Prescott's Microbiology
10th Edition
ISBN: 9781259281594
Author: Joanne Willey, Linda Sherwood Adjunt Professor Lecturer, Christopher J. Woolverton Professor
Publisher: McGraw-Hill Education
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Textbook Question
Chapter 27.2, Problem 1.4RIA
Describe the roles of cII, CIII, λ repressor (CI), Cro, Q, RecA, integrase, and excisionase in lysogeny and induction.
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Chapter 27 Solutions
Prescott's Microbiology
Ch. 27.1 - List some characteristics used in classifying...Ch. 27.1 - Consider these terms: Equine torovirus,...Ch. 27.2 - What enzyme found in the T4 baseplate facilitates...Ch. 27.2 - Why do you think T4 evolved to initiate DNA...Ch. 27.2 - What function does HMC glycosylation serve?Ch. 27.2 - How is the envelope of this virus formed? How does...Ch. 27.2 - Explain why the T4 genome is circularly permuted.Ch. 27.2 - Prob. 1.2RIACh. 27.2 - How is a prophage induced to become active again?Ch. 27.2 - Describe the roles of cII, CIII, repressor (CI),...
Ch. 27.2 - How do the temperate phages Mu and P1 differ from...Ch. 27.2 - The CRISPR/Cas system has been a boon to...Ch. 27.2 - Why do cold sores recur throughout the lifetime of...Ch. 27.2 - In what part of the host cell does a herpesvirus...Ch. 27.2 - Many small DNA viruses rely on host enzymes for...Ch. 27.3 - Why is the X174 genome considered plus stranded?Ch. 27.3 - Prob. 2MICh. 27.3 - Why is it necessary for some ssDNA viruses to...Ch. 27.3 - From the point of view of the virus, compare the...Ch. 27.3 - How do parvoviruses trick the host DNA polymerase...Ch. 27.4 - The rotavirus genome encodes 12 proteins. Suggest...Ch. 27.4 - Describe the life cycle of 6 phage. What makes...Ch. 27.4 - Prob. 3RIACh. 27.4 - In what ways are the life cycles of 6 and...Ch. 27.5 - Where in the host does the plus-strand RNA genome...Ch. 27.5 - How do some plus-strand viruses use polyproteins...Ch. 27.5 - What is an IRES? Why is it important?Ch. 27.5 - Prob. 3RIACh. 27.6 - How does that use of a segmented genome by...Ch. 27.6 - Prob. 2RIACh. 27.7 - Prob. 1MICh. 27.7 - Prob. 1RIACh. 27.7 - Prob. 2RIACh. 27.7 - What role does alternative splicing play in the...Ch. 27.8 - Prob. 1RIACh. 27.8 - Trace the HBV multiplication cycle, paying...Ch. 27 - No temperate RNA phages have yet been discovered....Ch. 27 - The choice between lysogeny and lysis is...Ch. 27 - Prob. 3CHICh. 27 - You are studying RNA viruses and have discovered a...Ch. 27 - Prob. 5CHICh. 27 - Upon infection of host epithelial cells,...Ch. 27 - Associated with the envelope of herpesviruses are...
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- Identify three key ways that TrXG genes promote transcription with respect to: Mediator Complex Structurearrow_forwardTransformation is a process in which bacteria take up new DNA released by dead cells and integrate it into their own genomes (see p. 265 in Chapter 9). In Streptococcus pneumoniae (which causes many cases of pneumonia, inner-ear infections, and meningitis), the ability to carry out transformation requires from 105 to 124 genes, collectively termed the com regulon. The com regulon is activated in response to a protein called competence-stimulating peptide (CSP), which is produced by the bacteria and exported into the surrounding medium. When enough CSP accumulates, it attaches to a receptor on the bacterial cell membrane, which then activates a regulator protein that stimulates the transcription of genes within the com regulon and sets in motion a series of reactions that ultimately result in transformation. Does the com regulon in Streptococcus pneumoniae exhibit positive or negative control? Explain your answer.arrow_forwardiestiðn Pre-initiation complex (PIC) is formed right before the initiation of genes, which of the following steps seemes to NOT help for the PIC formation? O TBP protein to recognize TATA box at promoter region O TFIIA to recognize the TAFS in TFIID O Enhancers to attract coactivators and activators O Silencers to attract repressor and corepressorsarrow_forward
- According to the scenario shown, how many segments of DNA (one, two, or three) are removed during site-specific recombination within the gene that encodes the κ (kappa) light chain for IgG proteins? How many segments are spliced out of the pre-mRNA?arrow_forwardGive 4 examples of transcription factors / non-structural proteins you have learned in this course? Identify the most commonly used technique for diagnosing COVID-19? And the clinical sample for each technique. …….…… virus can cause encephalitis, cancer, sexually transmitted disease, infect external genitalia, mucosal surfaces, gladiatorum, and/or other diseases. How can the infection with this virus be avoided? Suggest two ways of prevention or to destroy the virus.arrow_forwardContrast the role of the repressor in an inducible system and in a repressible system.arrow_forward
- Compare and contrast the use of histochemical reporter genes and fluorescent reporter genes — what are the advantages and disadvantages of each?arrow_forwardWhat is the main lincRNA involved in X-inactivation, and what is known about its function?arrow_forwardExplain any five of the following terminologies: gene regulation, enhancer or silencer sequences, promoter, designer genes, translation, transgene, terminator sequence, marker genes, transcription.arrow_forward
- Explain why the events shown in part (a) inhibittranscription.arrow_forwardMembers of a family are affected by a hereditary inflammatory and immunodeficiency disease. The disease phenotype was due to a mutation in the PLCG2 gene, determined by comparing an affected father and an affected daughter, both heterozygotes, with the homozygous unaffected mother (Zhou et al, 2012). The wild-type protein acts to regulate various immune and inflammatory pathways within the cell. The variant protein is over expressed (more abundant), leading to increased intracellular signaling and leading to increased inflammation and reduced immunity in the affected people. Select all the statements below that can be correctly applied to this system. [Hint: There are FOUR.] O co-dominance dominant negative mutant allele O gain-of-function mutation O haplosufficent wildtype allele O haploinsuffient wildtype allele O leaky mutant allele O loss-of-function mutation O null mutant allele O partial dominance O simple dominance O the mutant allele is hypermorphic O the mutant allele is…arrow_forwardName TWO functions of the 3’UTR.arrow_forward
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