ANAT.+PHYSIO.1-LAB.MAN. >CUSTOM<
ANAT.+PHYSIO.1-LAB.MAN. >CUSTOM<
20th Edition
ISBN: 9781264303106
Author: VanPutte
Publisher: MCGRAW-HILL HIGHER EDUCATION
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Chapter 17, Problem 1CT
Summary Introduction

To analyze:

The levels of the hormone in the blood of the person suffering from liver and kidney disease.

Introduction:

The endocrine system releases various hormones that function to regulate different physiological processes of the body. The hormones control vital processes like electrolyte balance, temperature regulation, and osmotic balance.

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Consider a hormone that is secreted in large amounts at a given interval, modified chemically by the liver, and excreted by the kidneys at arapid rate, thus making the half-life of the hormone in the circulatorysystem very short. The hormone therefore rapidly increases in theblood and then decreases rapidly. Suppose that a patient has both liverand kidney disease. Predict the consequences on the blood levels ofthat hormone.
The kidneys are important regulators of blood volume (and thus MAP) and the ionic composition and pH of plasma . The amount of water reabsorbed or lost is hormonally regulated. Both the amount of blood that is filtered (glomerular filtration rate) and the amount of water that is reabsorbed is determined by hormones binding with receptors. In order to understand why the level of any given hormone is elevated or low in the blood, it is necessary to think in terms of homeostasis loops for each hormone, and what the initial disturbance to homeostasis was. In other words “who started it”! What STEROID hormone is elevated by increased activation of the RAS pathway?
The kidneys are important regulators of blood volume (and thus MAP) and the ionic composition and pH of plasma . The amount of water reabsorbed or lost is hormonally regulated. Both the amount of blood that is filtered (glomerular filtration rate) and the amount of water that is reabsorbed is determined by hormones binding with receptors. In order to understand why the level of any given hormone is elevated or low in the blood, it is necessary to think in terms of homeostasis loops for each hormone, and what the initial disturbance to homeostasis was. In other words “who started it”! What NON STEROID hormone is produced at the end (but as part of) of the RAS pathway?

Chapter 17 Solutions

ANAT.+PHYSIO.1-LAB.MAN. >CUSTOM<

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