II 1 2 3 4 2 III 2 3 individual II-3, is affected. So are all her three children. However, Erza's me a does not show any of the symptoms of this mitochondrial disease. are two possible explanations for this?

II 1 2 3 4 2 III 2 3 individual II-3, is affected. So are all her three children. However, Erza's me a does not show any of the symptoms of this mitochondrial disease. are two possible explanations for this?

Name: TSParagraph15......1 HAC VINIR213HE4Question 9:A rare mitochondrial d
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Description: TSParagraph15......1 HAC VINIR213HE4Question 9:A rare mitochondrial disease is shown in this pedigree.12II354I2III23Erza individual II-3, is affected. So are all her three children. However, Erza's motherHinata does not show any of the symptoms of t

Human Heredity: Principles and Issues (MindTap Course List)

11th Edition

ISBN:9781305251052

Author:Michael Cummings

Publisher:Michael Cummings

Chapter10: From Proteins To Phenotypes

Section: Chapter Questions

Problem 15QP: Phenylketonuria and alkaptonuria are both autosomal recessive diseases. If a person with PKU marries...

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In addition to the predominant adult hemoglobin,HbA, which contains two α-globin chains and twoβ-globin chains (α2β2), there is a minor hemoglobin,HbA2, composed of two α and two δ chains (α2δ2).The β- and δ-globin genes are arranged in tandem andare highly homologous. Draw the chromosomes thatwould result from an event of unequal crossing-overbetween the β and δ genes
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From the pedigree in Figure 3-25, what principle can youdeduce about the inheritance of mitochondrial diseasefrom affected fathers?
How many affected females and males does this pedigree show? (2) Who is the proband? (3) How many generations are included? (4) What kind of inheritance pattern is the disease-causing allele in this pedigree? Why? (5) Who have the same genotype as the proband? Why?
Phenylketonuria and alkaptonuria are both autosomal recessive diseases. If a person with PKU marries a person with AKU, what will the phenotype of their children be?
could you plz help with part c on this problem thank you!
7.7 Genes a and b are 20 cM apart. An a* b*la* b* individual was mated with an a bla b individual. (a) Diagram the cross and show the gametes produced by each parent and the genotype of the F₁. (b) What gametes can the F, produce, and in what proportions? (c) If the F, was crossed to a bla b individuals, what off- spring would be expected, and in what proportions? (d) Is this an example of the coupling or repulsion link- age phase? (e) If the F, were intercrossed, what offspring would be expected, and in what proportions?
3.9 Which mode of inheritance is suggested by the fol- lowing pedigree? Based on this hypothesis, and assuming that the trait is rare and has complete pen- etrance, what are the possible genotypes of all indi- viduals in this pedigree? norative
1. The pedigree below shows the incidence of rare, autosomal dominant disorder called Ehlers-Danlos disease. The pedigree covers three generations of a particular family and also shows individual genotypes at a potential marker locus (M). a) Indicate the phase of all gen II and III individuals. DdM1M3 ddM2M6 II DDM3M6 ddM4M5 III DdMзM4 DdMЗМ5 DDM3M4 ddM3M5 DDM3M4 ddM5M6 DDM3M4 ddM4M6 ddM5M6 ddM5M6 b) Which, if any, of the gen III individuals are recombinants? c) Calculate the LOD score as a test of physical linkage between the marker (M) and the disease locus. d) What do you conclude about linkage between D and M?