3. Below are the fates of the carbon skeletons of amino acids. Fill out the table with thecorresponding amino acid source of each metabolic intermediatePyruvate (list at least 5)Аcctyl CoAAlpha-ketoglutarate1.1.1.2.2.2.3.3.3.4.4.5.5.

The amino acids formed during protein catabolism may be recycled to form new proteins, converted…

Answered: 3. Below are the fates of the carbon…

Biochemistry

9th Edition

ISBN:9781319114671

Author:Lubert Stryer, Jeremy M. Berg, John L. Tymoczko, Gregory J. Gatto Jr.

Publisher:Lubert Stryer, Jeremy M. Berg, John L. Tymoczko, Gregory J. Gatto Jr.

Chapter1: Biochemistry: An Evolving Science

Section: Chapter Questions

Problem 1P

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A patient who has been drinking large amounts of alcohol for long periods of time shows thefollowing symptoms: apathy, loss of memory, and a rhythmical to-and-fro motion of the eyeballs.Which of the following reactions are most likely to be affected in the patient? A. Conversation of pyruvate to acetyl-CoA B. Conversation of a-ketoglutarate to succinyl-CoA C. Both A and B D. Neither A nor B
Which of the following is incorrect about the conversion of pyruvate (with 3 C) and CO₂ to oxaloacetate (with 4 C) catalyzed by pyruvate carboxylase? O a. Pyruvate is being carboxylated O b. Pyruvate carboxylase is not a regulatory enzyme Oc. Pyruvate carboxylase uses biotin (a vitamin) as a cofactor Od. The reaction is.irreversible
Name the three processes circled, say where they occur/how they relate, and what their significant contribution is?
3) Indicate the order in which the metabolites in citric acid cycle are produced; acetyl CoA as 1 and oxaloacetate as 9. Metabolites in Citric Acid Cycle Order Enzyme involved in the formation of the metabolite Acetyl CoA 1 Oxaloacetate 9. Succinyl SCOA Fumarate Malate Citrate Succinate a-Ketoglutarate Isocitrate
Which of the following is NOT a fate of pyruvate in metabolism? .. Conversion to alanine as its storage form or for use by peripheral tissues such as skeletal muscles. Conversion to acetyl CoA for complete oxidation in the TCA cycle. Conversion to oxaloacetate in gluconeogenesis Conversion to lactate in a completely anaerobic process. L. Conversion to phophoenolpyruvate for the synthesis of glucose
Which of the following is NOT a fate of acetyl-CoA in mammals?a. Is used to synthesize cholesterolb. Is used to synthesize ketone bodiesc. Is used to synthesize fatty acidsd. Is used to synthesize malonyl-CoA that is in turn converted into alaninee. Is oxidized by citrate acid cyclef. All of above are fates of acetyl-CoA
OH The usage of acetyl-CoA in the cell depends on the metabolic / physiological state of the body. The table below shows the possible uses of acetyl-CoA, the corresponding physiological states of the body and the organ/cellular location where these processes occur. Identify which option is correct. Option Use of acetyl-CoA 1 2 3 4 Ketogenesis Cholesterol synthesis OOOO Fatty acid synthesis Oxidation via the citric acid cycle Select one: a. Option 1 b. Option 2 c. Option 3 O d. Option 4 Metabolic / Physiological state of the body Fasting state Fasting state Well fed Well fed Organ and cellular site Most cells / mitochondria Liver / cytoplasm Adipocyte / mitochondria Red blood cell / mitochondria Report question issue No
1.a. Given what you know about glycolysis and gluconeogenesis, does it make sense that insulin activates PDH phosphatase? Why? b.How do vitamins increase to the breadth of chemical reactions available within a biological system?
19. Which of the following statements is true for the shown reactions? i C-S-COA CH₂ H₂C-C-OH CH₂ coo W Coo CH₂ C=0 I Acetyl-CoA CH3 x 214 Coo- 1 CH ₂ I CHOH CH3 Y A. Deficiency of oxaloacetate stimulates the formation of X and y B. Insulin signaling stimulates the formation of X and Y C. Both A and B D. Neither A nor B
Back in the 1930s, a chemical called DNP (2,4-Dinitrophenol) was prescribed as a weight loss pill. Describe how DNP is a metabolic poison , and why it was thought to be a good mechanism for weight loss?
[References) Which of the following statements explains why acetyl-CoA Iis considered the central molecule of metabolism? (Select all that apply.) O The addition of acetyl-CoA to compounds stabilises them. Acetyl-CoA contains a thioester bond that makes it one of the high energy compounds in metabolic pathways. O Acetyl-CoA is an inhibitor that plays a central role in regulating metabolic pathways. O Acetyl-CoA feeds directly into the citric acid cycle. U None of the above.
2. 3. 4. 5. 6. 8. 7. 9. NADH Glutamate NAD 77 a-Ketoglutarate NADH NAD 7. a-Ketoglutarate 15/ Glutamate Cytosol 10. Fumarate 2. Citrulline Oxaloacetate 3. Citrulline 6. 1. Mitochondrial matrix 1. Metabolite name: Metabolite name (this metabolite breaks down into #3 and #8): Metabolite name: Metabolite name: Metabolite name: Metabolite name (this is the same metabolite as #5): Metabolite name (this same metabolite is moved to the cytosol can combine with citrulline to make #2 OR can be transaminated to make #9): 8. Metabolite name: 9. Metabolite name: 10. Metabolite name: 5.

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3. Below are the fates of the carbon skeletons of amino acids. Fill out the table with the corresponding amino acid source of each metabolic intermediate Pyruvate (list at least 5) Acetyl CoA Alpha-ketoglutarate 1. 1. 1. 2. 2. 2. 3. 3. 3. 4. 4. 5. 5.