Pancreatitis

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School

Temple University *

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Course

512

Subject

Nursing

Date

Nov 24, 2024

Type

docx

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5

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Pancreatitis Inflammation of pancreas. Acute or chronic. Acute: Digestion of the organ by enzymes it produces, particularly trypsin. Indications: Severe abdominal pain. Pain often radiates to back. Nausea and vomiting. Fever. Jaundice. Confusion. Hyperglycemia. Chronic: Obstruction of pancreatic ducts. Destruction of secreting cells. Frequently related to alcohol consumption. Indications: Those for acute. Weight loss. Diabetes mellitus. Steatorrhea. Surgery if pain does not respond to medical management. Nursing interventions: Keep NPO to rest pancreas and stop production of enzymes. Establish and monitor nasogastric tube with suction. Maintain fluid and electrolyte balance. Place in semi-Fowler position or, especially for pain relief, keep knees flexed when sitting in bed or lying on side with pillow pressed to abdomen. Monitor for: Infection. Shock. Hyperglycemia. Administer parenteral nutrition. Administer medications such as: Antacids. Analgesics. Anticholinergics. For chronic pancreatitis, give medications for treatment of exocrine insufficiency. Nursing Focus & Concepts Pancreatitis Definition: Inflammation of pancreas. Results in premature activation of digestive enzymes. Either acute or chronic. Concepts: Digestion.
Pancreatitis. Assessment: Acute pancreatitis: Sudden, severe epigastric and/or abdominal pain. Tachypnea. Tachycardia. Basilar crackles. Hypotension. Abdominal distention. Decreased bowel sounds. Nausea. Vomiting. Fatigue. Chronic pancreatitis: Similar in presentation to acute. Weight loss. Constipation. Mild jaundice. Early and correct assessment of subjective and objective data is key to client survival and return to health. Diagnosis: Acute pain from distention and inflammation of pancreas. Deficit in fluid volume due to nausea, vomiting and NPO status. Planning: Plan for: Relief of pain. Bowel rest. Return to normal fluid balance. Reducing or eliminating complications. Preventing future attacks. Prepare to administer: IV opioids. Antiemetics. IV fluids. Parenteral nutrition depending on length of inflammation. Nothing by mouth (NPO). Monitor for: Fluid shifts. Hypovolemia. Respiratory distress. Implementation: Administer: IV opioids. Antiemetics. IV fluids as prescribed. Monitor: Respiratory rate. Constipation.
Vital signs. Respiratory function. O 2 saturation. Tetany related to hypocalcemia. Fever. Kidney failure. Paralytic ileus. Changes in level of consciousness. Maintain bowel rest by keeping the client NPO. Teach about relationship of alcohol consumption to pancreatitis. Evaluation: Client will experience adequate pain control. Client’s fluids and electrolytes will be in balance. Client will resume oral intake with no reports of pain or nausea. Client will verbalize understanding of alcohol use. Client will verbalize signs and symptoms of impending pancreatitis attack. Interdisciplinary Focus & Medical Management Pancreatitis Definition: Inflammation of pancreas. Results in premature activation of digestive enzymes. Either acute or chronic. Physiology: The pancreas has two important functions: Exocrine gland: Releases digestive enzymes. Enzymes activated in small intestine. Endocrine gland: Specific cells release insulin and glucagon. Essential to keep blood glucose in balance. Pathophysiology: Either acute or chronic. Result of injury to pancreas. Most common causes in United States: Gallstones - block enzymes from being released into small intestine. Chronic alcohol use - causes inflammation. Enzymes prematurely activated in pancreas causing damage/inflammation. As inflammation occurs, pancreatic cells are damaged. In severe cases, pancreatic cells are permanently lost resulting in diabetes mellitus. Risk Factors: Chronic alcohol use. Middle aged. Smoking. Family history. Injury to pancreas. Pregnancy. Hyperlipidemia. Medications:
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Diuretics. Estrogen. NSAIDs. Infection. Signs and Symptoms: Severe abdominal pain. Acute symptoms: Nausea. Vomiting. Jaundice. Decreased or absent bowel sounds. Shock due to hemorrhage. Chronic symptoms: Weight loss. Constipation. Diabetes mellitus. Steatorrhea (fatty stool). Diagnostic Testing: Observation of signs and symptoms. Elevated: Serum amylase. Serum lipase. Sedimentation rate. Leukocytosis - may be first indication. Chronic may not see rise in lab values. Imaging studies may determine: Calcifications. Ductal dilatation. Pancreatic enlargement. Treatment: Fluid and electrolyte imbalances - fluid replacement. Antibiotics for infections. NPO and NG suction for bowel rest. IV pain relief. Chronic pancreatitis: Pancreatic enzyme replacement. Low fat diet. Pain relief. Surgical removal of gallstones or diversion of bile. Decreased alcohol intake. Complications: Acute may become chronic. Loss of pancreatic cells can be life threatening. Local pseudocysts and abscesses. Systemic complications: Pulmonary effusion. Acute respiratory distress syndrome (ARDS).
Hypotension. Hypocalcemia. Diabetes mellitus. Respiratory failure. Kidney failure. Abdominal compartment syndrome. Hypovolemic shock. Expected Outcomes: Clients with acute pancreatitis can regain pancreatic function with early identification. Addressing pain and fluid balance are essential for positive outcome. Client will resume a diet with no reports of abdominal pain and nausea. Clients with chronic pancreatitis will improve by decreasing alcohol consumption and following medication regimens.