16 carbon fatty acid? B) assuming 2.5 ATP molecules are produced from one NADH, how many molecules of ATP are produced from the NADH produced during beta oxidation of a 16-carbon fatty acid
What are some of the difficulties in extracting respiratory complexes from the inner mitochondrial membrane to investigate their properties?
Focusing on the mechanism linking complex I and ATP synthase depicted in figure 3 in the article, compare that hypothetical mechanism to the classical presentation described in our textbook. What are the major differences between this mechanism and Peter Mitchel’s original chemiosmotic theory? What are the similarities.
What compounds are the immediate source of the "high energy electrons" that enter the electron transport system of mitochondria? Where do these electron-donating compounds gain their electrons? What type of compounds are these electron-donating compounds?
Free radicals are an interesting and important aspect of cellular energy generation that are implicated in a variety of biological conditions. Answer the following questions in one, succint & complete sentence. What are some examples of free radicals in the cell? How are free radicals generated in the cell from mitochondrial actions and what cellular effects do these molecules have?
7) The following questions apply to glycolysis (figure 3-41 on page 79 of the 12th edition), the Krebs cycle (figure 3-44 on page 82) and to oxidative phosphorylation (figure 3-45 on page 84). Calculate the total amount of ATP produced from one glucose molecule if: a. The oxidation of NADH + H* to NAD* + 2H* produced 4 ATP. b. The oxidation of FADH₂ to FAD + 2H* produced 1 ATP. c. Each step of the Krebs cycle that produces NADH+H* produced FADH2 instead. d. Each step of the Krebs cycle that produces FADH2 produced NADH + H+ instead. e. Sucrose is substituted for glucose.
Yeast cells lacking the electron transport system still have a weak membranepotential across the mitochondrial inner membrane. How do these cells generatea membrane potential?
The proton-motive force is a measure of the potential energy generated across the mitochondrial inner membrane during electron transport. Explain the factors that constitute this potential energy, that will be converted to the chemical energy of ATP by the cell. 2. The function of the Electron Transport Chain (ETC) in eukaryotic cells is to produce a proton gradient across the mitochondrial inner membrane. How many protons are taken up from the matrix and transferred or used at each ETC complex?
How does DCPIP act as an indicator of presence of mitochondria? Will mitochondria in the presence of DCPIP still make ATP? Why or why not?

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Question 1: How do actryloside and bongkrekic acid affect mitochondrial respiration? What is the target of these molcules and how does it do what you think it does? Question 2: Why can proton ionophores be marketed as diet drugs and is it a good idea to take them?