Fundamentals of Anatomy & Physiology (11th Edition)
11th Edition
ISBN: 9780134477336
Author: Martini
Publisher: PEARSON
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Chapter 16, Problem 23CP
Summary Introduction
To determine:
The consequences of a drug that substantially increases the amount of serotonin released in the brain.
Introduction:
Serotonin is a neurotransmitter derived from amino acid tryptophan. It regulates both the physiological and behavioral processes of the body. It mediates communication between neurons in the central nervous system. In addition to this, it also promotes the feeling of well-being and happiness in humans. The deficiency of serotonin in the brain results in depression and anxiety.
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Chapter 16 Solutions
Fundamentals of Anatomy & Physiology (11th Edition)
Ch. 16 - Prob. 1CPCh. 16 - Prob. 2CPCh. 16 - Prob. 3CPCh. 16 - Prob. 4CPCh. 16 - Prob. 5CPCh. 16 - Prob. 6CPCh. 16 - Prob. 7CPCh. 16 - Prob. 8CPCh. 16 - Prob. 9CPCh. 16 - Prob. 10CP
Ch. 16 - Name the two types of cholinergic receptors on the...Ch. 16 - Prob. 12CPCh. 16 - Prob. 13CPCh. 16 - Prob. 14CPCh. 16 - Prob. 15CPCh. 16 - Prob. 16CPCh. 16 - Prob. 17CPCh. 16 - Prob. 18CPCh. 16 - Prob. 19CPCh. 16 - Prob. 20CPCh. 16 - Prob. 21CPCh. 16 - Prob. 22CPCh. 16 - Prob. 23CPCh. 16 - Prob. 24CPCh. 16 - Prob. 25CPCh. 16 - Prob. 26CPCh. 16 - Prob. 27CPCh. 16 - Prob. 28CPCh. 16 - Prob. 1RQCh. 16 - Prob. 2RQCh. 16 - Prob. 3RQCh. 16 - Prob. 4RQCh. 16 - Prob. 5RQCh. 16 - Prob. 6RQCh. 16 - Prob. 7RQCh. 16 - What are the components of a visceral reflex arc?Ch. 16 - Prob. 9RQCh. 16 - Prob. 10RQCh. 16 - Prob. 11RQCh. 16 - All preganglionic autonomic fibers release _____...Ch. 16 - Prob. 13RQCh. 16 - Prob. 14RQCh. 16 - Prob. 15RQCh. 16 - Prob. 16RQCh. 16 - Prob. 17RQCh. 16 - Prob. 18RQCh. 16 - Prob. 19RQCh. 16 - Prob. 20RQCh. 16 - Prob. 21RQCh. 16 - Prob. 22RQCh. 16 - Prob. 23RQCh. 16 - Prob. 24RQCh. 16 - Prob. 25RQCh. 16 - Prob. 26RQCh. 16 - Prob. 27RQCh. 16 - Prob. 28RQCh. 16 - Prob. 29RQCh. 16 - Prob. 30RQCh. 16 - Prob. 31RQCh. 16 - Prob. 1CCCh. 16 - Prob. 2CCCh. 16 - Prob. 3CC
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Need a deep-dive on the concept behind this application? Look no further. Learn more about this topic, biology and related others by exploring similar questions and additional content below.Similar questions
- Reserpine is a drug that can control high blood pressure by reducing the number of catecholamine neurotransmitters present in the synapse. Epinephrine, norepinephrine, and dopamine are examples of catecholamine neurotransmitters. One of the known side effects of reserpine is to cause the symptoms of Parkinson’s disease. Parkinson's disease is associated with dopamine. Parkinson's disease occurs when the nerve cells in the part of the brain that controls muscle movement are gradually destroyed and the neurons can no longer produce dopamine to coordinate muscle movements. Reserpine causes symptoms by a. inhibiting the release of dopamine from the presynaptic neuron b. blocking the dopamine receptor in the postsynaptic neuron c. breaking down the neurotransmitter acetylcholine in the synapse d. breaking down cholinesterase enzyme in the synapsearrow_forwardWhat would happen if there is an increase of acetylcholine and serotonin receptor activation in the body?arrow_forwardCocaine blocks the removal of dopamine from the synapse, leading to a rapid rise of dopamine in the synapse. Which of the following best describes what will likely occur as a result of increased dopamine in the synapse? The presynaptic cell will be unable to release more neurotransmitter. the postsynaptic cell will return to its resting potential, causing feelings of well-being. The postsynaptic cell will be constantly activated, causing feelings of euphoria. The postsynaptic cell will respond more quickly to a change in output of the presynaptic cell, causing feelings of euphoria. The buildup of neurotransmitter will activate the presynaptic cell, causing feelings of euphoria.arrow_forward
- In some cases, people who suffer from depression can be prescribed a serotonin re-uptake inhibitor if they are determined to be resistant to behavioral treatment. Explain how a serotonin re-uptake inhibitor would alter concentrations of serotonin in the brain. Would this change occur at all synapses within the brain? Why or why not?arrow_forwardWhy might drugs that work upon dopamine receptors might also work upon norepinephrine receptors, as well? What concept does this illustrate?arrow_forwardThe following diagram represents a typical serotonergic synapse. Where, specifically, do antidepressants work (e.g. SSRI)? Neurotransmitter Neurotransmitter transporter Аxon Synaptic vesicle terminal Voltage- gated Ca?+ channel Synaptic cleft Receptor Postsynaptic density Dendrite Neurotransmitter Synaptic Vesicle Neurotransmitter transporter (aka Reuptake transporter) Receptor O All of the abovearrow_forward
- What do you mean by effectors?arrow_forwardIf we give a patient an SSRI, then what would we expect to happen to the response of the postsynaptic cells that express serotonin receptors?arrow_forwardStudies have shown that individuals with attention deficit hyperactivity disorder (ADHD) may have lower than normal amounts of dopamine and norepinephrine excitatory neurotransmitters in their synapses. Methylphenidate is one of the drugs prescribed to individuals with ADHD. It prevents the reuptake of dopamine and norepinephrine neurotransmitters from the synapses, thereby keeping these neurotransmitters in the synapses longer. Which of the following rows correctly identify the effects of methylphenidate in individuals with ADHD? Select one: a. There is an increase in the permeability of the presynaptic neuron to potassium ions. b. There is a increase in the permeability of the postsynaptic neuron to potassium ions. c. There is an increase in the permeability of the presynaptic neuron to sodium ions. d. There is a increase in the permeability of the postsynaptic neuron to sodium ions.arrow_forward
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