If action potential propagation from one neuron to another neuron requires AChrelease, what event at the synaptic cleft explains the need for action potentialsto arrive at a high rate before the post-synaptic membrane achieves threshold?the lack of sodium voltage gated channels on the post-synaptic membraneO the rapid breakdown of ACh by ACHE at the synaptic cleftthe slow release of ACh by the synaptic vessiclesthe slow movement of sodium through chemical gatesPreviousNextMacBook Air

Introduction: The brief sequence of events in the membrane potential following excitation is…

Answered: If action potential propagation from…

Human Anatomy & Physiology (11th Edition)

11th Edition

ISBN:9780134580999

Author:Elaine N. Marieb, Katja N. Hoehn

Publisher:Elaine N. Marieb, Katja N. Hoehn

Chapter1: The Human Body: An Orientation

Section: Chapter Questions

Problem 1RQ: The correct sequence of levels forming the structural hierarchy is A. (a) organ, organ system,...

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Similar questions

Which of the following situations in a neuron results in action potentials that have extended depolarization phases? O None of these result in extended depolarization phase. O Compromising the inactivation mechanism in voltage-gated Na* channels. O Stimulating the voltage-gated K* channels. O Inhibiting the voltage-gated Na* channels. O Compromising the inactivation mechanism in voltage-gated K* channels.
What stage of the action potential is illustrated by the set up shown? interstitial fuid axon cytoplasm interstitial fiuid Resting potential Hyperpolarization O Depolarization Repolarization K'charnel
Which very specific synapse will produce the fastest propagation of an action potential? axosomatic synapse spine synapse axodendritic synapse spinosomatic shaft synapse
A theoretical drug blocks Na* leak channels - what effect would this have on a neuron's resting membrane potential and why? Would this drug affect the neuron's ability to generate an action potential and why/why not?
A muscle cell contains acetylcholine receptors that act as ligand-gated cation channels,and it also contains voltage-gated Na+ channels of the type found in neurons. Intheory, could a muscle cell get away with having only acetylcholine-gated cationchannels, which would serve the dual purpose of receiving neurotransmitter signalsand propagating action potentials? Why or why not?
In terms of function when a presynaptic neuron receiving input from a nearby inhibitory neuron, it can be inferred that the both neurons firing won't be synchronized activation of the inhibitory neuron is nullified activation of the presynaptic neurotransmitter release is enhanced this type of interaction represents a regulatory input of neuronal activation
8. a) Label the following diagram of a synapse with the following terms: axon terminal, synaptic cleft, synaptic vesicles, pre-synaptic membrane, post-synaptic membrane. A SYNAPSE Mitochondrion produce ATP Dendrite of second neuron
1. Action potential down a neuron: For each step of an action potential, provide a brief summary of events, the particular membrane potential voltage number it should be, and which ion is moving in which direction. A ' B C D E
Which statement is true regarding chemical neurotransmission? O An IPSP closer to the soma will diminish the effects of an EPSP on a dendrite Chemical neurotransmission is mediated by gap junctions All neurotransmitter receptors are located on the postsynaptic cell membrane Neurotransmitters remain in the synaptic cleft indefinitely
Synaptic terminal Neurotransmitter SENDING NEURON Vesicle Calcium channel Receptor for neurotransmitter RECEIVING NEURON Synaptic cleft
Using a stimulus intensity of 65 µA, produce an action potential in the simulated neuron on Metaneuron. What happens when tetrodotoxin is applied? Select the (one) CORRECT answer. O a. an action potential occurs O b.a small depolarisation, but no action potential occurs O c. no depolarisation occurs O d. the neuron becomes hyperpolarised
Describe the Neuronal Excitation Contraction Coupling and Describe the role of calcium as the coupler between the neuronal excitation and sliding filament theory

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