Campbell Biology 11th Edition - Valuepack
11th Edition
ISBN: 9780134833545
Author: Michael L. Cain, Steven A. Wasserman, Peter V. Minorsky, Jane B. Reece Neil A. Campbell Lisa A. Urry
Publisher: PEARSON
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Textbook Question
Chapter 48, Problem 6TYU
Suppose a particular neurotransmitter causes an IPSP in postsynaptic cell X and an EPSP in postsynaptic cell Y. A likely explanation is that
(A) the threshold value in the postsynaptic membrane is different for cell X and cell Y.
(B) the axon of cell X is myelinated, but that of cell Y is not.
(C) only cell Y produces an enzyme that terminates the activity of the neurotransmitter.
(D) cells X and Y express different receptor molecules for this particular neurotransmitter.
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Read the following statements
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Choose the option representing correct statements.
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. Suppose a particular neurotransmitter causes an IPSP inpostsynaptic cell X and an EPSP in postsynaptic cell Y. A likelyexplanation is that(A) the threshold value in the postsynaptic membraneis different for cell X and cell Y.(B) the axon of cell X is myelinated, but that of cell Y is not.(C) only cell Y produces an enzyme that terminates the activityof the neurotransmitter.(D) cells X and Y express different receptor molecules for thisparticular neurotransmitter.
Assume a neuron is at rest and the following changes happen:
Step 1: Neurotransmitter binds to an ionotropic excitatory neurotransmitter receptor causing an EPSP.
Step 2: With the neurotransmitter still bound either a NAM or non-competitive antagonist bind to the allosteric site of the receptor.
Answer the following questions that explains how the NAM and non-competitive antagonist would differ in their actions.
4.)
A. Going from step 1 to step 2 what would happen to the ionotropic receptor (ion channel) as a result of the NAM.
B. Going from step 1 to step 2 what would happen to the ionotropic receptor (ion channel) as a result of the non-competitive antagonist.
C. Going from step 1 to step 2 how would membrane potential change as a result of the NAM
D. Going from step 1 to step 2 how would the membrane potential change as a result of the non-competitive antagonist.
Chapter 48 Solutions
Campbell Biology 11th Edition - Valuepack
Ch. 48.1 - Prob. 1CCCh. 48.1 - Describe the basic pathway of information flow...Ch. 48.1 - WHAT IF? How might increased branching of an axon...Ch. 48.2 - Under what circumstances could ions flow through...Ch. 48.2 - WHAT IF? Suppose a cell's membrane potential...Ch. 48.2 - MAKE CONNECTiONS Review Figure 7.10, which...Ch. 48.3 - How do action potentials and graded potentials...Ch. 48.3 - In multiple sclerosis (from the Greek skleros,...Ch. 48.3 - How do both negative and positive feedback...Ch. 48.3 - WHAT IF? Suppose a mutation caused gated sodium...
Ch. 48.4 - Prob. 1CCCh. 48.4 - Some pesticides inhibit acetylcholinesterase, the...Ch. 48.4 - Prob. 3CCCh. 48 - How would severing an axon affect the flow of...Ch. 48 - Suppose you placed an isolated neuron in a...Ch. 48 - Prob. 48.3CRCh. 48 - Prob. 48.4CRCh. 48 - Level 1: Knowledge/Comprehension 1. What happens...Ch. 48 - Level 1: Knowledge/Comprehension 1. What happens...Ch. 48 - Where are neurotransmitter receptors located? (A)...Ch. 48 - Why are action potentials usually conducted in one...Ch. 48 - Which of the following is the most direct result...Ch. 48 - Suppose a particular neurotransmitter causes an...Ch. 48 - WHAT IF? Ouabain, a plant substance used in some...Ch. 48 - Prob. 8TYUCh. 48 - DRAW IT Suppose a researcher inserts a pair of...Ch. 48 - EVOLUTION CONNECTION An action potential is an...Ch. 48 - Prob. 11TYUCh. 48 - WRITE ABOUT A THEME: ORGANIZATION In a short essay...Ch. 48 - Prob. 13TYU
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- which one of the following statements is incorrect? a. temporal summation occurs when a single synaptic input is activated twice in succession, with the second postsynaptic potential occurring before the first postsynaptic potential is over. b. blocking voltage-gated k* channels in the presynaptic membrane of a typical chemical synapse is likely to reduce the amount of neurotransmitter released in response to a single action potential in the presynaptic axon. c. in the optic tectum of the hunting rattlesnake, spatial summation is used to combine inputs from visual and thermoreceptive layers in order to make a decision whether to strike at a mouse-like object. d. a single type of neurotransmitter can have different postsynaptic actions depending on the type of receptor to which it binds.arrow_forwardA loss-of-function mutation in the ion channel responsible for thegeneration of end-plate potentials results in a diminished permeabilityof the ion channel. 1) How would you experimentally test the effect of the mutation on thefunctional properties of the cell (synapse)? You should discuss the typesof postsynaptic responses and physiological parameters you couldmeasure.arrow_forwardLet’s say the synapses of a neuron’s dendrites are filled with glutamate-gated channels which, when activated by glutamate, cause an excitatory postsynaptic potential (EPSP) in the neuron. In one instance, glutamate is released at all of the synapses simultaneously and this leads to an action potential in the neuron. A) What type of summation of PSPs is this? Why? B) List the sequence of steps that occur starting from binding of glutamate to the glutamate-gated channels at the synapses and ending with the membrane potential at the axon hillock returning to resting potential at the end of the action potential. (Include all the changes in voltage-gated channels underlying the action potential.)arrow_forward
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