Microbiology: An Introduction
11th Edition
ISBN: 9780321733603
Author: Gerard J. Tortora, Berdell R. Funke, Christine L. Case
Publisher: Benjamin Cummings
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Chapter 17, Problem 3CAE
A patient with chronic diarrhea was found to lack IgA in his secretions, although he had a normal level of serum IgA. What was this patient found to be unable to produce?
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Chapter 17 Solutions
Microbiology: An Introduction
Ch. 17 - Contrast the terms in the following pairs: a....Ch. 17 - What does MHC stand for? What is the function of...Ch. 17 - Why is dietary protein deficiency associated with...Ch. 17 - Prob. 4RCh. 17 - Prob. 5RCh. 17 - DRAW IT a. In the graph below, at time A the host...Ch. 17 - How would each of the following prevent infection?...Ch. 17 - Prob. 8RCh. 17 - Explain why a person who recovers from a disease...Ch. 17 - Prob. 10R
Ch. 17 - Prob. 1ACh. 17 - Why is dietary protein deficiency associated with...Ch. 17 - A positive tuberculin skin test shows cellular...Ch. 17 - On her vacation to Australia, Janet was bitten by...Ch. 17 - Prob. 1MCQCh. 17 - The type of protection provided by the injection...Ch. 17 - Prob. 3MCQCh. 17 - Prob. 4MCQCh. 17 - Prob. 5MCQCh. 17 - Prob. 6MCQCh. 17 - Prob. 7MCQCh. 17 - Prob. 8MCQCh. 17 - Prob. 9MCQCh. 17 - Prob. 10MCQCh. 17 - Prob. 1CAECh. 17 - Prob. 2CAECh. 17 - A patient with chronic diarrhea was found to lack...Ch. 17 - Newborns (under 1 year) who contract dengue have a...
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- What is complement? How does it cause bacterial lysis? What are some of the other roles of complement?arrow_forward(56) A 1 year old boy dies of staphylococcal pneumonia. He has had multiple skin infections since birth. Separation of the umbilical cord was delayed. A skin window test was performed and no leukocyte accumulated on the cover ship. The cytometric analysis showed that only 5% of the T lymphocytes reacted with a monoclonal antibody to leukocyte functional antigen-1 chain (CD 18) compared with reactivity of 62 % for a normal subject. Just prior to death, his leukocyte count was 75,000/mm3. Which of the following is the most likely diagnosis? (A) AIDS (B) Cogenital Asplenia (C)Leukocyte Adhesion Deficiency (D) Severe Combined Immunodeficiency Disease (E) X linked agammaglobulinemiaarrow_forwardHow is HLA serologic testing performed? Question 28 options: a) HLA antigens move through a limited pH gradient and are immobilized, complexed with antibody, and stained. b) T lymphocytes are harvested and introduced to a panel of test sera specific for antigens of the HLA system. c) DNA is isolated from peripheral white blood cells, fragmented, and hybridized. d) B lymphocytes are harvested and introduced to a panel of test sera specific for antigens of the HLA system.arrow_forward
- A person with type A+ blood gets a transfusion with type O- blood. What is most likely to happen to the recipient? A) The recipient's blood will agglutinate (clump) due to the presence of natural antibodies in the recipient's blood. B) Nothing because the donor's blood is compatible with the recipient's blood. C) The recipient's blood will agglutinate (clump) due to the presence of natural antigens on the recipient's blood cells.arrow_forwardMike, an unvaccinated university student traveled to Western Europe, where he contracted mumps (viral infection). A month after his return, his roommate, Josh, also complained of symptoms of mumps. However, Josh had received his MMR vaccination as part of his childhood immunizations. IgM antibodies were detected in Mike’s serological test but not in Josh’s bloodwork. However, circulating IgG was detected in both patients. * IgM antibodies have a short half-life. IgG have a longer half-life (remain in an “active state” for longer, not broken down as quickly). Why was it significant that Mike had not been vaccinated and had traveled to Western Europe? What is the significance of finding Ig M antibodies? Why might Josh’s serology test results have been negative for mumps IgM and positive for IgG? Is it possible for Josh to have a mumps infection, even if he had been vaccinated as a child?arrow_forwardhere is a second, delayed spike of IgM in the secondary response. Why might this be occurring?arrow_forward
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