6. You are interested in how the density of NMDA receptors in the postsynaptic membrane prior tostimulation will affect future LTP and LTD production. You prepare mouse hippocampal slices with threetypes of postsynaptic neurons - neurons that have either very high densities, very low densities, or zerodensity of NMDA receptors. You stimulate the neurons presynaptic to these different types ofpostsynaptic neurons using a brief, high-frequency stimulation protocol (100 Hz) or a prolonged,low-frequency stimulation protocol (10 Hz). You then record whether an LTP or LTD was produced. Matchthe different NMDA receptor densities in the first column with the expected LTP/LTD occurrences in thesecond column of the table below:NMDA Receptor/Channel Density BeforeResultsStimulation1. Very highA. No LTP during brief, high-frequencystimulation.No LTD during prolonged, low-frequency stimulation.2. No NMDA receptorsB. No LTP during brief, high-frequencystimulation.LTD during brief, high-frequencystimulation.3. Very lowC. No LTD during prolonged, low-frequency stimulation.LTP during prolonged, low-frequencystimulation.

NMDA is the N-Methyl-D-aspartic acid or it is also called N-Methyl-D-aspartate. It is an amino acid…

Answered: 6. You are interested in how the…

Human Anatomy & Physiology (11th Edition)

11th Edition

ISBN:9780134580999

Author:Elaine N. Marieb, Katja N. Hoehn

Publisher:Elaine N. Marieb, Katja N. Hoehn

Chapter1: The Human Body: An Orientation

Section: Chapter Questions

Problem 1RQ: The correct sequence of levels forming the structural hierarchy is A. (a) organ, organ system,...

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1. Now assume that you have a typical neuron that has very few Na+ channels open relative to the number of K+ channels. This is the condition of a neuron that is at rest. What will the resting membrane potential be? 2. Suppose a neuron’s resting potential is -70 mV. What will happen to the membrane potential if you could open more K+ channels open more Na+ channels
Physiology question
Name and describe the roles of key molecules and membrane structures in maintaining axon membrane resting potential of-70 mV Name and describe the roles of key molecules and membrane structures in propagation of axon membrane action potentials during depolarization at +35 mV Explain why they myelin sheath of white matter neurons allows faster action potential transmission than that of neurons found in gray matter.
neurotransmitters 1 In what way may drugs be used to affect neurotransmitters? Select an answer and submit. For keyboard navigation, use the up/down arrow keys to select an answer. A drug may prevent the binding of a neurotransmitterto a receptor. b, A drug may increase the amount of neurotransmitter released. A drug may prevent the release of aneurotransmitter, d. A drug may affect the degradation of a neurotransmitter Drugemay affect neurotranemitters usng al of the mechanismslsted. Unanswered
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87.
Describe the 6 step sequence of events that occurs at the NMJ in response to a nerve Action Potential.
You apply a drug to a batch of neurons and record membrane potential changes in the presynaptic and postsynaptic neurons. You record an action potential in the presynaptic neuron and a change from -70mV to -55mV in the postsynaptic neuron. Your drug most likely allowed which ion into the postsynaptic neuron? Sodium (Na+) Glutamate Potassium (K+) GABA Chloride (Cl-)
4.3. After the rising phase, which ion channel is responsible for action potential returning to its resting V and why? Na channels, because positive Na* ions are at higher concentration outside the axon than inside, and so they enter the axon and raise V O Na* channels, because positive Na* ions are at higher concentration inside the axon than outside, and so they leave the axon and raise V, m O K* channels, because positive K* ions are at higher concentration outside the axon than inside, and so they enter the axon and raise Vm U K* channels, because positive K* ions are at higher concentration inside the axon than outside, and so they leave the axon and lower Vm
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Calcium entry through NNDA receptor can potentiate synaptic transmission through the following mechanisms EXCEPT: a.) Insertion of AMPA receptors to postsynaptic membrane b.) Synthesis of NO to increase presynaptic transmitter release c.) Phosphorylation of AMPA receptors d.) Slowing down glutamate reupdate, so it remains longer in the synapse

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