Prescott's Microbiology
10th Edition
ISBN: 9781259281594
Author: Joanne Willey, Linda Sherwood Adjunt Professor Lecturer, Christopher J. Woolverton Professor
Publisher: McGraw-Hill Education
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Chapter 37.7, Problem 1RIA
Summary Introduction
The epidemic is a sudden spread of disease in a population with a wide spread of infection rate that affects most of the population within a short period of time. For example, Cholera. The epidemic can be a common source epidemic or propagated epidemic.
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Discuss how the SARS-CoV-2 virus causes Covid-19 disease became a "new" zoonotic disease and has resulted in a "fast" spreading World-Wide Pandemic. how has it evolute as a zoonotic disease, the mechanism of infection in humans, symptoms of disease, treatment and prevention. Describe how to "flatten" the curve and make the epidemic a "slow" epidemic by acting as a "social" vaccine.
5) Consider the following simulation result:
120
100
80
60
40
20
0
10
20
30
c) When does the epidemic peak?
40
50
Time
60
b) What proportion of the population gets infected?
70
a) What model do you think this simulation result is from?
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d) Suppose 3 = 0.005 and y = 0.1. Calculate Ro for this model.
100
e) Can you stop the epidemic by adding more people to the population? If so, how many people
would you need to add?
The Zoomland epidemic does not mimic a real epidemic in every respect. What is ONE way the Zoomland epidemic is not an entirely realistic simulation?
A.
In the Zoomland epidemic we could tell who had interacted with whom (contact tracing). In real life there is no way to do any contact tracing.
B.
In the Zoomland epidemic people interacted randomly with others. In real life people interact much more with some members of the population than others.
C.
In the Zoomland epidemic, transmission only occurred when a person came into contact with an infected person AND a suitable vector. In real life some diseases do not require a vector for successful transmission.
Chapter 37 Solutions
Prescott's Microbiology
Ch. 37.2 - How might biased data collection impact the public...Ch. 37.2 - Prob. 1RIACh. 37.2 - Prob. 2RIACh. 37.2 - Prob. 3RIACh. 37.2 - Prob. 4RIACh. 37.2 - Prob. 5RIACh. 37.3 - Prob. 1MICh. 37.3 - Prob. 2MICh. 37.3 - Prob. 1RIACh. 37.3 - Prob. 2RIA
Ch. 37.3 - Prob. 3RIACh. 37.3 - Prob. 4RIACh. 37.4 - Prob. 1RIACh. 37.4 - What are some of the factors in your community...Ch. 37.4 - Prob. 3RIACh. 37.5 - Describe a health-careassociated infection, and...Ch. 37.5 - Prob. 2RIACh. 37.5 - Prob. 3RIACh. 37.7 - Prob. 1RIACh. 37.7 - Prob. 2RIACh. 37.7 - Prob. 3RIACh. 37.7 - What is the Public Health Security and...Ch. 37 - Why is international cooperation a necessity in...Ch. 37 - What common sources of infectious disease are...Ch. 37 - Prob. 3CHICh. 37 - How can changes in herd immunity contribute to an...Ch. 37 - College dormitories are notorious for outbreaks of...Ch. 37 - Prob. 6CHICh. 37 - Prob. 7CHICh. 37 - Prob. 8CHI
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- (c) Consider spread of an epidemic of some infectious disease in some spatially distributed population of individuals, e.g. spread of Covid-19, or flu. List characteristics of such epidemic which a bottom up approach to mathematical modeling allows incorporating more realistically, or more easily, than a top down approach.arrow_forwardWhat is disease causation theory? Define personalistic and naturalistic disease causation using films and/or readings.arrow_forwarddiscuss methods used to control vector-borne diseases, specifically mosquitos. 1) Is there one method you think is more beneficial than others when it comes to the controlling and prevention of mosquito-borne diseases? Why/why not? 2) Also, how large do you think is the impact of human environmental change (man's activities) on emerging infectious diseases in animals and man?arrow_forward
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- Why is it important to calculate disease rates to report disease outbreaks accurately?arrow_forwardWhat are the modifiable and non modifiable risk factors involved in multi causation disease model ? Like: environmental conditions, medical care, social circumstances, behavior choices, geneticarrow_forwardExplain the relationship between the three levels of prevention and the prevalence and/or incidence of a specific disease of your choice in a specific population. What are the health disparities for this disease and population you chose? Which of the three levels of prevention do you think receives the most public attention and resource allocation to address this disease? What do you think the rationale is and why?arrow_forward
- This article highlights a young doctor at Elmhurst Hospital during the beginning of Covid-19 pandemic. Dr. Zikry is quoted as saying: “It’s become very clear to me what a socioeconomic disease this is...”. In addition, the textbook discusses the personal variables and socioeconomic status (SES) that are used to find patterns in disease (pp.112-118). What do you think Dr. Zikry meant by referring to the SES of his patients? Why was it important to find a pattern of personal variables and SES among the of victims of Covid-19 at the beginning of the pandemic?arrow_forwardDescribe how we can use Epidemiology to explain and predict the Ebola outbreak. What kind of questions should we attempt to answer?? Only typed answerarrow_forwardPathogen A has an ID50 of 10 cells and an LD50 of 10,000,000 cells. Pathogen B has an ID50 of 10,000 cells and an LD50 of 20,000 cells. From this information, we can determine that: ○ Pathogen A has higher infectivity but lower virulence than Pathogen B. ○ Pathogen A has lower infectivity but higher virulence than Pathogen B. ○ Pathogen B has higher infectivity but lower virulence than Pathogen A. ○ Pathogen B has higher infectivity and higher virulence than Pathogen A.arrow_forward
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