Concept explainers
(a)
Interpretation:
Whether the occurrence of two different dehydrogenation reactions is a characteristic of (1) the β-oxidation pathway but not ketogenesis, (2) ketogenesis but not β-oxidation pathway, (3) both the β-oxidation pathway and ketogenesis or (4) neither the β-oxidation pathway nor ketogenesis has to be determined.
Concept introduction:
Ketogenesis occurs in the mitochondria of the liver cells. The initial reactants are the molecules of acetyl CoA which are produced by the β-oxidation of fatty acid molecules.
β-oxidation is a catabolic process occurring in the body through which, fatty acid molecules are broken down in the mitochondria of the cells to generate energy. The process involves breaking down long fatty acid chains that have been converted to acyl CoA chains into smaller fatty acyl CoA chains. The fatty acid chain is broken down until the final acyl CoA chain that cannot be broken down any further. The end products of this
(b)
Interpretation:
Whether the breakdown of a molecule into two parts is a characteristic of (1) the β-oxidation pathway but not ketogenesis, (2) ketogenesis but not β-oxidation pathway, (3) both the β-oxidation pathway and ketogenesis or (4) neither the β-oxidation pathway nor ketogenesis has to be determined.
Concept introduction:
Ketogenesis occurs in the mitochondria of the liver cells. The initial reactants are the molecules of acetyl CoA which are produced by the β-oxidation of fatty acid molecules. Ketone bodies are generally produced when the amount of acetyl CoA in the body is much larger than the amount of oxaloacetate. This happens due to the carbohydrate-lipid imbalance in the body caused by much smaller intake of food rich in carbohydrates, or inefficient processing of glucose by the body.
β-oxidation is a catabolic process occurring in the body through which, fatty acid molecules are broken down in the mitochondria of the cells to generate energy. The process involves breaking down long fatty acid chains that have been converted to acyl CoA chains into smaller fatty acyl CoA chains. The fatty acid chain is broken down until the final acyl CoA chain that cannot be broken down any further. The end products of this metabolic process are acetyl CoA, FADH2 and NADH. These three molecules later enter the Krebs cycle where they are used to produce ATP molecules.
(c)
Interpretation:
Whether the occurrence of a hydrolysis reaction is a characteristic of (1) the β-oxidation pathway but not ketogenesis, (2) ketogenesis but not β-oxidation pathway, (3) both the β-oxidation pathway and ketogenesis or (4) neither the β-oxidation pathway nor ketogenesis has to be determined.
Concept introduction:
Ketogenesis occurs in the mitochondria of the liver cells. The initial reactants are the molecules of acetyl CoA which are produced by the β-oxidation of fatty acid molecules. Ketone bodies are generally produced when the amount of acetyl CoA in the body is much larger than the amount of oxaloacetate. This happens due to the carbohydrate-lipid imbalance in the body caused by much smaller intake of food rich in carbohydrates, or inefficient processing of glucose by the body.
β-oxidation is a catabolic process occurring in the body through which, fatty acid molecules are broken down in the mitochondria of the cells to generate energy. The process involves breaking down long fatty acid chains that have been converted to acyl CoA chains into smaller fatty acyl CoA chains. The fatty acid chain is broken down until the final acyl CoA chain that cannot be broken down any further. The end products of this metabolic process are acetyl CoA, FADH2 and NADH. These three molecules later enter the Krebs cycle where they are used to produce ATP molecules.
(d)
Interpretation:
Whether the occurrence of a hydration reaction is a characteristic of (1) the β-oxidation pathway but not ketogenesis, (2) ketogenesis but not β-oxidation pathway, (3) both the β-oxidation pathway and ketogenesis or (4) neither the β-oxidation pathway nor ketogenesis has to be determined.
Concept introduction:
Ketogenesis occurs in the mitochondria of the liver cells. The initial reactants are the molecules of acetyl CoA which are produced by the β-oxidation of fatty acid molecules. Ketone bodies are generally produced when the amount of acetyl CoA in the body is much larger than the amount of oxaloacetate. This happens due to the carbohydrate-lipid imbalance in the body caused by much smaller intake of food rich in carbohydrates, or inefficient processing of glucose by the body.
β-oxidation is a catabolic process occurring in the body through which, fatty acid molecules are broken down in the mitochondria of the cells to generate energy. The process involves breaking down long fatty acid chains that have been converted to acyl CoA chains into smaller fatty acyl CoA chains. The fatty acid chain is broken down until the final acyl CoA chain that cannot be broken down any further. The end products of this metabolic process are acetyl CoA, FADH2 and NADH. These three molecules later enter the Krebs cycle where they are used to produce ATP molecules.
Want to see the full answer?
