he RNA-induced silencing complex takes dsRNA and a. Forms ssRNA that will target mRNA b. Exports it to the cytoplasm c. Chops it up into smaller dsRNA pieces d. Exports it to the nucleus
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he RNA-induced silencing complex takes dsRNA and
| a. |
Forms ssRNA that will target mRNA |
|
| b. |
Exports it to the cytoplasm |
|
| c. |
Chops it up into smaller dsRNA pieces |
|
| d. |
Exports it to the nucleus |
Step by step
Solved in 4 steps
- People with the CCR5Δ32 mutation of a T-cell surface protein can be exposed to some strains of HIV-1 without becoming sick. What step of the virus life cycle is likely to be inhibited with this mutation? a. Release b. Reverse transcription c. Uncoating d. AttachmentWhy are vaccines and/or passive immunization the method of choice in the treatment or prevention of viral infections? A. There are no known inhibitors to reverse transcription B. Viruses are non-living and do not have their own genetic mechanisms to reproduce by themselves hence host cells must rely on targeting the virus or their products. C. Antibodies prevent replication of viral genome D. Antibodies prevent transcription and translation of viral genomes E. All of theseWhich of these BEST DESCRIBE tryptophan in the Trp Operon? A. Acts as a corepressor B. Acts as a coactivator C. Acts as an inducer D. Acts as an enhancer
- Which of the following is NOT true of ITAMs? a. They are attached to the tails of the Ig b. Their phosphorylation eventually leads to a change in gene expression c. They send a signal to the interior of the cell d. ITAM is short for immunoreceptor tyrosine-based activation motifsDefine the following terms: a. cell transformation b. oncogene c. apoptosis d. early response gene e. delayed response geneEnzymes that have binding sites for modulators are specifically called a. zymogens b. quaternary proteins c. induced enzymes d. allosteric enzymes
- 39. HIV could be used to cure Kaposi’s sarcoma by a. causing the cells to express exogenous surface cell markers b. activating genes for the expression of endogenous surface cell markers c. inactivating the patient’s tumor suppressing genes d. inactivating contact inhibition in the cell e. inactivating cellular adherenceHow does a T-cell recognize a cell infected by a virus? Select one: a. binding of viral DNA in an infected cell. b. binding of viral peptide in the cytosol of infected cell c. binding of viral mRNA in the cytosol of the infected cell d. binding of viral peptide on the surface of the infected cell e. binding of viral mRNA on the surface of the infected cellHumans with defective expression of the integrin LFA-1 have an immunodeficiency disease characterized by the failure of lymphocytes and granulocytes to migrate to tissues at sites of infection or inflammation. A similar immunodeficiency would be expected if individuals had mutations disrupting the gene for: A. CD3 B. The complement receptor, CD21 C. WASp D. Rap1 E. SLP-76
- A study is performed in persons with an immunodeficiency disease in which activation of macrophages and B cells by CD4 cells is ineffective. It is observed that all affected persons are males. Laboratory studies show an elevated serum IgM but decreased IgG and IgA. This disorder is most likely to be caused by a mutation in a gene encoding for which of the following? a.CD40 ligand b.Adenine deaminase c.Cytochrome b558 d.Complement C3The HIV virus produces copies of its genome through: A. DNA replication B. Reverse transcription followed by integration then normal transcription C. RNA replication from an RNA template D. differential splicingRefer to the following illustration to answer the question. VI V2 V3 Rearranged DNA in B lymphocyte V250 VI Primary transcript V2 O transcription denaturation of DNA RNA splicing -20 M Immunoglobulin mRNA V3 13 translation V3 13 V3 13 11 J4 In the production of an immunoglobulin light-chain, the joining of the J3 segment with the C segment occurs through: 14 site-specific recombination of DNA C 12