Biology 2e
2nd Edition
ISBN: 9781947172517
Author: Matthew Douglas, Jung Choi, Mary Ann Clark
Publisher: OpenStax
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Chapter 21, Problem 15RQ
People with the CCR5?32 mutation of a T-cell surface protein can be exposed to some strains of
HIV-1 without becoming sick. What step of the virus life cycle is likely to be inhibited with this mutation?
- Release
- Reverse transcription
- Uncoating
- Attachment
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Which form of HIV RNA, fully spliced, incompletely spiced, or unspliced, is needed during the early stage of the synthesis of HIV components?
Viral DNA synthesis cannot take place in a resting cell. How is this problem solved?
Which one is correct?
Early viral proteins like T antigen and E1A inactivate Rb protein
Viral late promoters are activated by DNA replication
DNA polymerase is encoded in the viral genome
Viral DNA genomes have multiple origins of replication
Chapter 21 Solutions
Biology 2e
Ch. 21 - Figure 21.5 Which of the following statements...Ch. 21 - Figure 21.8 Influenza virus is packaged in a viral...Ch. 21 - Figure 21.10 Which of the following statements is...Ch. 21 - Which statement is true? A virion contains DNA and...Ch. 21 - The viral______play(s) a role in attaching a...Ch. 21 - Viruses. all have a round shape cannot have a long...Ch. 21 - The observation that the bacteria genus Chlamydia...Ch. 21 - A scientist discovers a new virus with a linear,...Ch. 21 - Which statement is not true of viral replication?...Ch. 21 - Which statement is true of viral replication? In...
Ch. 21 - Which statement is true of reverse transcriptase?...Ch. 21 - Oncogenic virus cores can be. RNA DNA neither RNA...Ch. 21 - Which is true of DNA viruses? They use the host...Ch. 21 - A bacteriophage can infect the lungs viruses...Ch. 21 - People with the CCR5?32 mutation of a T-cell...Ch. 21 - An apple grower notices that several of his apple...Ch. 21 - Which of the following is NOT used to treat active...Ch. 21 - Vaccines. are similar to viroids are only needed...Ch. 21 - A patient presents at the clinic with an acute...Ch. 21 - Which of the following is not associated with...Ch. 21 - Which statement is true of viroids? They are...Ch. 21 - The first electron micrograph of a virus (tobacco...Ch. 21 - Varicella-zoster virus is a double-stranded DNA...Ch. 21 - Classify the Rabies virus (a rhabdovirus family...Ch. 21 - Why can’t dogs catch the measles?Ch. 21 - One of the first and most important targets for...Ch. 21 - In this section, you were introduced to different...Ch. 21 - Although plant viruses cannot infect humans, what...Ch. 21 - A bacteriophage with a lytic life cycle develops a...Ch. 21 - Why is immunization after being bitten by a rabid...Ch. 21 - The vaccine Gardasil that targets human papilloma...Ch. 21 - Prions are responsible for variant Creutzfeldt...Ch. 21 - How are viroids like viruses?Ch. 21 - A botanist notices that a tomato plant looks...
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- 31arrow_forwardss (-) RNA viral genome RNA polymerase RNA-dependent # O dsRNA O dsDNA. RNA-dependent RNA polymerase Coples (-) RNA strand transcription Copies (-) RNA strand. 80 ss (+) RNA S5 FA viral translation protein (+)RNA (viral mRNA) RNA-dependent RNA polymerase Copies (+) RNA strand. What type of virus is replicating in the picture? (-)ssRNA (+)ssRNA % C wil ss (-) RNA viral genome <(C 16 & 7arrow_forwardAdapting to Host Defenses Surface proteins called HLAs allow white blood cells to detect HIV particles and fight an infection. In a recent study, scientists tested whether HIV is adapting to this host defense. They did so by looking at the frequency of a specific mutation (1135X) in HIV. This escape mutation helps the virus avoid detection by a version of the HLA protein (HLA-B51) that is common in some regions of the world, but not in others. FIGURE 20.19 shows the percentage of HIV-positive people who had HIV with the 1135X mutation. Data were collected at medical centers from several parts of the world. FIGURE 20.19 Regional variation in the frequency of the 1135X escape mutation among HIV-positive people. For each region, pink bars represent the percentage of people whose blood cells have HLA-B51. and thus cannot detect 1135X mutants. Blue bars represent the percentage of people with other versions of the HLA protein. These people have blood cells that can detect and fight HIV even if it has the 1135X mutation. 4. Japan has a high frequency of HLA-B51; about half the population has it. How might this explain the high frequency of the 1135X mutation in Japanese with other HLAs?arrow_forward
- Adapting to Host Defenses Surface proteins called HLAs allow white blood cells to detect HIV particles and fight an infection. In a recent study, scientists tested whether HIV is adapting to this host defense. They did so by looking at the frequency of a specific mutation (1135X) in HIV. This escape mutation helps the virus avoid detection by a version of the HLA protein (HLA-B51) that is common in some regions of the world, but not in others. FIGURE 20.19 shows the percentage of HIV-positive people who had HIV with the 1135X mutation. Data were collected at medical centers from several parts of the world. FIGURE 20.19 Regional variation in the frequency of the 1135X escape mutation among HIV-positive people. For each region, pink bars represent the percentage of people whose blood cells have HLA-B51. and thus cannot detect 1135X mutants. Blue bars represent the percentage of people with other versions of the HLA protein. These people have blood cells that can detect and fight HIV even if it has the 1135X mutation. 1. What percentage of people with H LA-B51 in Vancouver had HIV with the escape mutation for this protein?arrow_forwardViruses. all have a round shape cannot have a long shape do not maintain any shape vary in shapearrow_forwardFigure 21.10 Which of the following statements is false? In the lytic cycle, new phages are produced and released into the environment. In the lysogenic cycle, phage DNA is incorporated into the host genome. An environmental stressor can cause the phage to initiate the lysogenic cycle. Cell lysis only occurs in the lytic cycle.arrow_forward
- Please help explain this figurearrow_forwardWould you be able to help with this? Number 1. I’ve watched the video and can’t figure it out :/arrow_forwardWhich of the following is an example of virus-encoded molecules modifying signal transduction pathways to promote replication? Production of a small subgenomic RNA in flavivirus-infected cells Degradation of Xrn1 in poliovirus-infected cells Herpes simplex virus protein that blocks pre-mRNA splicing Poliovirus cleavage of nuclear export proteinsarrow_forward
- Viruses can easily mutate exchange genetic material, mechanisms referred to as antigenic drift and antigenic shift. These changes allow the virus to evade the immune system defenses. Match each term with the correct definition Antigenic drift [ Choose ] [Choose ] Different strains of a virus or strain of different viruses, combine and form a new strain that now has a mixture of the surface antigens Antigenic shift The virus changes genetically by accumulating mutations within the genes that code for antibody-binding sitesarrow_forwardViral DNA synthesis cannot take place in a resting cell. How is this problem solved? Group of answer choices Early viral proteins like T antigen and E1A inactivate Rb protein Viral late promoters are activated by DNA replication DNA polymerase is encoded in the viral genome VIral DNA genomes have multiple origins of replicationarrow_forwardThe omicron variant of SARS-CoV-2 has the amino acid substitution G339D in the spike protein on the virus surface. In cellular fluids at -pH 7 the wild type spike protein has a net negative charge. This substitution will make the spike protein: less negative absorb UV light more strongly at 280 nm more negative more hydrophobicarrow_forward
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