Check out a sample textbook solutionChapter 25 Solutions
EBK GENERAL, ORGANIC, AND BIOLOGICAL CH
- UARDIAN SIGNA Life Sciences/ Baseline Test Grade 10 ry must be written in point form. pot in full sentences using NO MORE than 70 words sentences from 1 to 7. only ONE point per sentence. words as far as possible. number of words you have used in brackets at the end GDE/2024 QUESTION 3 The table below shows the results of an investigation in which the effect of temperature and light on the yield of tomatoes in two greenhouses on a farm was investigated. TEMPERATURE (°C) AVERAGE YIELD OF TOMATOES PER 3.1 PLANT (kg) LOW LIGHT LEVELS HIGH LIGHT LEVELS 5 0,5 0,5 10 1,5 2,5 15 3,0 5,0 20 3,6 8,5 25 3,5 7,8 30 2,5 6,2 State TWO steps the investigator may have taken into consideration during the planning stage of the investigation. (2) 3.2 Identify the: a) Independent variables (2) b) Dependent variable (1) 3.3 Plot a line graph showing the results of the average yield of the tomatoes from 5°C to 30°C for low light levels. (6) 3.4 State ONE way in which the scientists could have improved the…arrow_forwardExplain why you chose this mutation. Begin by transcribing and translating BOTH the normal and abnormal DNA sequences. The genetic code below is for your reference. SECOND BASE OF CODON כ FIRST BASE OF CODON O THIRD BASE OF CODON SCAGUCAGUGAGUCAG UUU UUC UCU UAU UGU Phenylalanine (F) Tyrosine (Y) Cysteine (C) UCC UAC UGC Serine (S) UUA UUG Leucine (L) UCA UCG_ UAA UGA Stop codon -Stop codon UAG UGG -Tryptophan (W) CUU CUC CCU CAU CGU Histidine (H) CCC CAC CGC -Leucine (L) Proline (P) CUA CCA CAA CUG CCG CAG-Glutamine (Q) -Arginine (R) CGA CGG AUU ACU AAU AGU AUC Isoleucine (1) Asparagine (N) ACC AAC Threonine (T) AUA ACA AAA Methionine (M) Lysine (K) AUG ACG Start codon AAG AGC-Serine (S) -Arginine (R) AGA AGG GUU GCU GAU GUC GUA GUG GCC Valine (V) -Alanine (A) GCA GCG GAC GAA GAG Aspartic acid (D) GGU Glutamic acid (E) GGC GGA GGG Glycine (G) In order to provide a complete answer to the question stated above, fill in the mRNA bases and amino acid sequences by using the Genetic Code…arrow_forwardidentify the indicated cell in white arrowarrow_forward
- Gloeocaspa Genus - diagram a colony and label the sheath, cell wall, and cytoplasm. Oscillatoria Genus - Diagram a trichome, and label the shealth and individual cells Nostoc Genus- diagram a sketch of the colonoy microscopically from low power to the left of the drawing. Draw a filament showing intercalary heterocysts, and vegatative cells to the right of the drawing Merismopedia Genus- diagram a sketch of the colony. draw and label a filament showing the colony, cell wall, and sheath. Gloeotrichia Genus- diagram a habit sketch of the colony. draw a filament showing the heterocyst, akimetes and vegatative cells of the filamentarrow_forwardOf this list shown, which genus does the image belong toarrow_forwardidentify the cell shownarrow_forward
- identify the genusarrow_forwardWhat Genus is this?arrow_forwardAs a medical professional, it is important to be able to discuss how genetic processes such as translation regulation can directly affect patients. Think about some situations that might involve translation regulation. Respond to the following in a minimum of 175 words: Why is translation regulation important? What are some examples of translation regulation in humans? Select one of the examples you provided and explain what happens when translation regulation goes wrong.arrow_forward
- The metabolic pathway below is used for the production of the purine nucleotides adenosine monophosphate (AMP) and guanosine monophosphate (GMP) in eukaryotic cells. Assume each arrow represents a reaction catalyzed by a different enzyme. Using the principles of feedback inhibition, propose a regulatory scheme for this pathway that ensures an adequate supply of both AMP and GMP, and prevents the buildup of Intermediates A through G when supplies of both AMP and GMP are adequate.arrow_forwardQUESTION 27 Label the structures marked A, B, C and explain the role of structure A. W plasma membrane For the toolbar, press ALT+F10 (PC) or ALT+FN+F10 (Mac). BIUS ☐ Paragraph Π " ΩΘΗ Β Open Sans, a... 10pt EEarrow_forwardexamples of synamptomorphyarrow_forward
- BiochemistryBiochemistryISBN:9781305577206Author:Reginald H. Garrett, Charles M. GrishamPublisher:Cengage LearningHuman Heredity: Principles and Issues (MindTap Co...BiologyISBN:9781305251052Author:Michael CummingsPublisher:Cengage Learning
- Anatomy & PhysiologyBiologyISBN:9781938168130Author:Kelly A. Young, James A. Wise, Peter DeSaix, Dean H. Kruse, Brandon Poe, Eddie Johnson, Jody E. Johnson, Oksana Korol, J. Gordon Betts, Mark WomblePublisher:OpenStax CollegeHuman Physiology: From Cells to Systems (MindTap ...BiologyISBN:9781285866932Author:Lauralee SherwoodPublisher:Cengage Learning