Drag and drop the following events of an action potential into its correct order: Voltage-gated Na* channels open and Na* floods into the neuron. Nat channels inactivate and voltage gated K* channels open. Ligand-gated Na* channels open. Voltage gated K* channels close and membrane potential approaches the resting membrane potential.

Drag and drop the following events of an action potential into its correct order: Voltage-gated Na* channels open and Na* floods into the neuron. Nat channels inactivate and voltage gated K* channels open. Ligand-gated Na* channels open. Voltage gated K* channels close and membrane potential approaches the resting membrane potential.

Human Anatomy & Physiology (11th Edition)

11th Edition

ISBN:9780134580999

Author:Elaine N. Marieb, Katja N. Hoehn

Publisher:Elaine N. Marieb, Katja N. Hoehn

Chapter1: The Human Body: An Orientation

Section: Chapter Questions

Problem 1RQ: The correct sequence of levels forming the structural hierarchy is A. (a) organ, organ system,...

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An influx of positive sodium ions eventually causes closing of the inactivation gates of voltage regulated sodium channels, stopping the influx of positive sodium ions, and thus the final stages of an action potential are regulated by _____ feedback.
Which of these ions is actively transported through the cell membrane to re-establish a resting potential? K and Mg Na and K Mg and Cl Ca and Cl
A membrane potential (Vm) labeled axis on the graph In the graph draw the phases of the action potential Include the channels involved and when they open and close matching them to the Vm Indicate the periods in which the action potential can or cannot occur
Which of the following situations in a neuron results in action potentials that have extended depolarization phases? O None of these result in extended depolarization phase. O Compromising the inactivation mechanism in voltage-gated Na* channels. O Stimulating the voltage-gated K* channels. O Inhibiting the voltage-gated Na* channels. O Compromising the inactivation mechanism in voltage-gated K* channels.
rearrange these in the correct order of events in an action potential.
A theoretical drug blocks Na* leak channels - what effect would this have on a neuron's resting membrane potential and why? Would this drug affect the neuron's ability to generate an action potential and why/why not?
Place the following events in chronological order from 1-8: Nat enters the cell, and depolarization occurs to approximately +30 mV. The voltage across the cell membrane is -70 mV, the resting membrane potential. Upon reaching the peak of the action potential, the VG Nat channels are inactivated by the closing of their inactivation gate and the activation gate of each VG K channel opens. VG K channels close by the closing of their activation gate, and the resting membrane potential is gradually restored. An excitatory post-synaptic potential depolarizes the membrane to threshold and the activation gate of VG Nat channels open. Upon returning to the resting membrane potential, VG Na channels are reset by opening of the inactivation gate and the closing of the activation gate. VG K+ channels are slow to close, resulting in an excess of K* efflux and hyperpolarization. Depolarization occurs as K+ flows out of the cell.
Inhibitory postsynaptic potentials cause what type of change at the post-synaptic membrane? Hyperpolarization Depolarization Decreased threshold No change
Which of the following can increase the likelihood of a neuron generating an action potential? tetrodotoxin (TTX) reducing the external concentration of K+ decreased diffusion of K+ through leak channels out of the neuron All of the above
State whether the channels listed would be open or closed at the corresponding numbered phases of the action potential graph. Phase on Graph Sodium Voltage Gated channels Potassium Voltage Gated channels 1 2 3 4
Which of the following is FALSE about action potentials? At resting potential, there are more potassium channels open and most sodium channels are closed. Depolarization is due to the flow of Na+ out of the axon via voltage-gated Na+ channels. Hyperpolarization is due to continued loss of K+ from the axon. During repolarization, K+ flows out of the axon along its concentration gradient. Resting membrane potential is primarily established by the Na+/K+ pump.
The membrane potential caused by differences in ions across the plasma membrane of an electrically excitable cell (when not stimulated) is called: action potential resting membrane potential graded potential local potential