Campbell Biology in Focus, Books a la Carte Edition; Modified Mastering Biology with Pearson eText - ValuePack Access Card - for Campbell Biology in Focus (2nd Edition)
2nd Edition
ISBN: 9780134433769
Author: Lisa A. Urry, Michael L. Cain, Steven A. Wasserman
Publisher: PEARSON
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Chapter 35.3, Problem 1CC
Summary Introduction
To determine:
The type of disorder of myasthenia gravis. In this condition, antibodies bind to and block certain receptors on muscle cells, preventing muscle contraction.
Introduction:
This condition called myasthenia gravis weakens the skeletal muscles and causes difficulty in the movement of muscles and leads to the weakness. The destruction of nerves, results in the difficulty in locomotion and movement.
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Myasthenia gravis is an autoimmune disorder. Tag the statement true or false.
Why do antibodies contribute to the symptoms of myasthenia gravis?
Explain about the mechanism for involvement autoimmune of autoantibodies in myasthenia gravis.
Chapter 35 Solutions
Campbell Biology in Focus, Books a la Carte Edition; Modified Mastering Biology with Pearson eText - ValuePack Access Card - for Campbell Biology in Focus (2nd Edition)
Ch. 35.1 - Pus is both a sign of infection and an indicator...Ch. 35.1 - MAKE CONNECTIONS How do the molecules that...Ch. 35.1 - Prob. 3CCCh. 35.2 - Prob. 1CCCh. 35.2 - Explain how memory cells strengthen the immune...Ch. 35.2 - WHAT IF? If both copies of a light-chain gene and...Ch. 35.3 - Prob. 1CCCh. 35.3 - Prob. 2CCCh. 35.3 - Prob. 3CCCh. 35 - Prob. 1TYU
Ch. 35 - Prob. 2TYUCh. 35 - Prob. 3TYUCh. 35 - Prob. 4TYUCh. 35 - Prob. 5TYUCh. 35 - Prob. 6TYUCh. 35 - Prob. 7TYUCh. 35 - DRAW IT Consider a pencil-shaped protein with two...Ch. 35 - MAKE CONNECTIONS Contrast clonal selection with...Ch. 35 - Prob. 10TYUCh. 35 - FOCUS ON EVOLUTION Describe one invertebrate...Ch. 35 - Prob. 12TYUCh. 35 - Prob. 13TYU
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- what would some effective treatments be for this condition?arrow_forwardA nerve conduction velocity test measures nerve conduction velocity. In this test, an electrical stimulus is applied to a peripheral nerve and the length of time it takes for the corresponding muscle to contract is measured. Because the length of the nerve from stimulus point to muscle and the time it takes for the muscle to contract are both known, NCV can be calculated. This test result is helpful in distinguishing between different types of pathologies. For example, a patient with muscle weakness is being tested by a physician. There are three possible sources of the weakness: Disease of the muscle itself such as muscular dystrophy. In this condition, normal muscle tissue is replaced by adipose tissue and the muscle becomes incapable of contraction. Disease at the neuromuscular junction (NMJ) such as myasthenia gravis. This condition is described in the Skeletal Muscle Histology exercise and results from damage to the acetylcholine receptors on the muscle end plate. Disease…arrow_forwardWhy are direct acting cholinomimetics NOT used in the management of myasthenia gravis?arrow_forward
- "Motor neurons trigger action potentials in muscle cell membranes that open voltage-sensitive Ca2+ channels in T tubules, allowing extracellular Ca2+ to enter the cytosol, bind to troponin C, and initiate rapid muscle contraction", is true or false.arrow_forwardA patient has been exposed to the organophosphate pesticide malathion,which inactivates acetylcholinesterase. Which of the following symptoms would you predict: blurring of vision, excess tear formation, frequent or involuntary urination, pallor (pale skin), muscle twitching, orcramps? Would atropine be an effective drug to treat the symptoms?(See Clinical Impact 16.2 for the action of atropine.) Explain.arrow_forwardover a period of 3 years , a 40 year old man develops generalized dystonic , hyperkinetic movements and progressive dementia , his mother and one sister have a similar disease but his father and another sister are healthy. which of the following lesions is mostly likely to be present in the cerebral nervous system of the patient and his affected family members. A) atrophy of the caudate nuclei, B) loss of pigmented cells in the sustengria nigra, C) microglial nodules and multinucleated giant cells in the white matter, D) neurofibrillary tangles in the cortical neurons, E) spongiform changes in the cerebral cortexarrow_forward
- Why talk about Duchenne muscular dystrophy (DMD)?arrow_forwardA neurotransmitter is a chemical messenger that carries, boosts, and balances signals between neurons, nerve cells, and other cells in the body. a) Identify two disorders that can result in a depletion, damage, or lack of neurotransmitters in our body. Then list and explain at least two neurotransmitters that can result in these disorders developing.arrow_forwardOutline the pathogenesis of Myasthenia gravis and the consequential effects from the disruption at the neuromuscular junction. Is Myasthenia gravis considered an upper or lower motor neuron disorder?arrow_forward
- In the rare neuromuscular disorder Myasthenia Gravis, autoantibodies are produced which inhibit acetylcholine receptor (nicotinic receptor) activity. Explain how inhibition of acetylcholine receptor activity at the neuromuscular junction will affect the sequence of events in muscular excitation and contraction in response to stimuli, and muscle function? (Mention the effects on all the significant events involved in excitation and contraction of muscles, and how it would affect muscle function).arrow_forwardExplain why nerve impulse propagation is compromised in patients with multiple sclerosis.arrow_forward2) List the names of the two major Contractile Proteins. Which of the proteins is "bound" and "anchored" to the cell membrane? The other protein essentially "floats" within the ctyoplasm; which contractile protein "floats". 3) Contractions in a muscle cell (or fiber) is always stimulated and intiated by a rapid rise in intrallcellular calcium. Write a brief paragraph or two explaining the differences in the sources of Contraction- causing-Calcium within the 3 different muscle-cell types.arrow_forward
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