Molecular mimicry is the possibility that resemblance of sequences between self-peptides and foreign-peptides is enough to cause the cross-activation of B or T-cells through pathogen-derived proteins.

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Chapter 14, Problem 4Q

Summary Introduction

Introduction:

Molecular mimicry is the possibility that resemblance of sequences between self-peptides and foreign-peptides is enough to cause the cross-activation of B or T-cells through pathogen-derived proteins.

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Chapter 14, Problem 4Q

Summary Introduction

Introduction:

Molecular mimicry is the possibility that resemblance of sequences between self-peptides and foreign-peptides is enough to cause the cross-activation of B or T-cells through pathogen-derived proteins.

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Chapter 14, Problem 4Q

Summary Introduction

Introduction:

Molecular mimicry is the possibility that resemblance of sequences between self-peptides and foreign-peptides is enough to cause the cross-activation of B or T-cells through pathogen-derived proteins.

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Answer 4

Question

Chapter 14, Problem 4Q

Summary Introduction

Introduction:

Molecular mimicry is the possibility that resemblance of sequences between self-peptides and foreign-peptides is enough to cause the cross-activation of B or T-cells through pathogen-derived proteins.

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Answer 5

Chapter 14, Problem 4Q

Summary Introduction

Introduction:

Molecular mimicry is the possibility that resemblance of sequences between self-peptides and foreign-peptides is enough to cause the cross-activation of B or T-cells through pathogen-derived proteins.

Answer 6

Chapter 14, Problem 4Q

Summary Introduction

Introduction:

Molecular mimicry is the possibility that resemblance of sequences between self-peptides and foreign-peptides is enough to cause the cross-activation of B or T-cells through pathogen-derived proteins.

Answer 7

Chapter 14, Problem 4Q

Summary Introduction

Introduction:

Molecular mimicry is the possibility that resemblance of sequences between self-peptides and foreign-peptides is enough to cause the cross-activation of B or T-cells through pathogen-derived proteins.

Answer 8

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Answer 11

Ch. 14.1 - Define immunopathology, and identify the two major...Ch. 14.1 - Identify the four major categories of...Ch. 14.2 - Summarize genetic and environmental factors that...Ch. 14.2 - Identify three conditions caused by IgE-mediated...Ch. 14.2 - Identify the two clinical forms of anaphylaxis,...Ch. 14.2 - List the three main ways to prevent or...Ch. 14.2 - NCLEX PREP 1. During a visit to his physician, a...Ch. 14.2 - Prob. 2NP Ch. 14.3 - List the three immune components causing cell...Ch. 14.3 - Prob. 8AYP

Molecular mimicry is the possibility that resemblance of sequences between self-peptides and foreign-peptides is enough to cause the cross-activation of B or T-cells through pathogen-derived proteins.

Solution Summary: The author explains that molecular mimicry is dependent on sequence similarity between host and pathogen, which causes the host to generate an immune response against host tissues.

Introduction:

Molecular mimicry is the possibility that resemblance of sequences between self-peptides and foreign-peptides is enough to cause the cross-activation of B or T-cells through pathogen-derived proteins.

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Chapter 14 Solutions

Molecular mimicry is the possibility that resemblance of sequences between self-peptides and foreign-peptides is enough to cause the cross-activation of B or T-cells through pathogen-derived proteins.

Solution Summary: The author explains that molecular mimicry is dependent on sequence similarity between host and pathogen, which causes the host to generate an immune response against host tissues.

Introduction: Molecular mimicry is the possibility that resemblance of sequences between self-peptides and foreign-peptides is enough to cause the cross-activation of B or T-cells through pathogen-derived proteins.

Want to see the full answer?

Chapter 14 Solutions

Introduction:

Molecular mimicry is the possibility that resemblance of sequences between self-peptides and foreign-peptides is enough to cause the cross-activation of B or T-cells through pathogen-derived proteins.