Which of the following drugswould directly target the ability ofcancer cells to evade cell growthsuppressors? (evading the growthsuppressor hallmark)Anti-inflammatory drugsVEGF signalinginhibitorsCyclin-dependent kinaseinhibitorsTelomerase inhibitors

What is VEGF? Many cells generate vascular endothelial growth factor (VEGF), formerly known as…

Answered: would directly target the ability of…

Phlebotomy Essentials

6th Edition

ISBN:9781451194524

Author:Ruth McCall, Cathee M. Tankersley MT(ASCP)

Publisher:Ruth McCall, Cathee M. Tankersley MT(ASCP)

Chapter1: Phlebotomy: Past And Present And The Healthcare Setting

Section: Chapter Questions

Problem 1SRQ

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Similar questions

Explain what it means when the developmental steps for cancer are “not obligatory” and give one example
Why is the p53-meditated apoptosis pathway, and the GSK3 signaling pathway important for cancer therapy. Explain each of them in 3-5 sentences.
please help
Which of the following are charcteristics you would expect of a circulating tumor cell that has undergone Epithelial-Mesenchymal Transition (select all that apply)? A. Increased N-cahedrin expression B. Increased resistance to apoptosis C. Loss of ability to secrete fibronectin D. High cytokeratin expression E. Elevated PDGF Receptor Expression
Discuss how growth factors influence cell signaling and proliferation that leads to cancer induction, progression, and metastasis. Be sure to include the involvement of apoptosis and angiogenesis.
Briefly describe how inflammation can contribute to carcinogenesis and disease progression through heterotypic signaling.
how is evading growth receptors necessary for cancer development and how is it driving cancer formation. in 3-6 sentences.
Explain in detail two different genetic mutations that can promote the development of cancer. Include the name (or type) of gene mutated and whether the expression of the gene is upregulated or downregulated. Provide a description of the role the gene plays in regulating the cell cycle and how the cell is affected when the gene no longer produces a functional protein
Explain why each of the following is a risk factor for cancer: age, loss-of-function mutations in repair or tumor-suppressor genes, certain viruses such as HPV, chemical mutagens, radiation/UV light
In your own words, explain how cancer cells differ from normal cells in regard to the following: Molecular controls of the cell cycle (include Cdk and Cdk/cyclin complexes, p53 gene/protein, Rb gene/protein in response)
Please answer it asap
TNF-alpha treatment of prostate carcinoma, LNCAP cells decreases cell survival as shown in the graph below. Which of the following would you observe in these cells treated with TNF- alpha? Select all that apply TNF-a 120 100 80 - 60- 40- 20 - 24 72 (hrs) + Control - TNF-a 10ng + TNF-a 100 ng O Activation of extrinsic pathway of apoptosis O Activation of intrinsic pathway of apoptosis Activation of executioner caspases Recruitment of adapter protein FADD to the TNF-alpha receptor LNCAP cell viability (% of control)

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