4. A sportsman is doing a 10 km marathon for 1.5 hours. What changes in lipidmetabolism in adipose tissue and muscles are triggered by long-term physicalactivity? To answer the question:a) name the hormone, controlling the lipid metabolism in adipocytes and writedown the scheme of the process;b) explain, how some of the products of lipid mobilization, transported fromadipocytes to the bloodstream, would be utilized by skeletal muscles and liverin these conditions;c) calculate the ATP yicld in the result of palmitolcic acid oxidation.

The breakdown or storage of fats for energy, as well as the production of structural and functional…

Answered: activity? To answer the question: a)…

Biochemistry

9th Edition

ISBN:9781319114671

Author:Lubert Stryer, Jeremy M. Berg, John L. Tymoczko, Gregory J. Gatto Jr.

Publisher:Lubert Stryer, Jeremy M. Berg, John L. Tymoczko, Gregory J. Gatto Jr.

Chapter1: Biochemistry: An Evolving Science

Section: Chapter Questions

Problem 1P

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ATP Accounting Upon digestion of starch, maltose, one of its degradation products, is further hydrolyzed into its monosaccharide components prior to intestinal absorption and entry into the glycolysis. Calculate the number of ATP molecules produced from the digestion and complete oxidation of 3 molecules of maltose considering the malate-aspartate shuttle. Answer the following items using numerical value only (e.g. 1, not "1 ATP") which will help you arrive at the final answer for this question. a. Total number of glucose molecules entering glycolysis: b. Total number of pyruvate molecules produced at the end of glycolysis: c. Total number of mitochondrial NADH produced after pyruvate is acted upon by the pyruvate dehydrogenase complex: d. Total number of CO2 released right after the pyruvate dehydrogenase complex reaction: e. Total number of acetyl CoA molecules entering the citric acid cycle: f. Total number of net cytosolic ATP molecules produced right after glycolysis: g. Total…
Give Detailed Solution with explanation...(no need Handwritten)
Considering the complete oxidation of an 18-C fatty acid. Give the answer to the following question.a. What is the total number of NADH produced in TCA if all of the acetyl CoA enters the cycle? b. What is the total number of FADH2 produced in TCA if all the acetyl CoA enters the cycle?c. How much ATP is produced in the overall oxidation?
Back in the 1930s, a chemical called DNP (2,4-Dinitrophenol) was prescribed as a weight loss pill. Describe how DNP is a metabolic poison , and why it was thought to be a good mechanism for weight loss?
- The following both link carbohydrate metabolism with lipid biosynthesis: (a) How many molecules of glucose are required to provide the carbon for the synthesis of one molecule of palmitate? (b) How many molecules of glucose are required if all of the glucose first proceeds through the pentose phosphate pathway before proceeding through the rest of glycolysis on its way to pyruvate?
please answer the following:
In response low density lipoprotein (LDL), cells elevate the activity of ATP-citrate lyase, which catalyzes the conversion of citrate to acetyl-CoA and oxaloacetate. TRUE or FALSE?
Calculate the energy(Kcal.) produced from full oxidation of (3) mole of glucose (glucose --> CO2+ H2O), Compare the energy yield with that produced from oxidation of (1) mole .of stearic acid (C18)
Glucagon The function of glucagon is in promoting energy release and inhibiting energy storage in response to low circulating glucose levels, which are largely controlled by insulin [24]. Besides its role in blood–glucose regulation, patients treated with glucagon receptor an tagonists (GRA) often suffer from dyslipidemia, fatty liver and weight gain, suggesting that glucagon play an important role in lipid metabolism [25]. Glucagon acts mainly on the hepatocytes which possess the highest level of glucagon receptors. Following mecha nisms may be involved during this process: (1) At the molecular level, following glucagon binding to its receptor on the hepatocyte, cAMP will be activated and accumulated, which in turn activates cAMP-response element-binding protein (CREB). As a result, the tran scription of carnitine acyl transferase (CPT-1) increases, which converts fatty acids into acylcarnitine and activates β-oxidation-increasing fatty acid catabolism [26]. In addition,…
Help me understand this…I’m confused
Discuss the metabolic control for HMG CoA reductase synthesis and activity
Indicate whether each of the following statements is true or false, and briefly explain your answer. (a) In general, the metabolic oxidation of protein in mammals is less ef- ficient, in terms of energy conserved, than the metabolic oxidation of carbohydrate or fat. (b) Given that the nitrogen of glutamate can be redistributed by transami- nation, glutamate should be a good supplement for nutritionally poor proteins. (c) Arginine is a nonessential amino acid for mammals because the enzymes of arginine synthesis are abundant in liver. (d) Alanine is an essential amino acid because it is a constituent of every protein.

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