Consider a gene in the mitochondrial genome that codes for a channel protein in the inner membrane that allows magnesium ions to travel freely across. If a mutation in that gene resulted in this protein allowing H+ to pass through it rather than magnesium, what would be the consequence for cellular respiration?
Consider a gene in the mitochondrial genome that codes for a channel protein in the inner membrane that allows magnesium ions to travel freely across. If a mutation in that gene resulted in this protein allowing H+ to pass through it rather than magnesium, what would be the consequence for cellular respiration?
Human Anatomy & Physiology (11th Edition)
11th Edition
ISBN:9780134580999
Author:Elaine N. Marieb, Katja N. Hoehn
Publisher:Elaine N. Marieb, Katja N. Hoehn
Chapter1: The Human Body: An Orientation
Section: Chapter Questions
Problem 1RQ: The correct sequence of levels forming the structural hierarchy is A. (a) organ, organ system,...
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Consider a gene in the mitochondrial genome that codes for a channel protein in the inner membrane that allows magnesium ions to travel freely across. If a mutation in that gene resulted in this protein allowing H+ to pass through it rather than magnesium, what would be the consequence for
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Step 1 Introduction:
Mitochondria are membrane-bound cell organelles that generate most of the chemical energy needed to power the cell's biochemical reactions. Chemical energy produced by the mitochondria is stored in a small molecule called adenosine triphosphate.
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