Brock Biology of Microorganisms (15th Edition)
15th Edition
ISBN: 9780134261928
Author: Michael T. Madigan, Kelly S. Bender, Daniel H. Buckley, W. Matthew Sattley, David A. Stahl
Publisher: PEARSON
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Textbook Question
Chapter 28.11, Problem 2MQ
Why are there fewer clinically effective antifungal and antiparasitic agents than antibacterial agents?
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Which of the following types of antimicrobial drugs make microbes more susceptible
to osmotic pressure?
1) drugs that inhibit nucleic acid synthesis
O 2) drugs that inhibit metabolic pathways
3) drugs that inhibit cell wall synthesis
4) drugs that block a pathogen's recognition of its host
5) drugs that inhibit protein synthesis
There are fewer antifungal, anti protozoal, and antihelminth drugs compared to antibacterial drugs because these organism:
a) do not cause many human infectious
b) are not affected by antimicrobial drugs
c) are so similar to human cells that drug selective toxicity is difficult
d) are parasites found inside human cells
e) have fewer target sites compared to bacteria
Which one of the following categories for antibacterial agents is most likely to have
toxic side effects for humans?
a) disruptors of cell membrane structure
b) inhibitors of cell wall synthesis
Oc) inhibitors of protein synthesis
d) antimetabolites
RESET
e) inhibitors of DNA synthesis
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Chapter 28 Solutions
Brock Biology of Microorganisms (15th Edition)
Ch. 28.1 - The use of personal protective equipment (PPE) is...Ch. 28.1 - Identify and discuss the standard safety...Ch. 28.1 - Prob. 1CRCh. 28.2 - Prob. 1MQCh. 28.2 - How can the spread of HAIs be controlled?Ch. 28.2 - Prob. 1CRCh. 28.3 - What are the key points necessary for proper...Ch. 28.3 - Identify culture methods and conditions used for...Ch. 28.3 - QWhy is it important to process clinical specimens...Ch. 28.4 - Describe the disc diffusion test and the Etest for...
Ch. 28.4 - What is the value of antimicrobial drug...Ch. 28.4 - QDescribe the disc diffusion test for antibiotic...Ch. 28.5 - Explain the reasons for changes in antibody titer...Ch. 28.5 - Describe the method, time frame, and rationale for...Ch. 28.5 - What advantages do monoclonal antibodies have...Ch. 28.5 - QWhy does antibody titer rise after infection? Is...Ch. 28.6 - How is the bivalence of antibodies significant for...Ch. 28.6 - What are the advantages and disadvantages of...Ch. 28.6 - Why are agglutination tests so widely used in...Ch. 28.7 - Prob. 1MQCh. 28.7 - Compare the advantages and disadvantages of EIA,...Ch. 28.7 - Prob. 1CRCh. 28.8 - What advantage(s) does nucleic acid amplification...Ch. 28.8 - How do quantitative PCR (qPCR) and qualitative PCR...Ch. 28.8 - Distinguish between quantitative and qualitative...Ch. 28.9 - Compare and contrast live attenuated vaccines,...Ch. 28.9 - Identify the advantages of alternative...Ch. 28.9 - QList the immunizations recommended for children...Ch. 28.10 - Prob. 1MQCh. 28.10 - How does the activity of each antibiotic class...Ch. 28.10 - What are the sources of aminoglycosides,...Ch. 28.10 - Antibiotics are chemically diverse antimicrobial...Ch. 28.11 - What steps in the viral maturation process are...Ch. 28.11 - Why are there fewer clinically effective...Ch. 28.11 - Why is host toxicity a common problem with...Ch. 28.12 - Identify the basic mechanisms of antibiotic...Ch. 28.12 - What does vancomycin have in common with...Ch. 28.12 - Prob. 3MQCh. 28.12 - What practices contribute to the spread of...Ch. 28 - Define the procedures you would use to isolate and...Ch. 28 - Prob. 2AQCh. 28 - Describe three important reasons why semisynthetic...Ch. 28 - Imagine yourself as a clinical microbiologist with...
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Need a deep-dive on the concept behind this application? Look no further. Learn more about this topic, biology and related others by exploring similar questions and additional content below.Similar questions
- Why does Helicobacter pylori cause ulceractions in the lining of the Stomach? 1)The microbes secrete a toxin that prevents the host from detecting invasion of the microbe and thus allows it to thrive in the stomach and damage the host cells. 2)After invasion of the microbe in the cells lining the stomach, they are no longer able to produce mucous to protect the host cells from acid erosion 3)The microbe produces an enzyme that causes the cell to produce potent digestive enzymes that erode the stomach lining. 4)Once the microbe has entered the stomach, it releases digestive enzymes that degrade the mucous secreting cells.arrow_forwardCompared with all other foodborne or waterborne pathogens,what is unique about prions?arrow_forwardWhat makes bacterial diseases such as Clostridium botulinum, Clostridium perfringens & Clostridium tetani very effective to cause infection in a human host?arrow_forward
- The type of antimicrobial drug that would be most likely to have toxic effects in humans based on the premise of selective toxicity when treating microbial infection would be a drug that O 1) inhibits the synthesis of the cell wall O 2) inhibits metabolic pathways O 3) disrupts the cytoplasmic membrane 4) inhibits protein synthesis 5) All of the above have equal potential to be toxicarrow_forward19) Which of the following BEST describes an agent that is bactericidal? A) Windex. B) It suspends bacterial growth. C) It kills the bacteria. D) It causes the bacteria to destroy themselvesarrow_forwardDo common fungi such as bread mold produce antimicrobial compounds?arrow_forward
- 24) An opportunistic infection is a concern with which of the following medications? A) Antifungals B) Antiprotozoals C) Antibiotics D) All of the abovearrow_forwardWhich antimicrobial drug does not inhibit cell wall synthesis? a) gentamicin b) vancomycin c) cephalosporins d) penicillinsarrow_forwardExplain why there are fewer antifungal, antiparasitic, and antiviraldrugs than antibacterial drugs.arrow_forward
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USMLE-Rx Antimicrobial Therapy; Author: USMLE-Rx;https://www.youtube.com/watch?v=Pka1Ynlrqtw;License: Standard Youtube License