The diet Sarah detailed does not meet the requirements for EFAs (essential fatty acids). Why would Sarahwant to be sure to meet these requirements? Choose the ALL of the functions of EFAs listed below:Eicosanoids made from ALAS are often less potent mediators of inflammation than those made fromLAs.ALAS are not required to eat as humans can make them.Omega-6 (LA) consumption is linked to reduction of the incidence of CHD and reduces blood cholesterollevels.Omega-3 (ALA) consumption is linked to reduction of chronic inflammatory-based condition incidenceand severity like cardiovascular disease (CVD) and type 2 diabetes.EFAs form structural components of cell membranes - impact fluidity, flexibility, permeability, and theactivity of membrane-bound enzymes and cell-signaling pathways.OALAS are proven to make her smarter.

Sarah would want to be sure to meet the requirements for essential fatty acids (EFAs) because they…

Answered: The diet Sarah detailed does not meet…

Human Physiology: From Cells to Systems (MindTap Course List)

9th Edition

ISBN:9781285866932

Author:Lauralee Sherwood

Publisher:Lauralee Sherwood

Chapter3: The Plasma Membrane And Membrane Potential

Section: Chapter Questions

Problem 1ACR: When William H. was helping victims after a devastating earthquake in a region not prepared to...

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The conjugation of bile acids to glycine or taurine reduces their pKa making then more amphipathic and more effective emulsifiers of fat. True False
A patient with H. pylori infection develops a gastric ulcer and is treated with omeprazole. Which of the following is/are mechanism(s) of action of omeprazole? Group of answer choices Inhibition of ACh action on parietal cells stimulation of somatostatin action on parietal cells inhibition of CCKb receptors inhibition of H+-K+ ATPase inhibition of Na+-K+ ATPase.
All of the following are true with respect to alpha-glucosidase inhibitor, except:A. It inhibits intestinal alpha-glucosidase B. It decreases enzymatic conversion of oligo- to monosaccharidesC. It lowers postprandial glucoseD. It causes gastrointestinal disturbances.E. It may cause development of hypertension.
Allosteric inhibitors of hemoglobin will decrease enzyme activity (oxygen binding) through which of the following mechanisms? Binding the enzyme in "T" conformation and displacing an activator Binding the enzyme and enhancing the Vmax Binding the enzyme, and keeping it in the "R" conformation Binding the enzyme, and keeping it in the "T" conformation
When William H. was helping victims after a devastating earthquake in a region not prepared to swiftly set up adequate temporary shelter, he developed severe diarrhea. He was diagnosed as having cholera, a disease transmitted through unsanitary water supplies contaminated by fecal material from infected indiv iduals. The toxin produced by cholera bacteria causes Cl- channels in the lurninal membranes of the intestinal cells to stay open, thereby increasing the secretion of Cl- from the cells into the intestinal tract lumen. By what mechanisms would Na+ and water be secreted into the lumen in conjunction with Cl- secretion? How does this secretory response account for the severe diarrhea that is characteristic of cholera?
Give a few food sources of each type of fat: saturated, trans, PUFA, and MUFA. Describe the difference between an EAR, RDA, and AI. Know the AMDR and DRI for total fat and PUFAs. Know recommendations for saturated, trans, MUFAs, and cholesterol. Describe the importance of LA and ALA. Describe eicosanoids and give examples of functions in the human body. Give examples of traditional vs newer(ish) perspectives on fat in the diet and its impact on health. Describe typical recommendations and rationale behind a high-Fat vs a low-fat diet
A 2-year old girl presents with steatorrhea and is diagnosed with pancreatic lipase deficiency. She is still able to digest which of the following types of triacylglycerol (TAG)? TAG with VLCFA TAG with LCFA TAG with unsaturated FA TAG with SCFA TAG with branched-chain FA
Can you please explain the diagram it has lots of parts and can you explain in simple terms as to what it's trying to show and tell Summary of the effects of hyperthyroidism and eprotirome on serum markers and metabolites in lipid metabolism. 7?-OH-cholesterol, 7?-hydroxycholesterol; BAs, bile acids; CE, cholesteryl ester; CETP, cholesteryl ester transfer protein; CM, chylomicron; CMR, chylomicron remnant; HMG-CoAR, HMG-CoA reductase; HSL, hormone sensitive lipase; SRBI, scavenger receptor class B type I.
Describe the transport and stroage of fatty acids in humans with particular reference to disease states.
Absorbed from the intestines into the blood, nitrites (NO2-) interact with the hemoglobin of the blood and block its respiratory function, turning part of the hemoglobin (HbFe2+) into methemoglobin (HbFe3+), unable to transfer oxygen from the lungs to tissues. With the formation of a large amount of methemoglobin, oxygen starvation of tissues occurs, which can cause damage to the central nervous system.10 to 20 % - of methemoglobin (HbFe3+) - asymptomatic cyanosis,20 to 50% of methemoglobin (HbFe3+) - hypoxia develop,> 50% of methemoglobin (HbFe3+) - the person will die.The body weight of the average person is 60 kg. Blood mass averages 8% of a person’s body weight; blood density ρ = 1,050 g/cm3, the hemoglobin (Hb) content in it is 14 g per 100 ml.molecular weight of hemoglobin 65-68 kg/mol (use 68 kg/mol for calculation). Assume that 1 mole of hemoglobin reacts with one mole of nitrite ion to form 1 NO molecule:HbFe2+ + NO2- → HbFe3+ + NOwill a 290 mg dose of sodium nitrite be…
Acetyl-CoA is generated by E1 and E2 ... purpose of E3? (PDHC)
Glucose has been found to react nonenzymatically with hemoglobin, through Schiff base formation between C-1 of glucose and the amino termini of the β chains. How might this finding be applied in monitoring diabetic patients?

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