In response to extreme starvation, some bacterial cells undergo programmed cell death (PCD). In E. coli, PCD is mediated by the toxin-antitoxin pair MazF (the toxin that initiates PCD) and MazE (the antitoxin that keeps MazF in check). PCD lowers the fitness of the bacterial cell, but if the cells in the population are genetically related, then the sacrifice of some cells to keep a subset of the population alive may increase the fitness of that shared genome. There is, however, the risk of mutant "cheaters," cells that do not undergo PCD in response to extreme stress but benefit from the nutrients released from dead cells. Loss of which proteins could result in a cheater cell phenotype? Choose one or more: OA. MazF OB. CIPAP protease OC. glucose 6-phosphate dehydrogenase (G6PD) OD. MazE
In response to extreme starvation, some bacterial cells undergo programmed cell death (PCD). In E. coli, PCD is mediated by the toxin-antitoxin pair MazF (the toxin that initiates PCD) and MazE (the antitoxin that keeps MazF in check). PCD lowers the fitness of the bacterial cell, but if the cells in the population are genetically related, then the sacrifice of some cells to keep a subset of the population alive may increase the fitness of that shared genome. There is, however, the risk of mutant "cheaters," cells that do not undergo PCD in response to extreme stress but benefit from the nutrients released from dead cells. Loss of which proteins could result in a cheater cell phenotype? Choose one or more: OA. MazF OB. CIPAP protease OC. glucose 6-phosphate dehydrogenase (G6PD) OD. MazE
Human Anatomy & Physiology (11th Edition)
11th Edition
ISBN:9780134580999
Author:Elaine N. Marieb, Katja N. Hoehn
Publisher:Elaine N. Marieb, Katja N. Hoehn
Chapter1: The Human Body: An Orientation
Section: Chapter Questions
Problem 1RQ: The correct sequence of levels forming the structural hierarchy is A. (a) organ, organ system,...
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Transcribed Image Text:In response to extreme starvation, some bacterial cells undergo programmed cell
death (PCD). In E. coli, PCD is mediated by the toxin-antitoxin pair MazF (the toxin that
initiates PCD) and MazE (the antitoxin that keeps MazF in check). PCD lowers the
fitness of the bacterial cell, but if the cells in the population are genetically related,
then the sacrifice of some cells to keep a subset of the population alive may increase
the fitness of that shared genome. There is, however, the risk of mutant "cheaters," cells
that do not undergo PCD in response to extreme stress but benefit from the nutrients
released from dead cells. Loss of which proteins could result in a cheater cell
phenotype?
Choose one or more:
OA. MazF
OB. CIPAP protease
OC. glucose 6-phosphate dehydrogenase (G6PD)
OD. MazE
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