Match the bold DNA repair response(s) to the triggering type of DNA damage. Homologous Recombination Mishmatch Repair Base Excision Repair Nucleotide Excision Repair Non-homologous End Joining Single-strand DNA breaks Removal of repair lesions such as photoproducts caused by UV including Thymine dimers Double-Strand Break repair mechanism which is an accurate repair mechanism without any introduction of insertions or deletions. It requires a sister chromatid as a template. This repair mechanism uses just DNA glycosylase to remove
Match the bold DNA repair response(s) to the triggering type of DNA damage.
Homologous Recombination
Mishmatch Repair
Base Excision Repair
Non-homologous End Joining
Single-strand DNA breaks
Removal of repair lesions such as photoproducts caused by UV including Thymine dimers
Double-Strand Break repair mechanism which is an accurate repair mechanism without any introduction of insertions or deletions. It requires a sister chromatid as a template.
This repair mechanism uses just DNA glycosylase to remove Uracil (no other enzymes or complexes are required) then DNA polymerase can use the template stand to add the complementary base where the Uracil has been removedInterstrand Crosslink Repair
This repair mechanism is used to recognize and repair mis-incorporation of base that can arise during
Removal and replacement of modifying bases such as Uracil, 8-hyroxyguanine and others.
Double-strand Break that is termed as “Quick and Dirty” as it is more prone to cause mutations due to insertions, deletions or translocations.
This Repair mechanism can occur as an intermediate during base excision repair.
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