To determine: The way in which Rh incompatibility is involved in the hemolytic disease of a newborn.
Introduction: There are two types of Rh blood groups: Rh+ (have Rh antigen) and Rh- (do not have Rh antigen). Rh factor is a point of concern when the mother has Rh- and the fetus has Rh+ blood group. This condition can cause hemolytic disease of a newborn.
To determine: The Rh genotype of the mother in case of Rh incompatibility.
Introduction: There are two types of Rh blood groups: Rh+ (have Rh antigen) and Rh- (do not have Rh antigen). Rh factor is a point of concern when the mother has Rh- and the fetus has Rh+ blood group. This condition can cause hemolytic disease of a newborn.
To determine: The Rh genotype of the fetus in case of Rh incompatibility.
Introduction: There are two types of Rh blood groups: Rh+ (have Rh antigen) and Rh- (do not have Rh antigen). Rh factor is a point of concern when the mother has Rh- and the fetus has Rh+ blood group. This condition can cause hemolytic disease of the newborn.
To determine: The reason for the second child that has Rh+ antigen would be more susceptible to the attack from the mother’s immune system.
Introduction: There are two types of Rh blood groups: Rh+ (have Rh antigen) and Rh- (do not have Rh antigen). Rh factor is a point of concern when the mother has Rh- and the fetus has Rh+ blood group. This condition can cause hemolytic disease of the newborn.
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Chapter 17 Solutions
Human Heredity: Principles and Issues (MindTap Course List)
- Blood Types Are Determined by Cell-Surface Antigens Is it more important that transfused blood have antigens that will not react with the recipients antibodies, or antibodies that will not react with the recipients antigens?arrow_forwardWhy is it that Rh incompatibility can be a serious problem when anRh-negative mother is carrying an Rh-positive fetus, but ABOincompatibility between mother and fetus is usually no problem?That is, a type A mother can usually safely carry a type B fetus.(Hint: The antibodies produced by an Rh-negative mother againstthe Rh antigen are usually IgG, whereas the antibodies producedagainst the A or B antigens are IgM.) Because the Rh antigenobviously serves no vital function (most humans lack it), why do youthink it hasn’t been completely eliminated during human evolution?arrow_forwardWhy might erythroblastosis fetalis occur when an Rh- mother becomes pregnant with a second Rh+ baby (after exposure to the previous Rh+ baby's blood)? A) Erythroblastosis fetalis can only occur when an Rh+ mother becomes pregnant with an Rh- baby. B) After primary exposure, if the Rh- mother has an Rh+ baby, then antibodies the mom produces can cross the placenta and attack the baby's blood. C) The Rh- mother always produces antibodies to the Rh+ blood, so erythroblastosis fetalis is a condition that can happen to any Rh+ baby (first or subsequent).arrow_forward
- What Rh antigen is present in the red blood cells of this type of blood?arrow_forwardWhat will happen if an Rh-negative person receives a transfusion of Rh-positive blood? If a patient will receive a blood transfusion that is not matched to the patient’s blood type, what will happen? Explain briefly.arrow_forwardA woman who is Rh- mates with a man who is Rh+. Since the Rh+ is dominant, what would the fetus's Rh factor type be?A person who is Rh- can donate blood to other people who are either Rh+ or Rh-. However, an Rh+ person can only give blood to who? Please explain.arrow_forward
- If the thymus of an adult experimental animal is removed, the following observations can be made: (a) No immediate effect occurs, and (b) after 1 year, decreases occur in the number of lymphocytes in the blood, the ability to reject grafts, and the ability to produce antibodies. Explain these observations.arrow_forwardlist all of the blood types to which the following people could donate assuming the recipients have been exposed to Rh antigens: Donor 1 :type A Donor 2:type O+ Donor3:type AB+ Donor 4: type B-arrow_forwardExplain the differences between the development of anti-Rh antibodies and the development of anti-A and anti-B antibodies?arrow_forward
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