What is the medial forebrain bundle? Discuss the evidence for and against its involvement in reward. In addition, review the evidence showing that the brain’s dopamine pathways are critically involved in pleasure and reward. Please include peer reviewed references.
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What is the medial forebrain bundle? Discuss the evidence for and against its involvement in reward. In addition, review the evidence showing that the brain’s dopamine pathways are critically involved in pleasure and reward. Please include peer reviewed references.
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- Briefly describe our brain's mesolimbic dopamine system. (A) Which brain structures are involved in this system? (B) What type of behavior does it regulate? (C) Even though social media (or video games and the like) isn't something ingested, do you think this system is involved in our social media behavior? Or do you think something else is involved? Please provide a brief, but specific response. Answer as much as you can please!!!!!!! Thank you!!!!!!Propose reagents for the conversion of (B) to (C).Limbic System, Emotion, Reward, and Addiction Describe the role of the amygdala in emotion at the introduction level (Introduction to Neuroscience College Level). Provide many of your own digital or handwritten diagrams to complement your explanation.
- What is Brain-derived neurotrophic factor (BDNF)? What connection does it have to exercise, nature and cellphones?Introduction to Neuroscience Which of the following does NOT contribute to the pathology of Alzheimer’s disease? Group of answer choices beta amyloid plaques neurofibrillary tangles excess activity in the hippocampus loss of acetylcholine containing neurons all of the above contribute Please explain your explain full detail and explain your reasoning in detail.Selective serotonin reuptake inhibitors (SSRIs) are drugs that can alleviate symptoms of depression by blocking the reuptake of serotonin (5-HT) from the synaptic cleft, thereby increasing the amount of time that 5-HT remains active. Elevated levels of 5-HT within the synapse are associated with feelings of well-being; conversely, low levels of 5-HT are correlated with depressive symptoms. Recent studies have shown that SSRIs can also mediate their antidepressant effects by increasing brain levels of certain cytokines, including interferon gamma (IFNY). IFNY directly induces the expression of the protein p11 in neighboring neurons, which then interacts with 5-HTR4, a 5-HT transmembrane receptor. Figures 1 and 2 provide information about this interaction. 5-HTR4 protein (% of WT) expression CAMP levels (% change control) from 120T 100+ 80+ 60+ 40+ 20+ 0 MEM TOT Figure 1 5-HTR4 protein expression in plasma membrane-enriched fraction (MEM) of hippocampal lysate and in total hippocampal…
- What is localization of function? Describe how localization has been demonstrated by neuropsychology and recording from neurons. Be sure you understand the principle of double dissociations.Identify two neural pathways in the brain that use dopamine as a neurotransmitter, andexplain their significance.Alzheimer’s disease is a devastating neurodegenerative disease. Discuss approaches used to study the disease, giving examples of specific techniques and models, and evaluating their advantages and disadvantages. Discuss possible reasons for why effective treatments have not yet been developed. Use figures and diagrams to illustrate your answer. (This is a neuroscience question)
- discuss the excitatory NMDA receptor, the role of the Mg++ ion, and how NMDA activation can produce Long Term Potentiation (which could underlie memory). Finally, what is the seemingly unique involvement of neighboring glia cells in helping to regulate both GABA and Glutamate neurotransmission and why might that glia cell role occurDescribe brain imaging evidence for localization of function. Describe experiments thatinvolved looking at still pictures and that involved looking at movies. What does eachtype of experiment tell us about localization of function?Briefly describe how silent glutamatergic synapses become activated during long term potentiation, describing all of the key neurotransmitters and their post-synaptic receptors and events that give rise to the process.