3. In the experiment shown below, researchers looked at the effects of glucose-free medium onglutamate levels in synaptic nerve terminals. Analyze and describe the data. Then provide anexplanation for the findings.Glutamate (nmol/mg protein)O min10 min20 minControl36.1 + 3.234.5 + 2.435.9 + 2.2Glucose30.9 ± 3.725.1 + 5.8"16.9 + 0.5°

Glutamate is an amino acid that is synthesized from glutamine and through the transamination of…

Answered: 3. In the experiment shown below,…

Biochemistry

9th Edition

ISBN:9781319114671

Author:Lubert Stryer, Jeremy M. Berg, John L. Tymoczko, Gregory J. Gatto Jr.

Publisher:Lubert Stryer, Jeremy M. Berg, John L. Tymoczko, Gregory J. Gatto Jr.

Chapter1: Biochemistry: An Evolving Science

Section: Chapter Questions

Problem 1P

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. Intracellular concentrations in resting muscle are as follows: fructose- 6-phosphate, 1.0 mM; fructose-1,6-bisphosphate, 10 mM; AMP, 0.1 mM; ADP, 0.5 mM; ATP, 5 mM; and P;, 10 mM. Is the phosphofruc- tokinase reaction in muscle more or less exergonic than under stan- dard conditions? By how much?
3. Calculate the ATP that is produced when linoleic acid (9,12-octadecadienoic acid; 18:2) is (a) oxidized to CO2 and H20 or (b) converted to the ketone body acetoacetate in the liver and then oxidized to CO2 and H20 in the peripheral tissues. 4. Explain the role of AMP-dependent protein kinase in regulating fatty acid metabolism. What are the ways hormonal control of fatty acid metabolism works between fed and fasted states? Please draw by hand/digitally, schematics showing this regulation in adipose tissue, muscle and liver.
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Non-specific interactions present a real challenge to co-IP experiments because non-physiological binding to the target complex is likely to occur. Group of answer choices True False
5.Myasthenia (muscle weakness)is characterized by damage in the transmission of neurotic impuise and decreased amount of acetylcholine due to autoimmune mechanisms. Prozerine is used for treatment of this disease.Specify the mechanism of this drug action. For this: 1)Write the reaction of acetylcholine degradation at neuromuscular junction. 2)What enzyme will be inhibited with prozerine? Name the class of this enzyme. 3)What is the mechanism of this enzyme inhibition?
Ca2+ Using Figure 8.1, answer the following questions: 6) What is occurring in Step 2 of the figure above, and how is that process originated? 6) 7) Identify the structures on the postsynaptic membrane that bind with the neurotransmitter 7) in Steps 4 and 6 of the figure above, and indicate their function. 8) In the figure above, the overall function of Steps 6-8, which occur simultaneously, is to 8) 9) Based on the figure, describe the two possible mechanisms whereby Step 5 can occur on the postsynaptic membrane. 9) 10) Based on the figure, identify the structure in Step 6, if this presynaptic neuron is cholinergic. What if the neuron is adrenergic? 10)
A newly developed drug is suspected to act by inhibiting its enzymatic target. To test this, the rates of reaction in the presence and absence of the new drug were determined. The results of these experiments have been plotted as a double reciprocal plot shown in figure 1 below. Using this figure, calculate the values of Vmax and Km for the enzyme in the presence and absence of the drug. 2. Based on the figure and your answer, what type of inhibition is the new drug displaying? 3. Briefly explain how this type of inhibition exerts its action on an enzyme. Figure 1: Double reciprocal plot of reaction rate against substrate concentration in the presence and absence of a new drug.
Digoxin, a toxin derived from the foxglove (shown), can be used to treat heart disorders such as atrial fibrillation. Digoxin's mechanism of action is to Inhibit the Na+/K+ ATPase. Which of the following is the most likely side effect of Digoxin treatment? Failure to transport glucose into cells during an extended fast O Failure to transport glucose into cells after after eating a large meal O Enhanced response of muscle cells to Acetylcholine O Failure to transport Cl" out of the cell using direct active transport
When the Cu2+ concentration is 1.01 M, the observed cell potential at 298K for an electrochemical cell with the following reaction is 1.138V. What is the Cr3+ concentration? 3Cu2(aq) + 2Cr(s) 3Cu(s) + 2Cr3+(aq) Answer: M
Pls send solution fast within 10 minutes and i will give like for sure Solution must be in typed form Calcium ions bind to the SERCA Ca2+ -ATPase, which has two identical calcium sites, in two stages with apparent equilibrium constants K1=7×105M−1 and K2=2×106M−1 . a.) Calculate K. Note: K is not 0.1*1013 or 14*1011 or 1*1012. b.) Calculate τ.

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