taw a graph representing the changes in membrane potential across the axonal membranebefore, during, and after an action potential, On your graph, use a highlighter to clearly indicatefor which portions of your graph the letter corresponding to each cellular event listed belowbelongs.List of cellular events:A) All K* channels openB) All Nat channels closeC) The Na*/K* ATPase cotransporter transports Nat and K* across the membrane.D) All Na* channels openE) The threshold value of membrane potential is attained.F) All K* channels closeG) Some Nat channels openYour graph:o pecffecton

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Human Anatomy & Physiology (11th Edition)

11th Edition

ISBN:9780134580999

Author:Elaine N. Marieb, Katja N. Hoehn

Publisher:Elaine N. Marieb, Katja N. Hoehn

Chapter1: The Human Body: An Orientation

Section: Chapter Questions

Problem 1RQ: The correct sequence of levels forming the structural hierarchy is A. (a) organ, organ system,...

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Similar questions

What is the difference between a ligand-gated channel (like H1 or the AChR) and a voltage gated channel?
Place the following events in chronological order from 1-8: Nat enters the cell, and depolarization occurs to approximately +30 mV. The voltage across the cell membrane is -70 mV, the resting membrane potential. Upon reaching the peak of the action potential, the VG Nat channels are inactivated by the closing of their inactivation gate and the activation gate of each VG K channel opens. VG K channels close by the closing of their activation gate, and the resting membrane potential is gradually restored. An excitatory post-synaptic potential depolarizes the membrane to threshold and the activation gate of VG Nat channels open. Upon returning to the resting membrane potential, VG Na channels are reset by opening of the inactivation gate and the closing of the activation gate. VG K+ channels are slow to close, resulting in an excess of K* efflux and hyperpolarization. Depolarization occurs as K+ flows out of the cell.
Consider the following three diagrams of a nerve cell membrane. They show resting potential, depolarization, and hyperpolarization. Figure out which one is which, then draw them in the order they occur in a cell that undergoes an action potential outside + Na* inside K* Na* Nat K Nat K Na potential: -80 mV outside + Na K* Na* inside Na+ K Nat Na* K+ potential: +30 mV outside Na Na Na Na* K+ inside K* Na* Kt potential: -70 mV
3) Neurons cannot respond during the absolute refractory period because: a) the concentration gradients for Na+ and K+ are reversed. b) the cell is below its normal resting membrane potential. c) voltage-gated Na+ channel activation gates are closed. d) voltage-gated Na+ channel inactivation gates are closed.
A student is eating at the lab bench (a clear violation of lab policies) and mistakenly sprinkles tetrodotoxin on his fries. Explain the origin of the toxin and its normal function in that animal. Given that this substance inhibits voltage-gated sodium channels, indicate whether each of the statements below are true or false. Explain your answers. Explain the time course of the response. It will be more difficult for the student’s neurons to generate action potentials The student’s neurons will fire more frequently because membrane potential will be brough closer to threshold. The effect on the membrane potential o the student’s neurons could be predicted by the Nernst equation, which factors in the effects of both ion concentration and ion permeability.
a)The distribution of sodium ions across the cell membrane of a neuron is 20 mmol/L inside the cell and 200 mmol/L outside the cell. When the system reaches equilibrium, the concentration of sodium ions will be identical on both sides of the cell membrane. What is the standard Gibbs energy difference in the system? Assume the system is at body temperature (37 °C). b)What is the Gibbs energy difference across the membrane in the initial state (prior to reaching equilibrium)?
In a neuron at rest, there is a difference in the electrical charge across the cell membrane, with the inside of the cell being more negative than the outside. This is primarily due to: a) secondary active transport of Na+ b) the difference in the concentrations of Na+ inside and outside of the cell c) simple diffusion of K+ ions d) leak channels increasing the permeability of the membrane to K+ ions e) facilitated diffusion of Na+ ions
Which of the following occurs during synaptic transmission? Select ALL that apply A) vesicles filled with neurotransmitter are released into the synapse B) an action potential travels through a neuron like a wave C) neurotransmitter binds to receptors on the sending neuron D) neurotransmitter binding to a receptor triggers the opening of an ion channel E) ions enter the receiving neuron's dendrites
I need help please
The resting membrane potential…A) Can be approximated using the Nernst equation.B) Is established by a difference in charges across the cell membrane, with the inside more positive than the outside.C) Has no influence on the driving force for flow of a particular ion across the cell membrane.D) Is more dependent on the passive flow of potassium vs. sodium ions through leak channels across the cell membrane.E) Stays the same during an action potential.
Suppose that in a certain neuron, negatively charged protein molecules found in the intracellular compartment were suddenly able to diffuse through the cell membrane freely. What would be the result on this cell's resting membrane potential and action potential threshold? A) The resting potential would hyperpolarize and the action potential threshold would not change. B) The resting potential would depolarize and the action potential threshold would decrease. C) The resting potential would depolarize and the action potential threshold would not change. D) The resting potential would hyperpolarize and the action potential threshold would increase.
Conotoxin is produced by marine cone snails. Among its effects is to block voltage-gated Ca2+ channels in n eurons. A. What anatomical part of a neuron would be affected by conotoxin? B. How would the neuron's action potential be affected by conotoxin? Explain, using at least TWO of the following terms: threshold, depolarization, repolarization, hyperpolarization, summation, IPSP, EPSP, exocytosis C. If conotoxin affected a somatic motor neuron, would this toxin cause muscle weakness or increased muscle tension? Explain why.

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