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Concept explainers
We have a new patient to consider today. Mr. Gutteman, a 70-year-old male, was brought into the ER in a comatose state and has yet to come out of it. It is obvious that he suffered severe head trauma-his scalp was badly lacerated, and he has an impacted skull fracture. His initial lab tests (blood and urine) were within normal limits. His fracture was repaired and the following orders (and others) were given:
Check qh (every hour) and record: spontaneous behavior, level of responsiveness to stimulation, movements, pupil size and reaction to light, speech, and vital signs.
Turn patient q4h and maintain meticulous skin care and dryness.
Mr. Gutteman is found to be losing huge amounts of water in urine and the volume lost is being routinely replaced (via IV line). Mr. Gutteman’s blood and urine tests are negative for glucose and
Relative to these findings:
Is it life threatening? (Explain your answer.)
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Chapter 15 Solutions
Anatomy & Physiology
- circle a nucleotide in the imagearrow_forward"One of the symmetry breaking events in mouse gastrulation requires the amplification of Nodal on the side of the embryo opposite to the Anterior Visceral Endoderm (AVE). Describe one way by which Nodal gets amplified in this region." My understanding of this is that there are a few ways nodal is amplified though I'm not sure if this is specifically occurs on the opposite side of the AVE. 1. pronodal cleaved by protease -> active nodal 2. Nodal -> BMP4 -> Wnt-> nodal 3. Nodal-> Nodal, Fox1 binding site 4. BMP4 on outside-> nodal Are all of these occuring opposite to AVE?arrow_forwardIf four babies are born on a given day What is the chance all four will be girls? Use genetics lawsarrow_forward
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- Participants will be asked to create a meme regarding a topic relevant to the department of Geography, Geomatics, and Environmental Studies. Prompt: Using an online art style of your choice, please make a meme related to the study of Geography, Environment, or Geomatics.arrow_forwardPlekhg5 functions in bottle cell formation, and Shroom3 functions in neural plate closure, yet the phenotype of injecting mRNA of each into the animal pole of a fertilized egg is very similar. What is the phenotype, and why is the phenotype so similar? Is the phenotype going to be that there is a disruption of the formation of the neural tube for both of these because bottle cell formation is necessary for the neural plate to fold in forming the neural tube and Shroom3 is further needed to close the neural plate? So since both Plekhg5 and Shroom3 are used in forming the neural tube, injecting the mRNA will just lead to neural tube deformity?arrow_forwardWhat are some medical issues or health trends that may have a direct link to the idea of keeping fat out of diets?arrow_forward
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