Brock Biology of Microorganisms (15th Edition)
15th Edition
ISBN: 9780134261928
Author: Michael T. Madigan, Kelly S. Bender, Daniel H. Buckley, W. Matthew Sattley, David A. Stahl
Publisher: PEARSON
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Chapter 7, Problem 2AQ
Explain how cells exhibiting different
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Consider a liver cell. How many chromosomes are present and how many alleles of each gene are present?
Use the picture below to answer the questions that follow.
A
∞
SE
Which of letters indicate:
1) identical molecules of DNA [Select]
B
2) similar molecules of DNA, can have different alleles [Select]
3) the result of S phase [Select]
4) Do the two chromosomes (red and blue) have the same genes?
[Select]
Suppose that you are studying the role of Protein B, which you believe plays a role in regulating PCD/Apoptosis in mice. You create two lines of mutant mice. One (bb) is homozygous for a loss-of-function allele of gene B. The other (Bb) is heterozygous, with one wild-type allele and one loss-of function allele. Initially you pay particular attention to two phenotypes of the resulting mice:(i) The morphology of their paws (see picture) (ii) The size of their brains & shape of their skulls. The bb mice have unusually large brains and unusual protrusions from their skulls. Based on these data, does it appear that Protein B, when present and active, favors or inhibits PCD/Apoptosis?Briefly explain your reasoning. The answer should address both the paw and brain/skull data.
Chapter 7 Solutions
Brock Biology of Microorganisms (15th Edition)
Ch. 7.1 - What is the utility of a reporter gene?Ch. 7.1 - Prob. 2MQCh. 7.1 - Prob. 1CRCh. 7.2 - Prob. 1MQCh. 7.2 - Prob. 2MQCh. 7.2 - Prob. 1CRCh. 7.3 - What is the divisome?Ch. 7.3 - How does FtsZ find the cell midpoint of a...Ch. 7.3 - What is the role of the penicillin-binding protein...Ch. 7.4 - How does MreB control the shape of a rod-shaped...
Ch. 7.4 - What protein is thought to control the shape of...Ch. 7.4 - What relationships exist between cytoskeletal...Ch. 7.4 - What morphology do cells have that lack MreB or...Ch. 7.5 - Prob. 1MQCh. 7.5 - What is transpeptidation and why is it important...Ch. 7.5 - Prob. 1CRCh. 7.6 - How are different sets of genes expressed in the...Ch. 7.6 - Prob. 2MQCh. 7.6 - Prob. 1CRCh. 7.7 - Why are the levels of DnaA protein controlled...Ch. 7.7 - Prob. 2MQCh. 7.7 - Prob. 1CRCh. 7.8 - Prob. 1MQCh. 7.8 - What is the major transcriptional regulator that...Ch. 7.8 - What is meant by "patterning" during heterocyst...Ch. 7.9 - What are the four basic stages of biofilm...Ch. 7.9 - Besides autoinducer synthesis, what intracellular...Ch. 7.9 - What type of genes does c-di-GMP activate during...Ch. 7.10 - Describe two targets of antibiotics and discuss...Ch. 7.10 - Prob. 2MQCh. 7.10 - Prob. 3MQCh. 7.10 - Prob. 1CRCh. 7.11 - Is persistence a heritable trait?Ch. 7.11 - What prevents the toxin component of TA modules...Ch. 7.11 - Prob. 3MQCh. 7.11 - Prob. 1CRCh. 7 - If DnaA was not regulated in Escherichia coli and...Ch. 7 - Explain how cells exhibiting different phenotypes...Ch. 7 - Describe how you would genetically design a...
