3. How many molecules of ATP are generated when a. stearate (18:0), b. oleate (18:1 cisA9), and c. heptadecanoic (17:0) acid are completely oxidized via ß oxidation?Notes: Perform three separate calculations. Attempt one in class and save the rest forhomework.

β-oxidation is the process by which fatty acids are broken down to their acetyl-CoA (2 carbon…

Answered: How many molecules of ATP are generated…

Biochemistry

9th Edition

ISBN:9781319114671

Author:Lubert Stryer, Jeremy M. Berg, John L. Tymoczko, Gregory J. Gatto Jr.

Publisher:Lubert Stryer, Jeremy M. Berg, John L. Tymoczko, Gregory J. Gatto Jr.

Chapter1: Biochemistry: An Evolving Science

Section: Chapter Questions

Problem 1P

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In the complete b-oxidation of stearic acid (18:0), a)how many moles of acetyl CoA are produced? ________ b)how many times would the fatty acid pass through the fatty acid spiral for complete oxidation? ________ c)what is the total ATP produced in the citric acid cycle? ________ d)how many moles of NADH are produced? ________ e)how many moles of FADH2 are produced? ________ f)how many moles of ATP are produced after all NADH are reoxidized in the respiratory chain? ________ g)how many moles of ATP are produced after all FADH2 are…
1- Catalyzes the production of FADH, in the Krebs cycle: a) isocitrate dehydrogenase b) c) fumarasa aconitase d) a-ketoglutarate dehydrogenase e) succinate dehydrogenase 2- In the Krebs cycle (a single cycle) the following are released: a) one molecule of CO, b) three molecules of NADH c) two FADH2 molecule d) two GTP 3- The enzyme that converts citrate to isocitrate is known What: b) isocitrate dehydrogenase a) aconitase citrate synthetase d) citrate lyase e) fumarase c)
How many cycles of B-oxidation are necessary to completely oxidize 3 molecules stearic acid (C18H3602)?a. 24b. 180 27O 21
E3 of the pyruvate dehydrogenase complex performs which main function? A) removing a proton from TPP forming a carbanion b) catalyzing the condensation of the TPP carbanion with pyruvate to form hydroxyethyl-TPP c) oxidation and transfer of the hydroxyethyl group to lipoic acid d) reoxidation of lipoic acid in order to keep the reaction going Once acetyl-CoA is formed, it usually takes one of two paths. The first is further oxidation in the citric acid cycle. The second is: a) conversion to oxaloacetate which generates glucose by gluconeogenesis b) synthesis of galactose c) activates transcription factors which signal the synthesis of peptidase enzymes d) it is used in the synthesis of fatty acids.
Define the following terms: a. thiolytic cleavage b. ketogenesis c. ketone bodies d. α-oxidation e. ACC
The citric acid cycle enzyme at which FADH2 is generated is: A) isocitrate dehydrogenase. B) x-ketoglutarate dehydrogenase. C) succinate dehydrogenase. OD) malate dehydrogenase.
Consider the reaction converting succinate into fumarate (see p. 560). (a) Is succinate oxidized or reduced in this reaction (and how do you know)? (b) You add molecular O2 as the redox partner for this reaction. Draw the Lewis dot structures for the relevant moeities on succinate, fumarate, O2 and H2O. (c) What is AG" for for the conversion of succinate into fumarate in the presence of O2 and H2O?
Does Mannitol form a bit in the presence of H2O2?
What coenzymes are required for the ββ-oxidation pathway? A) NAD+ and FAD only B) NAD+ and CoASH only C) CoASH only D) FAD and CoASH only E) FAD, NAD+, and CoASH
If a slight deficiency in the Vitamin B1 derivative Thiamine Pyrophosphate (TPP) leads to an accumulation of substrates for reactions utilizing this cofactor, A) what substrates would ‘accumulate’ in a human after eating a an enormous amount of sugar-laden Halloween candy, AND what would be the regulatory consequences on carbohydrate metabolism? B) what substrates would accumulate in yeast cells cultured under anaerobic (no oxygen) conditions AND what would be the regulatory consequences on carbohydrate metabolism? C) and lastly, what substrates would accumulate in plant cells incorporating carbon and generating energy solely using photosynthesis?
Choose which enzyme is likely deficient based on the symptoms described below: Elevated blood lactate, alanine and pyruvate a) lactate dehydrogenase d) citrate synthase c) pyruvate dehydrogenase d) pyruvate kinase
What are the metabolic effects of not being able to produce the M subunit of phosphofructokinase?

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How many molecules of ATP are generated when a. stearate (18:0), b. oleate (18:1 cis A9), and c. heptadecanoic (17:0) acid are completely oxidized via ẞ oxidation?