
A.
To define: The term inflammatory bowel disease and compare the pathophysiologic processes and manifestations of Crohn disease and ulcerative colitis.
Introduction: Crohn's disease is a provocative bowel disease. It produces inflammation of the digestive tract, which can lead to abdominal pain, critical diarrhea, weakness, weight loss, and starvation. Infection caused by Crohn's syndrome can affect different areas of the digestive tract in several people.
B.
To describe: The possible association between genetic and environmental factors in the pathogenesis of Crohn’s disease.
Introduction: The pathogenesis of Crohn's disease includes host, genetic, and environmental factors. These determinants result in irregularities in the innate and adaptive immune systems and structure of the intestinal microbiota. Epidemiologic and migration studies support an environmental element in the expansion of CD.
C.
To describe: The relationship between the use of the monoclonal antibody infliximab and the pathogenesis of the inflammatory lesions that occur in Crohn’s disease.
Introduction: Inflammatory bowel disease (IBD), including Crohn's disease and ulcerative colitis, is described by chronic relapsing intestinal inflammation. It is estimated that IBD occurs from an unusual and ongoing immune reply to the microbes in the gut, which are catalyzed by the genetic sensitivity of the individual.

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Chapter 29 Solutions
Essentials of Pathophysiology: Concepts of Altered States
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