Post-synaptic receptors located on the membrane of skeletal muscle cells allowthe muscle cells/fibers to respond to the neural-stimulus. What is the name of thereceptors located on the post-synaptic membrane of the muscle cells (you can’t findthe answer in the textbook; you have to do some research)?A. beta receptorsB. alpha receptorsC. muscarinic receptorsD. nicotinic receptors

Post-synaptic receptors located on the membrane of skeletal muscle cells allowthe muscle cells/fibers to respond to the neural-stimulus. What is the name of thereceptors located on the post-synaptic membrane of the muscle cells (you can’t findthe answer in the textbook; you have to do some research)?A. beta receptorsB. alpha receptorsC. muscarinic receptorsD. nicotinic receptors

Basic Clinical Lab Competencies for Respiratory Care: An Integrated Approach

5th Edition

ISBN:9781285244662

Author:White

Publisher:White

Chapter14: Introduction To Respiratory Care Pharmacology

Section: Chapter Questions

Problem 3SEPT

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Local anesthetics "block" the action potential and therefore do not allow "pain" information to proceed to the brain and spinal cord. In the presence of these molecules (local anesthetics) 'threshold' is essentially "ignored". The molecules most logically work by: 23. a. Decreasing the intracellular [potassium] b. Blocking the opening of the voltage-gated potassium channels c. Decreasing the extracellular [sodium] d. Blocking the opening of the voltage-gated sodium channels
In the stretch reflex,a. Golgi tendon organs activate contraction in extrafusal muscle fibersconnected to that tendon.b. lengthening of muscle-spindle receptors in a muscle leads tocontraction in an antagonist muscle.c. action potentials from muscle-spindle receptors in a muscle formmonosynaptic excitatory synapses on motor neurons to extrafusal fiberswithin the same muscles.d. slackening of intrafusal fibers within a muscle activates gamma motorneurons that form excitatory synapses with extrafusal fibers within thatsame muscle.e. afferent neurons to the sensorimotor cortex stimulate the agonist muscle to contract and the antagonist muscle to be inhibited
Which of the following produces EPSP? A. GABA acting on GABAA receptors OB. GABA acting on GABAC receptors C. GABA acting on NMDA receptors D. Glutamate acting on AMPA receptors OE. Kainate acting on GABAB receptors
T O b. Regulate the flow of somatosensory information to the brain O c. Enhance the antigravity reflexes of spinal cord and help in standing posture O d. Liberate the antigravity muscles from the reflex control of the spinal cord QUESTION 14 What is the function of the presynaptic "autoreceptors" in axon terminals? a. To release substances into the blood Ob. To detect transmitter concentrations in the cleft Oc. To release neurotransmitters into extracellular fluid Od. To release the neurotransmitter acetylcholine esc QUESTION 15 What distinguishes the ANS from the somatic motor system? a. The ANS excites its peripheral targets. Ob. The ANS balances synaptic excitation and inhibition. c. The ANS has a single function. Od. The ANS constitutes total neural output. Click Save and Submit to save and submit. Click Save All Answers to save all answers. Type here to search f1 7 ? f2 C f3 # 3 A O +&+ 4 10 f5 % LO 5 f6 D 10 A 6 f7 & 7
In the disease myasthenia gravis, the human body makes—by mistake— antibodies to its own acetylcholine receptor molecules. These antibodies bind to and inactivate acetylcholine receptors on the plasma membrane of muscle cells. The disease leads to a devastating progressive weakening of the people affected. early on, they may have difficulty opening their eyelids, for example, and, in an animal model of the disease, rabbits have difficulty holding their ears up. As the disease progresses, most muscles weaken, and people with myasthenia gravis have difficulty speaking and swallowing. eventually, impaired breathing can cause death. explain which step of muscle function is affected.
At a normal NMJ, action potentials in the skeletal muscle cell can be blocked directly by? A. Curare, which inhibits nicotinic receptors. B. Atropine, which inhibits muscarinic receptors. C. Physostigmine, which inhibits ACHE. D. AP5, which inhibits NMDA receptors. E. Pentobarbital, which enhances the function of GABA on GABAA receptors.
Students in a veterinary school are given the following hypotheticalproblem: A dog ingests organophosphate poison, and the students areresponsible for saving the animal’s life. Organophosphate poisons bindto and inhibit acetylcholinesterase. Several substances the studentscould inject include the following: acetylcholine, curare (which blocksacetylcholine receptors), and potassium chloride. If you were a studentin the class, what would you advise to save the animal?
Which of the following produces IPSP? A. GABA acting on GABAA receptors B. Glutamate acting on AMPA receptors C. Glutamate acting on kainate receptors D. Glutamate acting on NMDA receptors E. Acetylcholine acting on nicotinic receptors
Which structure transmits impulses towards the nerve cell body? Select one: a. Dendrite b. Axon C. None is correct d. Axon hillock e. Synaptic knobs
Sarin nerve gas paralyzes muscle by interfering with neuronal transmission by which of the following? Select one: O A. encephalin O B. serotonin O C. acetylcholine O D. glutamate O E. GABA
Nicotinic receptors are located on which of the following? a. plasma membranes of ganglionic neurons b. target cells that receive parasympathetic innervation c. blood vessels in skeletal muscles d. sweat glands
Which structure transmits impulses towards the nerve cell body? Select one: O a. Dendrite b. Axon None is correct O d. Axon hillock e. Synaptic knobs