Why would a person who is fasting have high levels of acetyl-CoA?Match the words in the left column to the appropriate blanks in the sentence on the right.body fatFasting decreases the intake ofmuscle proteinsbody breaks downmolecules.carbohydratesfiberglycogenResetwhich is needed for energy. Without this/them, thefor energy. This results in the production of many acetyl-CoAHelp

Hepatic glycogenolysis is the principal source of glucose during the initial hours of fasting. The…

Answered: body fat muscle proteins carbohydrates…

Biochemistry

9th Edition

ISBN:9781319114671

Author:Lubert Stryer, Jeremy M. Berg, John L. Tymoczko, Gregory J. Gatto Jr.

Publisher:Lubert Stryer, Jeremy M. Berg, John L. Tymoczko, Gregory J. Gatto Jr.

Chapter1: Biochemistry: An Evolving Science

Section: Chapter Questions

Problem 1P

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Similar questions

Which of the following best explain lactose intolerance? Inability to digest lactose and causes digestive issues such as cramping and bloating. O Inability to digest maltase and releases excess maltase in the blood. Inability to digest lactose and releases excess lactose in the blood. Inability to produce the maltase enzyme and causes digestive issues such as cramping and bloating
Choose all of the molecules that are made in acetyl-CoA formation and the citric acid cycle that are important to note. NADH FADH2 CO2 ATP pyruvate glucose
Glucagon activates the enzyme glycogen synthase to stimulate glycogenolysis. O True O False
Compare and contrast glycogen synthesis/degradation in muscles as compared with the liver.
What molecule provides the energy for fatty acid synthesis starting from acetyl-CoA and malonyl-CoA as substrates? GTP NADH NADPH 4 FADH2 (2)
Can someone help with this question?
WHERE in the cell the reaction occurs? WHY the reaction occurs (in a general sense; Why does the cell do this?) HOW the reactions occur (in a general sense; not all the details- just a couple sentences) VISUALLY could you recognize images of any of these reactions?
If our body lack of insulin, is it mean the glucose in our blood is low concentration now. So is it will effect pathways in mucles cell like glycolysis, fatty acids synthesis, glycogen synthesis, gluconeogenesis (increase or decrease)? I know the liver can regconize that low or high concentration of blood glucose so insulin or glucagon can act as the signal for liver can know, but is the muscle cell can do it to? If not, how they know to adjust their pathways. And if our body lack of insulin, is the liver cells response same as glucagon signaling.
What molecule forms when Acetyl-CoA enters the Krebs cycle? ATP pyruvic acid glucose citric acid isocitrate
After absorption, nearly all fructose enters hepatocytes (liver cells) on the first pass and enters glycolysis for energy production, or if consumed in excess will get transformed into triglycerides. True False
How do glucagon and epinephrine affect glycogen in the liver? They increase both glycogen breakdown and gluconeogenesis. They increase both glycogen breakdown and glycolysis. They increase glycolysis to breakdown glucose. They increase glycogenin synthesis.
Triacylglycerol is the primary way that we store fat, once we have made our fatty acid chains we can then make our stored fats by linking them to what? This is this pathway called?

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body fat muscle proteins carbohydrates fiber glycogen Fasting decreases the intake of body breaks down molecules. which is needed for energy. Without this/them, the for energy. This results in the production of many acetyl-CoA