Anatomy & Physiology
1st Edition
ISBN: 9781938168130
Author: Kelly A. Young, James A. Wise, Peter DeSaix, Dean H. Kruse, Brandon Poe, Eddie Johnson, Jody E. Johnson, Oksana Korol, J. Gordon Betts, Mark Womble
Publisher: OpenStax College
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Chapter 17, Problem 41CTQ
Compare and contrast the signaling events involved with the second messengers cAMP and IP3.
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Compare and contrast the signaling events involved with the second messengers cAMP and IP3.
compare and contrast the signaling events involved with the second messanger camp and IP3
Trace the sequence of events in signal transduction for each of the following second messengers: cyclic AMP, inositol trisphosphate (IP3 ), diacylglycerol (DAG), and calcium ions.
Chapter 17 Solutions
Anatomy & Physiology
Ch. 17 - Visit this link...Ch. 17 - Visit this link...Ch. 17 - Visit this link...Ch. 17 - Visit this link...Ch. 17 - Visit this link...Ch. 17 - Endocrine glands ________. secrete hormones that...Ch. 17 - Chemical signaling that affects neighboring cells...Ch. 17 - A newly developed pesticide has been observed to...Ch. 17 - A small molecule binds to a G protein, preventing...Ch. 17 - A student is in a car accident, and although not...
Ch. 17 - The hypothalamus is functionally and anatomically...Ch. 17 - Which of the following is an anterior pituitary...Ch. 17 - How many hormones are produced by the posterior...Ch. 17 - Which of the following hormones contributes to the...Ch. 17 - Which of the following statements about the...Ch. 17 - The secretion of thyroid hormones is controlled by...Ch. 17 - The development of a goiter indicates that...Ch. 17 - Iodide ions cross from the bloodstream into...Ch. 17 - When blood calcium levels are low, PTH stimulates...Ch. 17 - Which of the following can result from...Ch. 17 - The adrenal glands are attached superiorly to...Ch. 17 - What secretory cell type is found in the adrenal...Ch. 17 - Cushings disease is a disorder caused by ________....Ch. 17 - Which of the following responses s not part of the...Ch. 17 - What cells secrete melatonin? melanocytes...Ch. 17 - The production of melatonin is inhibited by...Ch. 17 - The gonads produce what class of hormones? amine...Ch. 17 - The production of FSH by the anterior pituitary is...Ch. 17 - The function of the placental hormone human...Ch. 17 - If an autoimmune disorder targets the alpha cells,...Ch. 17 - Which of the following statements about insulin is...Ch. 17 - The walls of the atria produce which hoimone?...Ch. 17 - The end result of the RAAS is to ________. reduce...Ch. 17 - Athletes may take synthetic EPO to boost their...Ch. 17 - Hormones produced by the thymus play a role in the...Ch. 17 - The anterior pituitary gland develops from which...Ch. 17 - In the elderly, decreased thyroid function causes...Ch. 17 - Describe several main differences in the...Ch. 17 - Compare and contrast endocrine and exocrine...Ch. 17 - True or false: Neurotransmitters are a special...Ch. 17 - Compare and contrast the signaling events involved...Ch. 17 - Describe the mechanism of hormone response...Ch. 17 - Compare and contrast the anatomical relationship...Ch. 17 - Name the target tissues for prolactin.Ch. 17 - Explain why maternal iodine deficiency might lead...Ch. 17 - Define hyperthyroidism and explain why one of its...Ch. 17 - Describe the role of negative feedback in the...Ch. 17 - Explain why someone with a parathyroid gland tumor...Ch. 17 - What are the three regions of the adrenal cortex...Ch. 17 - If innervation to the adrenal medulla were...Ch. 17 - Compare and contrast the short-term and long-term...Ch. 17 - Seasonal affective disorder (SAD) is a mood...Ch. 17 - Retinitis pigmentosa (RP) is a disease that causes...Ch. 17 - Compare and contrast the role of estrogens and...Ch. 17 - Describe the role of placental secretion of...Ch. 17 - What would be the physiological consequence of a...Ch. 17 - Why is foot care extremely important for people...Ch. 17 - Summarize the role of GI tract hormones following...Ch. 17 - Compare and contrast the thymus gland in infancy...Ch. 17 - Distinguish between the effects of menopause and...
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- Briefly describe the series of events that takesplace when cAMP acts as a second messenger.arrow_forwardExplain why polar hormones cannot regulate their target cells without using second messengers. Alsoexplain in a step-by-step manner how cyclic AMP is used as a second messenger in hormone action.arrow_forwardDescribe the main mechanisms by which G proteins are activated upon norepinephrine bindingarrow_forward
- In the liver and muscle cells, epinephrine stimulates the release of glucose from glycogen by inhibiting glycogen synthesis and stimulating glycogen breakdown. Outline the signaling events that occur after epinephrine binds to its receptor and the resultant increase in the concentration of intracellular cAMP and release of glucose.arrow_forwardThe phosphodiesterase that catalyzes hydrolysis of cyclic AMP is inhibited by caffeine. What overall effect would caffeine have on a signal that is mediated by cAMP?arrow_forwardVisit this link (http://openstaxcollege.org/l/hormonebind) to watch an animation of the events thatoccur when a hormone binds to a cell membrane receptor.What is the secondary messenger made by adenylyl cyclaseduring the activation of liver cells by epinephrine?arrow_forward
- Describe two features of insulin signaling that affect glucose utilization. Aβ-adrenergic response can be modulated through the actions of a receptorkinase and arrestin because phosphorylation by the kinase desensitizes thereceptor. How might signaling by a tyrosine receptor kinase, such as theinsulin receptor, be modulated?arrow_forwardName two second messengers of GPCRs and identify the type of GPCR each second messenger commonly associates with (Gs, Gi, Gq, G12/13, Go; there are many possible answers for this question).arrow_forwardGive three examples of second messengers.arrow_forward
- Give one mechanism by which a GPCR signal is stopped following stimulation. Hint, it could also be by removing the downstream second messenger.arrow_forwardOverexpression of this receptor in the cells of the adrenal gland causes Cushing's syndrome, a disease caused by the overproduction of cortisol. One strategy that scientists have employed to treat this disease is the use of small molecules that bind to, but do not activate, MC2R. This type of molecule is generically referred to as an antagonist. How can an antagonist bind to the same receptor as ACTH but not activate it? O a. The antagonist can bind covalently while ACTH binds non-covalently O b. The antagonist is only partially complimentary to the binding pocket of MC2R O The antagonist binds to ACTH and blocks it from binding to the receptor properly O d. The antagonist is the exact same structure as ACTH but since it is synthetic it doesn't work One cause of Cushing's syndrome is the inappropriate production of GPCRS in the adrenal gland that are not normally present in those cells. When these receptors are activated they result in the production of cortisol. Which statement about…arrow_forwardWhich statement correctly identifies how a lipid-soluble hormone that diffuses from the blood and through the interstitial fluid identifies which cells in the tissue to act upon? O The cell closest to the diffusion site. O The presence of a receptor located in the cytoplasm or nucleus specific to the hormone in cells. O The presence of a receptor embedded in the plasma membrane of target cells. O The presence of a channel protein embedded in the plasma membrane for the lipid-soluble hormone to diffuse through. Save for Later 80 F3 F4 F5 F6 8 F7 Attempts: 0 of 3 used F8 F9 Submit Answer F10arrow_forward
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