68) When Sperry and colleagues ablated the dorsal RGCs, the remaining ventral RGCS projectedto the dorsal tectum. Conversely, when they ablated the ventral RGCs, the remaining dorsalRGCS projected to the ventral tectum.of suEphrins and Ephs are distributed in dorsoventral gradients in both structures. EphB1 isexpressed in the tectum, in a low-dorsal to high-ventral gradient. Ephrin-B1 and ephrin-B2 are expressed in the retina in a high-dorsal to low-ventral gradient. What do theseexpression patterns suggest about the mechanism used for dorsoventral patterning ofRGCS projecting to the tectum? (3-4 sentences Max.

The expression patterns of ephrins and Ephs suggest that the dorsoventral patterning of RGC…

Answered: When Sperry and colleagues ablated the…

Human Anatomy & Physiology (11th Edition)

11th Edition

ISBN:9780134580999

Author:Elaine N. Marieb, Katja N. Hoehn

Publisher:Elaine N. Marieb, Katja N. Hoehn

Chapter1: The Human Body: An Orientation

Section: Chapter Questions

Problem 1RQ: The correct sequence of levels forming the structural hierarchy is A. (a) organ, organ system,...

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A) Hypothesis a Null Hypothesis or an Alternate/Alternative Hypothesis In the background section the authors wrote “We hypothesized that activated microglia might disrupt synapses, resulting in cognitive disturbance in the context of euthyroid HT, and designed the present study to test this hypothesis.” Is their hypothesis a null hypothesis or an alternate/alternative hypothesis? In the background section the authors wrote “Based on these findings, we hypothesized that neonatal brain injury may be associated with NEC.” Is this a null hypothesis or an alternate/alternative hypothesis?
Suppose that you measured binding to the isolated EGF receptor of EGFat various concentrations. Would you expect the binding curve to be hyperbolic? Explain your answer.
dattu1
please answer 15,16,17
You decide to investigate cell signaling of a pair of newly identified GPCRs, GPCR-W and GPCR-Z. Each binds the same ligand, but activates different downstream heterotrimeric G-proteins that act on adenylyl cyclase. You discover that ligand binding has opposite effects on adenylyl cyclase activity for each receptor. GPCR-W causes an increase in adenylyl cyclase activity, while GPCR-Z causes a decrease in adenylyl cyclase activity. You obtain a cell line expressing GPCR-W, GPCR-Z, the relevant G-proteins, and adenylyl cyclase. There is baseline adenylyl cyclase activity producing a baseline amount of cAMP. You embark on a research project to characterize the following mutations in the components of the signaling pathway. 2. Will each of the following mutations increase or decrease the levels of cAMP inside the cell upon adding the ligand to the cell culture? A mutation in GPCR-W that prevents G-protein activation? A mutation in GPCR-Z that prevents G-protein activation? A mutation in…
Using molecular cloning, a hybrid receptoer is produced that consists of the extracellular domain of the B-adrenergic receptor (B-AR) and the transmembrane and intracellular domains of the EGF receptor (EGFR). Question: What is the stoichiometric ratio of epinephrine to the hybrid receptor (B-AR-EGFR)? 1:1 O 1:4 O 1:2 2:1 O O O
Looking at Fig. 3a and 3b, how do you know that 3a has a significant difference and that Fig. 3b does not? In the euthyroid HT induces ultrastructure changes in frontal synapses section of the results section the authors wrote “Quantitatively, the ultrastructure of the frontal lobe neurons revealed lower synaptic density (Fig. 5c), fewer presynaptic vesicles (Fig. 5d), shorter active zone length (Fig. 5e), thinner postsynaptic densities (Fig. 5g), and decreased synaptic curvature (Fig. 5h) in the HT mice compared with those in the Con mice.” Based on this quote would you expect nerve transmission to be faster or slower in HT mice?
The third interstitial nucleus of the anterior hypothalamus (INAH3) is generally larger in chromosomal male (XY) humans than in chromosomal females (XX). This is thought to be due to the effects of testosterone during fetal development. What else does research suggest about the INAH3 brain region? A) In male-to-female transgender individuals, it is more similar in size to cis-gender females. B) In male-to-female transgender individuals, it is more similar in size to cis-gender males. C) There has been no research connecting the stuctures in this brain region to differences in gender identity. D)It is the same size in all transgender individuals regardless of chromosomal sex.
Ocular dominance columns result from competition between LGN neurons of the opposite eyes for Stellate cells . (a) What is the key "ingredient" that enables a neuron to successfully compete for Stellate cells? (b) How is this ingredient demonstrated by Hubel and Wiesel using cats? (c) How is this ingredient demonstrated in more recent experiments using a toxin?
Trying to understand this part of the article “As white matter integrity did not return to baseline levels for any inter-injury interval (and did not trend towards returning to baseline levels in the case of the optic tract), it is possible that the window of vulnerability associated with our model may never close.” Based on the quote, what did not return when white matter integrity did not return? Artical Link: https://doi.org/10.1186/s12868-022-00730-y
Please both explain and give answer
Which of the following would you expect to happen if amino acids 579 to 582 (YIYV) in the PDGF-Beta receptor were exchanged with amino acids 992 to 995 (YLIP) from the EGF receptor to give PDGF (YLIP) and EGF (YIYV) (Select all that apply)? (A) (B) PDGF-B receptor Src $8 & P 579 P 581 YIYV PI3KY P 740 YMDM NCK-a P 751 YVPM P 771 YMAP GAP NCK-B SHP2 PLC-y P P 1009 YTAV 1021 YIIP ala 845 YHAE 992 YLIP P 1045 YSSD 1068 YINQ 1086 YHNK 1148 YQQD P 1173 YLRV P P P P P EGF receptor STAT3/5 PLC-y Shc Cbl JAK2 Grb2 STAT3/5 Shc SHP1 PTP1

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