1 SEM ACC W/RAVEN CARDED
12th Edition
ISBN: 9781265486297
Author: Raven
Publisher: MCGRAW-HILL HIGHER EDUCATION
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Textbook Question
Chapter 10, Problem 2S
Review you knowledge of signaling pathways (chapter 9). Create an outline illustrating how a growth factor (ligand) can lead to the production of a cyclin protein that would trigger S phase.
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Chapter 10 Solutions
1 SEM ACC W/RAVEN CARDED
Ch. 10.1 - Prob. 1LOCh. 10.2 - Prob. 1LOCh. 10.2 - Distinguish between homologues and sister...Ch. 10.2 - Prob. 3LOCh. 10.3 - Describe the eukaryotic cell cycle.Ch. 10.4 - Prob. 1LOCh. 10.4 - Prob. 2LOCh. 10.5 - Describe the phases of mitosis.Ch. 10.5 - Explain the importance of metaphase.Ch. 10.5 - Compare cytokinesis in plants and animals.
Ch. 10.6 - Prob. 1LOCh. 10.6 - Prob. 2LOCh. 10.7 - Prob. 1LOCh. 10.7 - Prob. 2LOCh. 10 - Binary fission in prokaryotes does not require the...Ch. 10 - Chromatin is composed of a. RNA and protein. b....Ch. 10 - What is a nucleosome? a. A region in the cells...Ch. 10 - What is the role of cohesin proteins in cell...Ch. 10 - The kinetochore is a structure that functions to...Ch. 10 - Separation of the sister chromatids occurs during...Ch. 10 - Why is cytokinesis an important part of cell...Ch. 10 - What steps in the cell cycle represent...Ch. 10 - Cyclin-dependent kinases (Cdks) are regulated by...Ch. 10 - Prob. 2ACh. 10 - Genetically, proto-oncogenes act in a dominant...Ch. 10 - The metaphase to anaphase transition involves a....Ch. 10 - The main difference between bacterial cell...Ch. 10 - In animal cells, cytokinesis is accomplished by a...Ch. 10 - Regulation of the cell cycle is very complex and...Ch. 10 - Review you knowledge of signaling pathways...Ch. 10 - Compare and contrast how mutations in cellular...
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Need a deep-dive on the concept behind this application? Look no further. Learn more about this topic, biology and related others by exploring similar questions and additional content below.Similar questions
- When a mitogen binds to a receptor on a cell, the signal is transported via a signaling pathway into the cell and a new cell cycle can start. Describe this signal path. Explain what would happen to this one signal path if the cell simultaneously detects that there is damaged DNA in the cell.arrow_forwardgive answer asap with explanationarrow_forwardHow does MPF trigger M phase of the cell cycle? A. MPF phosphorylates proteins that triggers the activation of M phase B. MPF has a regulatory role in breakdown of the nuclear envelope during M phase C. MPF is a dimer subunit that is turned on during S phase to aid with chromosome replication D. MPF Cdk subunits regulate M phasearrow_forward
- Consider the estrogen receptor (ER), a cell-cell signaling molecule which can be mutated to caus3 cancer. ER has a very short half life of 30 minutes. Consider a cell that starts out with 20,000 molecules of estrogen receptor at the start of your experiemnt. Ar t=0 you treat the cell. At t=0 you treat the cell with two drugs. 1)alpha-amanitin, a poisonous chemical from mushrooms that inhibits the enzymatic activity of RNA polymerase (this is why you should know your mushrooms) and 2) cycloheximide, a drug that inhibits translation by the ribosome. Under these conditions, how many molecules of ER protein will be present at the following times?arrow_forwardWhat would be the most likely effect of a mutation that causes cyclin B to be unable to bind to CDK? a. Cells pass through the G2/M checkpoint and enter mitosis even when DNA has not been replicated. b. Cells never pass through the G1/S checkpoint. c. Cells pass through mitosis more quickly than nonmutated cells. d. Cells fail to pass through the G2/M checkpoint and do not enter mitosis.arrow_forwardDescribe what happens to the G1 cell cycle check point of a skin cell that is stimulated to divide by neighboring cells following an injury. Describe the pathway from the growth factor signal to the cell cycle checkpoint regulation of gene expression resulting in cell duplication. Briefly explaining the mechanismarrow_forward
- The diagram below shows a model for the normal regulation of the transition to anaphase during mitosis. 1. Signals are released by unattached kinetochores that cause MAD to bind CDC20. MAD CDC20 6. Chromatids separate as mitosis procoeds to anaphase. COHESIN 3. The APCCDC20 CDC20 complex ubiquinates Securin. APC 2. When all kinetochores are attached, MAD releases CDC20 which then combines with APC UBIQUITIN SEPARASE . Separase destroys Cohesin APC SEPARASE SECURIN 4. Securin is destroyed, releasing Separase Your analyses of cancer cells that do not regulate this transition normally indicate that: MAD is binding to CDC20; • Ubiquitin is not attached to securin; • Sister chromatids separate and move to opposite poles of the cell. Which of the following explanations could account for all of your observations? a. The cell has an alteration in MAD that prevents it from releasing CDC20 when the 'unattached kinetochore' signal stops. O b. More than one of the other answer choices are correct.…arrow_forwardPlease answer the two belowarrow_forwardlist some reason why a cell that has just completed cytokinesis might enter the G0 phase instead of the g1 phase.arrow_forward
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