Three mechanisms that prevent inappropriate coagulation in undamaged vessels. Introduction: Hemostasis is the process by which the body stops blood loss, and there are three major hemostatic mechanisms: spasm of blood vessels, formation of platelet plug, and clotting of blood. When an injury occurs, local blood vessels first constrict to reduce blood flow to the area; next, platelets adhere to the collagen fibers of the vessel wall exposed by the injury and release substances that further promote platelet aggregation and vasoconstriction such as serotonin, adenosine diphosphate, and thromboxane A 2 ; and subsequently, the coagulation of blood occurs. These mechanisms together help to stop blood loss. Blood coagulation itself consists of two pathways (the intrinsic and extrinsic mechanisms) that reach a common convergence point after following different steps. The common convergence point of the two pathways is reached when factor X is formed. This continues on to the end point, which is the formation of fibrin polymer molecules that form the blood clot.

Question

Want to see the full answer?

Answer 1

Question

Chapter 18.5, Problem 10AYLO

Summary Introduction

To discuss:

Three mechanisms that prevent inappropriate coagulation in undamaged vessels.

Introduction:

Hemostasis is the process by which the body stops blood loss, and there are three major hemostatic mechanisms: spasm of blood vessels, formation of platelet plug, and clotting of blood. When an injury occurs, local blood vessels first constrict to reduce blood flow to the area; next, platelets adhere to the collagen fibers of the vessel wall exposed by the injury and release substances that further promote platelet aggregation and vasoconstriction such as serotonin, adenosine diphosphate, and thromboxane A₂; and subsequently, the coagulation of blood occurs. These mechanisms together help to stop blood loss. Blood coagulation itself consists of two pathways (the intrinsic and extrinsic mechanisms) that reach a common convergence point after following different steps. The common convergence point of the two pathways is reached when factor X is formed. This continues on to the end point, which is the formation of fibrin polymer molecules that form the blood clot.

Expert Solution & Answer

Want to see the full answer?

Check out a sample textbook solution

See solution

Students have asked these similar questions

Molecular Biology Please help with question. thank you You are studying the expression of the lac operon. You have isolated mutants as described below. In the presence of glucose, explain/describe what would happen, for each mutant, to the expression of the lac operon when you add lactose AND what would happen when the bacteria has used up all of the lactose (if the mutant is able to use lactose).5. Mutations in the lac operator that strengthen the binding of the lac repressor 200 fold 6. Mutations in the promoter that prevent binding of RNA polymerase 7. Mutations in CRP/CAP protein that prevent binding of cAMP8. Mutations in sigma factor that prevent binding of sigma to core RNA polymerase

Molecular Biology Please help and there is an attached image. Thank you. A bacteria has a gene whose protein/enzyme product is involved with the synthesis of a lipid necessary for the synthesis of the cell membrane. Expression of this gene requires the binding of a protein (called ACT) to a control sequence (called INC) next to the promoter. A. Is the expression/regulation of this gene an example of induction or repression?Please explain:B. Is this expression/regulation an example of positive or negative control?C. When the lipid is supplied in the media, the expression of the enzyme is turned off.Describe one likely mechanism for how this “turn off” is accomplished.

Molecular Biology Please help. Thank you. Discuss/define the following:(a) poly A polymerase (b) trans-splicing (c) operon