A young child with normal development had diarrhea, vomiting, and abdominal pain after eating. Exclusion of milk from the diet did not give a positive result. After feeding with sucrose, the level of glucose in the blood barely increased. Why could such symptoms occur? hereditary lactase deficiency hereditary insufficiency of sucrase disorders of transporters secondary lactase deficiency deficiency of amylase

A young child with normal development had diarrhea, vomiting, and abdominal pain after eating. Exclusion of milk from the diet did not give a positive result. After feeding with sucrose, the level of glucose in the blood barely increased. Why could such symptoms occur? hereditary lactase deficiency hereditary insufficiency of sucrase disorders of transporters secondary lactase deficiency deficiency of amylase

Biochemistry

9th Edition

ISBN:9781319114671

Author:Lubert Stryer, Jeremy M. Berg, John L. Tymoczko, Gregory J. Gatto Jr.

Publisher:Lubert Stryer, Jeremy M. Berg, John L. Tymoczko, Gregory J. Gatto Jr.

Chapter1: Biochemistry: An Evolving Science

Section: Chapter Questions

Problem 1P

See similar textbooks

Similar questions

Please indicate whether the fallowing statement is true or not? ''The net flow of glucose is inward if glucose concentration is higher inside than outside of the cell''
GluT transporters are responsible for passive transport of Glucose into the cell to yield G-6-P that cannot exit the cell; in liver cells this process is always regulated by insulin. True or False
All defects in the urea cycle lead to an elevated level of ammonium ion in blood, which is called: hyperammonemia. hypoammonemia. toxaemia. ammonemia. anemia.
What is the effect of the cholesterol diet on the amount of HMGCoA reductase protein?
Explain why reduction in ghrelin secretion in bariatric surgery would be beneficial on glucose homeostasis?
Explain why blood glucose after consumption of milk can indicate someone’s ability to digest lactose.
Saponification is a process by which triacylglycerols are hydrolyzed to produce glycerol and fatty acid salt by the action of a strong acid. True False
Hemoglobin molecules exposed to high levels of glucose are converted to glycated products. The glycated product used to measure blood glucose control in diabetics is hemoglobin A1C (HbA1C), contained in red blood cells that last around 3 months. In general terms, explain how HbA1C forms and why it is a useful marker for blood glucose control.
Pancreatic β cells express a receptor for fatty acids. Fatty acid binding to the protein appears to stimulate insulin secretion. Does this phenomenon make metabolic sense?
explain how a mutation could prevent the proper expression of amylase and what the consequences of not expressing amylase would be.
identify one Glycogen storage diseases and explain the pathway affected