BIOLOGY DUAL ENROLLMENT VERSION
12th Edition
ISBN: 9780136681311
Author: Urry
Publisher: PEARSON
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Chapter 11.4, Problem 3CC
Summary Introduction
To determine: The affect on the cell signaling pathways due to the activation of malfunctioning protein phosphatases.
Concept introduction: The mutation or the malfunctioning of the protein molecules will lead to a constant activation of the protein response without inhibition inside the cell. The function of the protein phosphatases is to remove the phosphate group from the protein signaling leading to an inhibition of the cell signaling pathway.
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Chapter 11 Solutions
BIOLOGY DUAL ENROLLMENT VERSION
Ch. 11.1 - Explain how signaling is involved in ensuring that...Ch. 11.1 - In liver cells, glycogen Phosphorylase acts in...Ch. 11.1 - Prob. 3CCCh. 11.2 - Prob. 1CCCh. 11.2 - WHAT IF? What would the effect be if a cell made...Ch. 11.2 - MAKE CONNECTIONS How is ligand binding similar to...Ch. 11.3 - What is a protein kinase, and what is its role in...Ch. 11.3 - When a signal transduction pathway involves a...Ch. 11.3 - What is the actual signal that is being transduced...Ch. 11.3 - WHAT IF? If you exposed a cell to a ligand that...
Ch. 11.4 - How can a targct cell's response to a single...Ch. 11.4 - WHAT IF? If two cells have different scaffolding...Ch. 11.4 - Prob. 3CCCh. 11.4 - Prob. 4CCCh. 11.5 - Give an example of apoptosis during embryonic...Ch. 11.5 - WH AT IF? If apoptosis occurred when it should...Ch. 11 - What determines whether a cell responds to a...Ch. 11 - How are the structures of a GPCR and an RTK...Ch. 11 - What is the difference between a protein kinase...Ch. 11 - What mechanisms in the cell terminale its response...Ch. 11 - What is an explanation for the similarities...Ch. 11 - Binding of a signaling molecule to which type of...Ch. 11 - The activation of receptor tyrosinc kinases is...Ch. 11 - Lipid-soluble signaling molecules, such as...Ch. 11 - Consider this pathway: epinephrine G...Ch. 11 - Apoptosis involves all but which of the following?...Ch. 11 - Which Observation suggestcd to Sutherland the...Ch. 11 - Prob. 7TYUCh. 11 - DRAW IT Draw the following apoptotic pathway,...Ch. 11 - EVOLUTION CONNECTION Identify the evolutlonary...Ch. 11 - Prob. 10TYUCh. 11 - SCIENCE, TECHNOLOGY, AND SOCIETY The aging process...Ch. 11 - WRITE ABOUT A THEME: ORGANIZATION The properties...Ch. 11 - SYNTHESIZE YOUR KNOWLEDGE There are five basic...
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- Does it show the level of proteins? What about the amount? Levels of protein activation? How can you tell? Does the thickness tell you anything? What about the number of the lines? And the other questionsarrow_forwardKD 200- 116- 66- Vec ATF6 (670) ATF6 (402) ATF6 (373) ATF6 (366) I I 45- 1 2 3 4 5 ATFG (360) (e/c) 9V ATFG (402) g ant- ATF anti-KDEL DAPI barrow_forwardWestern blot results: what information can you get? Presence of proteins of your interest Levels of protein expression Levels of protein activation (must use activation state-specific antibody) Decreased function of the ATM kinase in aging mice. A C57BL/6 female 6 month Con IR 20 month C57BL/6 male 6 month 28 month Con IR Con IR Con IR p-ATM (S1981) ATM P-p53 (ser18) Actinarrow_forward
- Does it show the level of proteins? What about the amount? Levels of protein activation? How can you tell? Does the thickness tell you anything? What about the number of the lines?arrow_forwardWB: Protein of interest visualized by fluorescent Protein A Protein Barrow_forwardQuestion #4: Assume you are able to use CRISPR to create an allele that will convert a cross-pollinated, sexually reproducing crop plant into an obligate apomict. Your edited obligate apomict plants retains all the CRISPR "machinery" necessary to convert the "sexually reproducing" allele to the "obligate apomict" allele. You plant 100 hectares of your edited obligate apomicts in order to increase seed for sale the following year. Neighboring farms and seed producers are growing many different un-edited sexually reproducing varieties of the crop. If your neighbors plant seed harvested from their crops that was pollinated by your crop, should they expect these seeds to generate apomictic or non-apomictic plants? Type your answer here:arrow_forward
- calculate the questions showing the solution including variables,unit and equations all the questiosn below using the data.show solving and answer a) B1, b) B2, c) hybrid rate constant (1) d) hybrid rate constant (2) e) t1/2,dist t1/2,absorb f) t1/2,elim k) apparent central compartment volume (V1,app) p) total AUC (using short cut method) apparent volume of distribution based on AUC (VAUC,app) apparent clearance (CLapp) absolute bioavailabilty of oral route ( AUCiv =116ml)arrow_forwardPlease help me to draw this by hand. In as much detail as possible, hand draw a schematic diagram of the hypothalamic-pituitary-gonad (HPG) axis in the human female. Be sure to include all the relevant structures and hormones. You must define all abbreviations the first time you use them. Please include (and explain) the feedback loops.arrow_forwardPlease refer belowarrow_forward
- AaBbCc X AaBbCc individuals are crossed. What is the probability of their offspring having a genotype AABBCC?arrow_forwardcircle a nucleotide in the imagearrow_forward"One of the symmetry breaking events in mouse gastrulation requires the amplification of Nodal on the side of the embryo opposite to the Anterior Visceral Endoderm (AVE). Describe one way by which Nodal gets amplified in this region." My understanding of this is that there are a few ways nodal is amplified though I'm not sure if this is specifically occurs on the opposite side of the AVE. 1. pronodal cleaved by protease -> active nodal 2. Nodal -> BMP4 -> Wnt-> nodal 3. Nodal-> Nodal, Fox1 binding site 4. BMP4 on outside-> nodal Are all of these occuring opposite to AVE?arrow_forward
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Intro to Cell Signaling; Author: Amoeba Sisters;https://www.youtube.com/watch?v=-dbRterutHY;License: Standard youtube license