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Question:-
list three sets of evidence support the adenoma to carcinoma progression in cancer development
which one is most convincing?
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- In your own words, what is angiogenesis and what is the importance of this process in cancer progression?What is meant by the "two-hit" model of cancer development? Describe this theory in detail and explain why this makes sense with the observation that cancer is typically seen in people who are >60 years of age.Despite all that we know about cancer today, some types of cancers are still increasing in frequency. Lung cancer among nonsmoking women is one of these. What reason(s) might there be for this increasing problem?
- Out of these 8 hallmarks ( sustaining proliferative signaling, evading growth suppressors, resisting cell death, enabling replicative immortality, inducing angiogenesis, activating invasion and metastasis, deregulating cellular energetics and metabolism, and avoiding immune destruction). Rank them from least important to most for cancer development. Explain in 2-3 sentences how.In your own words, explain each of the hallmarks of cancer. (immortality, produce go signals, override stop signals, resist cell death, angiogenesis and how it relates to metastasis.#1) Hyperplastic growth are not cancerous yet but show a higher than normal proliferation rate. True False #2) When cancer cells have not spread beyond its original site, the term used to describe it is benign growth intraepithelial neoplasia carcinoma stage 3 carcinoma in situ #9) Cancer cells generally have missense mutations in p53 gene, resulting in truncated p53 normally active p53 dominant negative p53 inactive p53 #10) Single or double stranded breaks in DNA activate Chk 1 and 2 kinases, which phosphorylates p53. This results in --- in the level of p53 in the cell. increase decrease
- Discuss how growth factors influence cell signaling and proliferation that leads to cancer induction, progression, and metastasis. Be sure to include the involvement of apoptosis and angiogenesis.Answer and explain your choice to the following multiple-choice questions about cancer-promoting mutations. The ABC gene undergoes multiple gene duplication events, producing multiple copies of this gene in the genome. This gene duplication event is associated with cancer. What would best describe the normal function of ABC gene? Inhibit cell growth and division Oncogene Inhibit apoptosis Metabolic enzyme EXPLAIN in 1-2 sentences: A mutation in the epidermal growth factor receptor (EGFR) causes it to send a positive signal along its intracellular signaling pathway, even without the EGF ligand bound to it. Knowing the function of a growth factor, how would you classify this mutation? an activating mutation in a tumor suppressor gene an activating mutation in a proto-oncogene a loss of function mutation in a tumor suppressor gene a loss of function mutation in a proto-oncogene EXPLAIN in 1-2 sentences: In an otherwise normal cell, what would be the immediate result of a…We know that there are several risk factors for cancer, which of those risk factors are significant for a 52 year old white male? What steps could you take could reduce that risk?
- Excessive angiogenesis and reduced apoptosis are two of the six characteristics that define cancerous tumors. Discuss how developing treatments for these two conditions might prevent the development of cancer altogether from pre-malignant tumors.The chapter points out that about one of every five women and one of every four men in the United States will die from cancer. Why are rates of death from cancer different in men and women? Provide some possible explanations.What the hallmarks of cancer are? and briefly explain two of them.
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