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- Suppose that you are studying the role of Protein B, which you believe plays a role in regulating PCD/Apoptosis in mice. You create two lines of mutant mice. One (bb) is homozygous for a loss-of-function allele of gene B. The other (Bb) is heterozygous, with one wild-type allele and one loss-of function allele. Initially you pay particular attention to two phenotypes of the resulting mice:(i) The morphology of their paws (see picture)(ii) The size of their brains & shape of their skulls. The bb mice have unusually large brains and unusual protrusions from their skulls. Suggest one other aspect of mouse morphology or physiology that you might expect to be altered in the absence of Protein B. Briefly explain your reasoning. Also, based on the apparent effect of Protein B on the likelihood of PCD/Apoptosis, would you classify Protein B as the product of a proto-oncogene or of a tumor suppressor gene?arrow_forwardGive typing answer with explanation and conclusionarrow_forwardIn your own words, explain the following concepts and provide at least one example for each: Polymorphism (Minimum of 2 complete sentences.)arrow_forward
- Colorblindness and hemophilia are both X-linked traits in humans. Explain how a female who has a defective color vision gene on one X chromosome and a defective blood clotting gene causing hemophilia on the other X chromosome can be neither a hemophiliac nor colorblind? Please discuss the effect of Gene dosage compensation in your answer and in your answer describe the molecular process by which this occurs.arrow_forwardImagine that you are a new breeder for caique parrots. You can sell normal green feather birds for $500 a bird. The mutation for blue feathers is rare and birds with this color can sell for $3000 a bird. Your goal is to produce as many blue feather offspring as you can, but you don't have the budget to buy blue feather birds as parents. Question: What genotype should you purchase for both parent blrds to get the best chance of getting blue feather offspring? Create and use your Punnett square results as evidence to support your answer. You will need to make multiple Punnett Squares to see which parent genotypes makes the most blue feathered offspring. Keep in mind that Green fealhers (G) is dominant over blue feathers (g), so birds with blue feathers have the genotype gg. Some Punnett squares are provided for you to determine the possible crosses, but remember yoU cannot afford a blue feathered bird, so neither of your parent birds can have the genotype gg. Complete a Punnett Square…arrow_forwardThe table below shows the progeny of a test cross of a heterozygote (HhFfGg). Each row shows the number of progeny that inherited each combination of alleles from the heterozygous parent. You've already determined the gene order (G is in the middle) and labeled each type of progeny. Using this information, what is the recombination frequency between H and G? Alleles: Type: HGF hgf HgF h Gf Hgf h GF HGf hg F O 0.543 0.302 O 0.241 O 0.23 0.139 Parental Parental DCO DCO SCO (H-G) SCO (H-G) SCO (G-F) SCO (G-F) Count: 124 119 15 18 51 55 38 40 O The correct answer is not available.arrow_forward
- Consult Figure 1, which shows a human karyotype with chromosomes arranged in order of decreasing size. Would you expect this cell to have any genes characterized as hemizygous? If so, what is an example of a hemizygous gene? On which chromosome would it be located?arrow_forwardB B BB Bb b Bb bb Brown rabbits have the genotype BB or Bb. White rabbits have the genotype bb. If two brown rabbits, with the genotypes seen in the Punnett square above, have baby rabbits, what is the probability that the baby rabbits will also be brown? A B) 50% 75% D) 100% 5) According to Mendel's is why gametes have half the usual number of chromosomes. one copy of a gene is passed randomly from each parent to their offspring. This Sign out acerarrow_forwardPlease help True or false, if false rewrite the statement in a way that makes it true. a) A cell spends most of its life dividing rather than carrying out typical cell functions. False b) We can know the genotype of an individual simply by looking at their phenotype. Falsearrow_forward
- Wild-type strains of the haploid fungus Neurospora canmake their own tryptophan. An abnormal allele td renders the fungus incapable of making its own tryptophan.An individual of genotype td grows only when its medium supplies tryptophan. The allele su assorts independently of td; its only known effect is to suppress the tdphenotype. Therefore, strains carrying both td and su donot require tryptophan for growth.a. If a td ; su strain is crossed with a genotypically wildtype strain, what genotypes are expected in the progenyand in what proportions?b. What will be the ratio of tryptophan-dependent totryptophan-independent progeny in the cross of part a?arrow_forwardBased on the data in Table 1, which individual(s) is/are heterozygous? Select all that apply a) B IV-8 b) C IV-3 c) B IV-9 d) A IV-3arrow_forwardMost of the genetic information we will get from our genome will not be hard evidence that we will or we won’t get a disease but is rather only probabilistic evidence. a. What does this mean? b. Why might knowing a single gene not tell you if you will get a particular condition?arrow_forward
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