Jnlike SA node cardiocytes, regular cardiocytes have a resting membrane potential that is . Regular cardiocytes become stimulated when calcium and sodium pass from neighboring excited cardiocytes through gap junctions (see 1). This causes to open, resulting in the regular cardiocytes to sharply depolarize (see 2). At 3, these channels close, and open, resulting in both depolarization and sarcomere contraction. Also at 3, open, causing the cell to repolarize. The opposing forces, depolarization and repolarization) of the two channels that are open simultaneously, results in a plateau (see 4). This plateau continues until voltage-gated calcium channels close. At 5, only remains open, resulting in the cell repolarizing.
Jnlike SA node cardiocytes, regular cardiocytes have a resting membrane potential that is . Regular cardiocytes become stimulated when calcium and sodium pass from neighboring excited cardiocytes through gap junctions (see 1). This causes to open, resulting in the regular cardiocytes to sharply depolarize (see 2). At 3, these channels close, and open, resulting in both depolarization and sarcomere contraction. Also at 3, open, causing the cell to repolarize. The opposing forces, depolarization and repolarization) of the two channels that are open simultaneously, results in a plateau (see 4). This plateau continues until voltage-gated calcium channels close. At 5, only remains open, resulting in the cell repolarizing.
Human Anatomy & Physiology (11th Edition)
11th Edition
ISBN:9780134580999
Author:Elaine N. Marieb, Katja N. Hoehn
Publisher:Elaine N. Marieb, Katja N. Hoehn
Chapter1: The Human Body: An Orientation
Section: Chapter Questions
Problem 1RQ: The correct sequence of levels forming the structural hierarchy is A. (a) organ, organ system,...
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![voltage-gated calcium channels
voltage-gated sodium channels
voltage-gated potassium channels
stable
not stable
Unlike SA node cardiocytes, regular cardiocytes have a resting membrane potential that is
Regular cardiocytes become
stimulated when calcium and sodium pass from neighboring excited cardiocytes through gap junctions (see 1). This causes
to
open, resulting in the regular cardiocytes to sharply depolarize (see 2). At 3, these channels close, and
open, resulting in both
depolarization and sarcomere contraction. Also at 3,
open, causing the cell to repolarize. The opposing forces,
(depolarization and repolarization) of the two channels that are open simultaneously, results in a plateau (see 4). This plateau continues until voltage-gated
calcium channels close. At 5, only
remains open, resulting in the cell repolarizing.](/v2/_next/image?url=https%3A%2F%2Fcontent.bartleby.com%2Fqna-images%2Fquestion%2F53083124-3bd2-451e-8d96-efadb662cb23%2F07f44988-821d-4a47-afb2-e8a1feccbda6%2Fqou666f_processed.png&w=3840&q=75)
Transcribed Image Text:voltage-gated calcium channels
voltage-gated sodium channels
voltage-gated potassium channels
stable
not stable
Unlike SA node cardiocytes, regular cardiocytes have a resting membrane potential that is
Regular cardiocytes become
stimulated when calcium and sodium pass from neighboring excited cardiocytes through gap junctions (see 1). This causes
to
open, resulting in the regular cardiocytes to sharply depolarize (see 2). At 3, these channels close, and
open, resulting in both
depolarization and sarcomere contraction. Also at 3,
open, causing the cell to repolarize. The opposing forces,
(depolarization and repolarization) of the two channels that are open simultaneously, results in a plateau (see 4). This plateau continues until voltage-gated
calcium channels close. At 5, only
remains open, resulting in the cell repolarizing.
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